29 research outputs found
Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte-6
No full text<p><b>Copyright information:</b></p><p>Taken from "Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte"</p><p></p><p>Molecular Human Reproduction 2008;14(7):393-398.</p><p>Published online 20 May 2008</p><p>PMCID:PMC2453241.</p><p>© The Author 2008. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: [email protected]</p
Alternative splicing of the mouse embryonic poly(A) binding protein mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte-3
No full text<p><b>Copyright information:</b></p><p>Taken from "Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte"</p><p></p><p>Molecular Human Reproduction 2008;14(7):393-398.</p><p>Published online 20 May 2008</p><p>PMCID:PMC2453241.</p><p>© The Author 2008. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: [email protected]</p
Alternative splicing of the mouse embryonic poly(A) binding protein mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte-5
No full text<p><b>Copyright information:</b></p><p>Taken from "Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte"</p><p></p><p>Molecular Human Reproduction 2008;14(7):393-398.</p><p>Published online 20 May 2008</p><p>PMCID:PMC2453241.</p><p>© The Author 2008. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: [email protected]</p
Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte-0
No full text<p><b>Copyright information:</b></p><p>Taken from "Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte"</p><p></p><p>Molecular Human Reproduction 2008;14(7):393-398.</p><p>Published online 20 May 2008</p><p>PMCID:PMC2453241.</p><p>© The Author 2008. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: [email protected]</p
Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte-4
No full text<p><b>Copyright information:</b></p><p>Taken from "Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte"</p><p></p><p>Molecular Human Reproduction 2008;14(7):393-398.</p><p>Published online 20 May 2008</p><p>PMCID:PMC2453241.</p><p>© The Author 2008. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: [email protected]</p
Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte-2
No full text<p><b>Copyright information:</b></p><p>Taken from "Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte"</p><p></p><p>Molecular Human Reproduction 2008;14(7):393-398.</p><p>Published online 20 May 2008</p><p>PMCID:PMC2453241.</p><p>© The Author 2008. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: [email protected]</p
Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte-1
No full text<p><b>Copyright information:</b></p><p>Taken from "Alternative splicing of the mouse embryonic poly(A) binding protein () mRNA is regulated by an exonic splicing enhancer: a model for post-transcriptional control of gene expression in the oocyte"</p><p></p><p>Molecular Human Reproduction 2008;14(7):393-398.</p><p>Published online 20 May 2008</p><p>PMCID:PMC2453241.</p><p>© The Author 2008. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: [email protected]</p
Long-term follicular dynamics and biochemical characteristics of dominant follicles in dairy cows subjected to acute heat stress
No full textFever in women may interfere with follicular development during controlled ovarian stimulation
No full textPurpose: This study aims to evaluate the effects of fever on follicular development in women undergoing controlled ovarian stimulation during in vitro fertilisation (IVF) and intracytoplasmic sperm injection (ICSI) cycles. Materials and methods: This was a retrospective observational self-controlled study at a tertiary-care fertility centre. Six gonadotropin stimulation cycles characterised by poor ovarian response in which women reported the occurrence of a febrile illness, were considered for evaluation. Fever-exposed cycles were compared to the next stimulation cycle in the same women. Primary outcome measures were final number of pre-ovulatory follicles (≥ 16 mm) and final peak serum estradiol levels (pg-mL). Other outcome measures were final number of medium-sized follicles (12-15 mm), final mean estradiol serum level per follicle ≥ 12 mm (pg-mL), total days of stimulation and total gonadotropin ampoules utilised. Results: Fever-exposed cycles were associated with significantly lower number of pre-ovulatory follicles (0.7 ± 0.8), significantly higher number of medium-size follicles (21.0 ± 4.5), and significantly reduced serum estradiol per follicle ≥12 mm (50.7 ± 11.7 pg-mL). They also required a significantly longer duration of ovarian stimulation (15.7 ± 3.3 days) and a significantly increased number of gonadotropin ampoules (47.2 ± 10.9). Four women had polycystic ovary syndrome and one hypothalamic hypogonadism. Conclusion: This preliminary report suggests a possible negative effect of fever on follicular development and ovarian estradiol production in some women undergoing controlled ovarian stimulation. © 2012 Informa UK Ltd.ARAD Z, 1982, BRIT POULTRY SCI, V23, P333, DOI 10.1080-00071688208447966; Aroyo A, 2007, THERIOGENOLOGY, V67, P1013, DOI 10.1016-j.theriogenology.2006.12.001; BADINGA L, 1993, THERIOGENOLOGY, V39, P797, DOI 10.1016-0093-691X(93)90419-6; BAUMGARTNER AP, 1987, ANIM REPROD SCI, V14, P309, DOI 10.1016-0378-4320(87)90021-2; BAUMGARTNER AP, 1988, J REPROD FERTIL, V84, P469; BAUMGARTNER AP, 1981, EXP CELL RES, V132, P359, DOI 10.1016-0014-4827(81)90111-7; Cheng H, 1999, ANIM REPROD SCI, V56, P279, DOI 10.1016-S0378-4320(99)00043-3; CURCI A, 1991, DEV BIOL, V144, P362, DOI 10.1016-0012-1606(91)90428-6; Edwards MJ, 2003, INT J HYPERTHER, V19, P295, DOI 10.1080-0265673021000039628; EDWARDS MJ, 1974, J EMBRYOL EXP MORPH, V32, P593; Feder ME, 1999, ANNU REV PHYSIOL, V61, P243, DOI 10.1146-annurev.physiol.61.1.243; Guzeloglu A, 2001, ANIM REPROD SCI, V66, P15, DOI 10.1016-S0378-4320(01)00082-3; HAFEZ ESE, 1964, J REPROD FERTIL, V7, P229; HOWELL JL, 1994, J DAIRY SCI, V77, P735; KHANNA A, 1995, MOL ENDOCRINOL, V9, P1431, DOI 10.1210-me.9.11.1431; KHANNA A, 1994, STEROIDS, V59, P4, DOI 10.1016-0039-128X(94)90037-X; Kim A H, 1996, Mol Hum Reprod, V2, P549, DOI 10.1093-molehr-2.8.549; MIHM M, 1994, J REPROD FERTIL, V102, P123; NOVERO RP, 1991, POULTRY SCI, V70, P2335; Ozawa M, 2005, REPRODUCTION, V129, P621, DOI 10.1530-rep.1.00456; RENSIS F. D., 2003, THERIOGENOLOGY, V6, P1139, DOI 10.1016-S0093-691X(03)00126-2; Roth Z, 2000, J REPROD FERTIL, V120, P83, DOI 10.1530-reprod-120.1.83; Roth Z, 2008, REPROD DOMEST ANIM, V43, P238, DOI 10.1111-j.1439-0531.2008.01168.x; Torres-Junior JRD, 2008, THERIOGENOLOGY, V69, P155, DOI 10.1016-j.theriogenology.2007.06.023; Wilson SJ, 1998, J DAIRY SCI, V81, P2132; Wolfenson D, 1997, ANIM REPROD SCI, V47, P9, DOI 10.1016-S0378-4320(96)01638-7; WOLFENSON D, 1995, BIOL REPROD, V52, P1106, DOI 10.1095-biolreprod52.5.1106; Ziskin MC, 2011, INT J HYPERTHER, V27, P374, DOI 10.3109-02656736.2011.5537690
