2 research outputs found
Solar energy integration in the steel industry : feasibility, challenges as well as solutions
This study addresses solar power feasibility within the steel industry, its feasibility, challenges, and solutions towards bridging the adoption barriers. Steel manufacturing has very high levels of energy, greenhouse gas emission, and substantial fossil fuel use. This study examines how solar power can achieve cost savings on operations, raise the level of sustainability, and alleviate environmental implications. The key challenge presented here is the front-end cost of solar systems by small steel makers. The system encourages industries to utilize solar power with a view to reducing carbon footprint, cutting costs, and applying stringent environmental policies. The system approaches these through literature review, economic analysis, technology innovation, policy analysis, and hybrid power system. Empirical observations show that even if high up-front cost is a source of concern, government incentives through subsidies and tax credits can override such concerns. Synergizing the sun with traditional sources of power and utilizing advanced technologies can make the steelmaking process cheaper and more eco-friendly
Impact of high salt diet on N-acetylgalactosamine-4-sulfatase activity, glycosaminoglycans, and kininogen in rat kidney
Glycoconjugate Journal, 30, 667–676Note : if this item contains full text it may be a preprint, author manuscript, or a Gold OA copy that permits redistribution with a license such as CC BY. The final version is available through the publisher’s platform.N-acetylgalactosamine-4-sulfatase (Arylsulfatase B; ARSB) is the enzyme that removes sulfate groups from the N-acetylgalactosamine-4-sulfate residue at the non-reducing end of chondroitin-4-sulfate (C4S) and dermatan sulfate (DS). Previous studies demonstrated reduction in cell-bound high molecular weight kininogen in normal rat kidney (NRK) epithelial cells when chondroitin-4-sulfate content was reduced following overexpression of ARSB activity, and chondroitinase ABC produced similar decline in cell-bound kininogen. Reduction in the cell-bound kininogen was associated with increase in secreted bradykinin. In this report, we extend the in vitro findings to in vivo models, and present findings in Dahl salt-sensitive (SS) rats exposed to high (SSH) and low salt (SSL) diets. In the renal tissue of the SSH rats, ARSB activity was significantly less than in the SSL rats, and chondroitin-4-sulfate and total sulfated glycosaminoglycan content were significantly greater. Disaccharide analysis confirmed marked increase in C4S disaccharides in the renal tissue of the SSH rats. In contrast, unsulfated, hyaluronan-derived disaccharides were increased in the rats on the low salt diet. In the SSH rats, with lower ARSB activity and higher C4S levels, cell-bound, high-molecular weight kininogen was greater and urinary bradykinin was lower. ARSB activity in renal tissue and NRK cells declined when exogenous chloride concentration was increased in vitro. The impact of high chloride exposure in vivo on ARSB, chondroitin-4-sulfation, and C4S-kininogen binding provides a mechanism that links dietary salt intake with bradykinin secretion and may be a factor in blood pressure regulation
