102,630 research outputs found
N. Gärde, T. Engströmer, T. Strandberg. E. Söderlund, Nya rättegângsbalken
N. Gärde, T. Engströmer, T. Strandberg. E. Söderlund, Nya rättegângsbalken. In: Revue internationale de droit comparé. Vol. 4 N°4, Octobre-décembre 1952. pp. 807-809
N. Gärde, T. Engströmer, T. Strandberg. E. Söderlund, Nya rättegângsbalken
N. Gärde, T. Engströmer, T. Strandberg. E. Söderlund, Nya rättegângsbalken. In: Revue internationale de droit comparé. Vol. 4 N°4, Octobre-décembre 1952. pp. 807-809
The microwave spectra of the deuteroammonias
"December 18, 1950."Bibliography: p. 16.Army Signal Corps Contract No. W36-039-sc-32037 Project No. 102B. Dept. of the Army Project No. 3-99-10-022.M.T. Weiss [and] M.W.P. Strandberg
Rotational absorption spectrum of OCS
"May 13, 1948."Bibliography: p. 17.Army Signal Corps Contract W-36-039 sc-32037.M.W.P. Strandberg, T. Wentink, Jr. [and] R.L. Kyhl
The microwave spectrum of carbonyl selenide
"June 21, 1948."Bibliography: p. [14]Army Signal Corps No. W-36-039-sc-32037.M.W.P. Strandberg, T. Wentink, Jr. [and] A.G. Hill
Letter, [Author unclear] to Paulina T. Merritt
Handwritten letter to Paulina Merritt from an unknown author, October 1, 1876.
Handwritten biographical information on Paulina T. McClung Merritt
A handwritten biography of Paulina T. McClung Merritt by an unknown author, 1892.
Heterogeneous and tissue-specific regulation of effector T cell responses by IFN-gamma during Plasmodium berghei ANKA infection.
IFN-γ and T cells are both required for the development of experimental cerebral malaria during Plasmodium berghei ANKA infection. Surprisingly, however, the role of IFN-γ in shaping the effector CD4(+) and CD8(+) T cell response during this infection has not been examined in detail. To address this, we have compared the effector T cell responses in wild-type and IFN-γ(-/-) mice during P. berghei ANKA infection. The expansion of splenic CD4(+) and CD8(+) T cells during P. berghei ANKA infection was unaffected by the absence of IFN-γ, but the contraction phase of the T cell response was significantly attenuated. Splenic T cell activation and effector function were essentially normal in IFN-γ(-/-) mice; however, the migration to, and accumulation of, effector CD4(+) and CD8(+) T cells in the lung, liver, and brain was altered in IFN-γ(-/-) mice. Interestingly, activation and accumulation of T cells in various nonlymphoid organs was differently affected by lack of IFN-γ, suggesting that IFN-γ influences T cell effector function to varying levels in different anatomical locations. Importantly, control of splenic T cell numbers during P. berghei ANKA infection depended on active IFN-γ-dependent environmental signals--leading to T cell apoptosis--rather than upon intrinsic alterations in T cell programming. To our knowledge, this is the first study to fully investigate the role of IFN-γ in modulating T cell function during P. berghei ANKA infection and reveals that IFN-γ is required for efficient contraction of the pool of activated T cells
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