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Chelerythrine rapidly induces apoptosis through generation of reactive oxygen species in cardiac myocytes.
Beta-adrenergic cardiac hypertrophy is mediated primarily by the beta(1)-subtype in the rat heart.
Chelerythrine rapidly induces apoptosis through generation of reactive oxygen species in cardiac myocytes.
Chelerythrine rapidly induces apoptosis through generation of reactive oxygen species in cardiac myocytes.
Beta-adrenergic cardiac hypertrophy is mediated primarily by the beta(1)-subtype in the rat heart.
The Akt-glycogen synthase kinase 3beta pathway regulates transcription of atrial natriuretic factor induced by beta-adrenergic receptor stimulation in cardiac myocytes.
Endocytosis machinery is required for beta1-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes.
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