1,721,017 research outputs found
Joint destruction in septic arthritis
No full text[No abstract available]Goldenberg DL, 1998, LANCET, V351, P197, DOI 10.1016-S0140-6736(97)09522-6; Kaandorp CJE, 1995, ARTHRITIS RHEUM, V38, P1819, DOI 10.1002-art.1780381215; Uthman I, 2003, CLIN RHEUMATOL, V22, P359, DOI 10.1007-s10067-003-0717-20
Joint destruction in septic arthritis
No full text[No abstract available]Goldenberg DL, 1998, LANCET, V351, P197, DOI 10.1016-S0140-6736(97)09522-6; Kaandorp CJE, 1995, ARTHRITIS RHEUM, V38, P1819, DOI 10.1002-art.1780381215; Uthman I, 2003, CLIN RHEUMATOL, V22, P359, DOI 10.1007-s10067-003-0717-20
The usefulness of PPD testing in inflammatory bowel disease patients before infliximab therapy
No full text[No abstract available][Anonymous], 2000, AM J RESP CRIT CARE, V161, pS221; Mow WS, 2004, CLIN GASTROENTEROL H, V2, P309, DOI 10.1053-S1542-3565(04)00060-6; Uthman I, 2004, CLIN RHEUMATOL, V23, P279, DOI 10.1007-s10067-004-0873-z21
Successful treatment of recalcitrant pyoderma gangrenosum with infliximab complicated by tuberculosis despite negative screening tests [1]
No full text[No abstract available]Grange F, 2002, DERMATOLOGY, V205, P278, DOI 10.1159-000065860; Jenne L, 2004, BRIT J DERMATOL, V150, P380, DOI 10.1111-j.1365-2133.2003.05760.x; Regueiro M, 2003, AM J GASTROENTEROL, V98, P1821, DOI 10.1016-S0002-9270(03)00430-1; Singh M, 2004, CLIN EXP DERMATOL, V29, P196, DOI 10.1111-j.1365-2230.2004.01470.x; Uthman I, 2004, CLIN RHEUMATOL, V23, P279, DOI 10.1007-s10067-004-0873-z54
The use of biological therapy in refractory rheumatic diseases other than rheumatoid arthritis: Experience at a tertiary care center in Lebanon
No full text[No abstract available]Arayssi T, 2005, CLIN EXP RHEUMATOL, V23, P427; Fricker M, 2007, RHEUMATOL INT, V27, P493, DOI 10.1007-s00296-006-0252-y; Josselin L, 2008, ANN RHEUM DIS, V67, P1343, DOI 10.1136-ard.2007.083584; Levine TD, 2005, ARTHRITIS RHEUM, V52, P601, DOI 10.1002-art.20849; Sfikakis PP, 2007, RHEUMATOLOGY, V46, P736, DOI 10.1093-rheumatology-kem034; Touma Z, 2006, SCAND J RHEUMATOL, V35, P323, DOI 10.1080-03009740500484056; Touma Z, 2008, JOINT BONE SPINE, V75, P334, DOI 10.1016-j.jbspin.2007.05.011; UTHMAN I, 2005, CLIN RHEUMATOL, V25, P109; Uthman I, 2007, CLIN RHEUMATOL, V26, P2001, DOI 10.1007-s10067-007-0614-1; Uthman I, 2004, RHEUMATOLOGY, V43, P1198, DOI 10.1093-rheumatology-keh268; Uthman I, 2006, CLIN RHEUMATOL, V25, P540, DOI 10.1007-s10067-005-0029-9; Uthman I, 2004, CLIN RHEUMATOL, V23, P279, DOI 10.1007-s10067-004-0873-z; Uthman I, 2004, SEMIN ARTHRITIS RHEU, V33, P422, DOI 10.1016-j.semarthrit.2003.12.005; Uthman I, 2005, RHEUMATOLOGY, V44, P1200, DOI 10.1093-rheumatology-keh680; Uthman I, 2008, DERMATOLOGY, V216, P257, DOI 10.1159-000112938; Uthman IW, 2005, J AM ACAD DERMATOL, V53, P353, DOI 10.1016-j.jaad.2005.02.009; Wendling D, 2007, REV MED INTERNE, V28, P194, DOI 10.1016-j.revmed.2006.12.001; YAMOUT B, 2007, LUPUS, V100, P10
The use of tumor necrosis factor neutralization strategies in rheumatologic disorders other than rheumatoid arthritis in Lebanon
No full text[No abstract available]Stokes DG, 2003, SEMIN ARTHRITIS RHEU, V33, P1, DOI 10.1053-sarh.2003.50022; Uthman I, 1997, BRIT J RHEUMATOL, V36, P80655
Thrombotic renal and adrenal manifestations of primary antiphospholipid syndrome
No full text[No abstract available]ASHERSON RA, 1991, J RHEUMATOL, V18, P1; Espinosa G, 2003, LUPUS, V12, P569, DOI 10.1191-0961203303lu404oa; INAM S, 1991, POSTGRAD MED J, V67, P385; Kurtulmus N, 2009, RHEUMATOL INT, V30, P259, DOI 10.1007-s00296-009-0922-7; Uthman I, 2006, SEMIN ARTHRITIS RHEU, V35, P360, DOI 10.1016-j.semarthrit.2006.01.001; Uthman I, 2008, BLOOD REV, V22, P187, DOI 10.1016-j.blre.2008.03.0050
Infliximab as monotherapy in giant cell arteritis
No full text[No abstract available]Andonopoulos AP, 2003, ANN RHEUM DIS, V62, P1116, DOI 10.1136-ard.62.11.1116; Cantini F, 2001, ARTHRITIS RHEUM, V44, P2933, DOI 10.1002-1529-0131(200112)44:122933::AID-ART4823.0.CO;2-Y; Field M, 1997, RHEUMATOL INT, V17, P113, DOI 10.1007-s002960050019; Hernandez-Rodriguez J, 2004, RHEUMATOLOGY, V43, P294, DOI 10.1093-rheumatology-keh058; Hernandez-Rodriguez J, 2002, ARTHRIT RHEUM-ARTHR, V47, P29, DOI 10.1002-art1.10161; NESHER G, 1994, J RHEUMATOL, V21, P1283; Rozin AP, 2004, ANN RHEUM DIS, V63, P751; Uthman I, 2004, SEMIN ARTHRITIS RHEU, V33, P422, DOI 10.1016-j.semarthrit.2003.12.005; Weyand CM, 2000, ARTHRITIS RHEUM, V43, P1041, DOI 10.1002-1529-0131(200005)43:51041::AID-ANR123.0.CO;2-7; Weyand CM, 2003, ANN INTERN MED, V139, P50529161
Deep venous thrombosis in a patient with Behçet's disease and homozygous prothrombin (factor II) G20210A mutation on oral contraceptive pills
No full textBehçet's disease (BD) is a multisystemic disease of unknown aetiology characterized by chronic relapsing oral aphthous lesions, genital ulcers, uveitis, and vasculopathy of both arteries and veins of all sizes. We present the case of a female patient, with BD on oral contraceptive pills, who sustained deep venous thrombosis of the long saphenous vein. The patient was found to be a carrier of a homozygous mutation of the prothrombin (factor II) G20210A gene. Our case suggests that a thrombophilia work-up may be warranted in BD patients, especially young women before they are prescribed oral contraceptives. © Springer-Verlag 2005.Akar N, 1998, AM J HEMATOL, V58, P249, DOI 10.1002-(SICI)1096-8652(199807)58:3249::AID-AJH203.0.CO;2-3; Bertina R M, 1998, Curr Opin Hematol, V5, P339, DOI 10.1097-00062752-199809000-00006; Espinosa G, 2002, AM J MED, V112, P37, DOI 10.1016-S0002-9343(01)01048-8; Gul A, 1999, RHEUMATOLOGY, V38, P1298, DOI 10.1093-rheumatology-38.12.1298; Howard TE, 1997, BLOOD COAGUL FIBRIN, V8, P316, DOI 10.1097-00001721-199707000-00010; Leiba M, 2001, ANN RHEUM DIS, V60, P1081, DOI 10.1136-ard.60.12.1081; LIE JT, 1992, J RHEUMATOL, V19, P341; Martinelli I, 1998, NEW ENGL J MED, V338, P1793, DOI 10.1056-NEJM199806183382502; Salvarani C, 2000, J RHEUMATOL, V27, P515; Silingardi M, 2004, ARTHRIT RHEUM-ARTHR, V51, P177, DOI 10.1002-art.20237; Taher A, 2001, THROMB HAEMOSTASIS, V86, P723; Taher A, 2003, THROMB HAEMOSTASIS, V89, P945; Toydemir PB, 2000, J RHEUMATOL, V27, P2849; Tursen U, 2001, ARCH DERMATOL RES, V293, P537, DOI 10.1007-PL00007469; Uthman I, 1997, BRIT J RHEUMATOL, V36, P806; Vaya A, 2000, HAEMATOLOGICA, V85, P42544
The abdominal manifestations of the antiphospholipid syndrome
No full textObjectives. To study the abdominal manifestations of the antiphospholipid syndrome (APS). Methods. We reviewed the medical literature from 1968 to 2006 using MEDLINE and the key words: APS, anticardiolipin antibodies, lupus anticoagulant, antiphospholipid (aPL) antibodies, catastrophic antiphospholipid syndrome, liver, hepatic biliary, pancreas, spleen, gastrointestinal and abdominal. Results. Liver involvement is the most frequent abdominal manifestation associated with APS. Various hepatic manifestations have been reported including Budd-Chiari syndrome, hepatic-veno-occlusive disease and occlusion of small hepatic veins, nodular regenerative hyperplasia, hepatic infarction, cirrhosis, portal hypertension, autoimmune hepatitis and biliary cirrhosis. Acute intestinal infarction, intestinal angina, and intestinal bleeding have also been reported in association with aPL in addition to few sporadic cases of splenic infarction and acute pancreatitis. Conclusion. A high index of suspicion for any signs of abdominal involvement should be considered in patients with APS. In addition screening for aPL should be carried out in patients who present with hepatic vein occlusion and unexplained signs of intestinal angina. © The Author 2007. Published by Oxford University Press on behalf of the British Society for Rheumatology. 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