100 research outputs found
Civil Aeronautics Board Control Over Domestic Routes and Competition, 1938-1952.
No full textPhDEconomicsUniversity of Michiganhttp://deepblue.lib.umich.edu/bitstream/2027.42/156651/1/0005666.pd
Im Spiegel der Tierwelt: Studien von Käthe Olshausen-Schönberger, II. Teil (cover: Neue Folge)
No full textSee my comments on the first, third, and fourth volumes of this lovely little collection, all under the same date of 1905? This volume starts off with a lively contrast between the official and non-official dining-out parties. The latter folks are having a much better time! Der wissenschaftlich-populäre Vortrag (17) comes too close to home; I feel as though I may have sometimes been that boring speaker! My top prize in this volume goes to Anschluss! (25). Actually it has a much fuller title, too long to copy here. It is about a telephone operator's ability to connect. Other outstanding illustrations include Mein Seliger! (7); Entsetzlich! Das einzig Stillose in uns'rer Wohnung ist mein Mann (9); Der Freier sitzt drin! (24); Das Rigorosum (26); and Die Schlange und das Kaninchen (31).Language note: GermanZweite AuflageKäthe Olshausen-Schönberge
201 ALTERATIONS IN CARDIAC SARCOPLASMIC RETICULUM FUNCTION IN A CONGESTIVE CARDIOMYOPATHY MODEL
No full textDifferential sensitivity of regional vascular beds in the dog to low-dose prostacyclin infusion
No full text The effect of low-dose prostacyclin (PGI2) infusion on abdominal organ, regional central nervous system, and regional myocardial blood flow distribution was studied in 14 open-chested, anesthetized dogs. Blood flow was measured using the radioactive microsphere technique and left atrial (LA) injection of 15-μm spheres. Continuous LA PGI2 infusion (25–35 ng/kg per minute) significantly (P < 0.05 to P < 0.005) increased renal cortical (+13%), splenic (+20%), small intestinal (+41%), large intestinal (+54%), fundic mucosal (+53%), and antral mucosal (+65%) blood flows whereas pancreatic, gallbladder, and hepatic (arterial) flow remained unchanged. Within the central nervous system blood flow increased in the medulla (+18%) and cerebral cortical gray matter (+17%) but was unchanged in the cervical spinal cord, pons, dorsal thalamus, cerebellum, caudate nucleus, and cerebral white matter. Atrial and ventricular myocardial blood flows and masseter muscle blood flow were unchanged during PGI2 administration. Thus blood flow changes are variable between and within regional vascular beds during low-dose PGI2 infusion in the dog. </jats:p
131 EFFECT OF DIGOXIN ON MORTALITY AND ON BLOOD FLOW DISTRIBUTION IN THE CARDIOMYOPATHIC TURKEY
No full text
Early outcome following arterial switch operation for infants presenting late with transposition of the great arteries with intact ventricular septum
No full textMyocardial perfusion abnormalities in carbon monoxide poisoned dogs
No full text The effect of carbon monoxide inhalation on the regional distribution of right and left ventricular myocardial blood flow was studied in 12 closed-chest anesthetized dogs. Dogs were exposed to a nonhypoxic mixture of oxygen (21–40%) and carbon monoxide (1.5–2.0%) for 10 min. Myocardial blood flow was measured (15-μm radionuclide-labeled spheres) during control conditions, and 10 and 60 min following discontinuation of carbon monoxide corresponding to carboxyhemoglobin levels (COHb) of 41.6 ± 2.8 and 26.5 ± 1.6% (mean ± SE), respectively. At a COHb level of 26.5%, right and left ventricular blood flows were increased to approximately 1.8–1.9 times the control values (1.06 ± 0.10 vs. 0.64 ± 0.08 mL/min per gram and 1.72 ± 0.12 vs. 0.91 ± 0.07 mL/min per gram, respectively, P < 0.002). At a COHb level of 41.6%, both right and left ventricular vascular beds were maximally or near maximally dilated as right ventricular and left ventricular myocardial blood flow values were increased approximately fivefold. The right and left ventricular subendocardial-subepicardial flow ratios were reduced at both COHb levels (P < 0.05). Thus, in addition to the global myocardial hypoxia that occurs following elevation of the COHb level, relative subendocardial underperfusion is a component of carbon monoxide poisoning in the intact dog. </jats:p
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