212,193 research outputs found
Entanglement entropy and D1-D5 geometries
No full texthttp://dx.doi.org/10.1103/PhysRevD.90.066004Giusto, Stefano, and Rodolfo Russo. "Entanglement Entropy and D1-D5 geometries." Physical Review D 90.6 (2014): 066004
Le antichità di Rodolfo Pio nel Palazzo in Campo Marzio.
No full textStoria della collezione di antichità del Cardinale Rodolfo Pio da Carpi, collocata nel palazzo di via de'Prefetti a Roma, con identificazione di nuovi elementi e note sulla dispersione
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A Christmas Card from Rodolfo Ramos to Dr. Hector P. Garcia
No full textA Christmas Card from Rodolfo Ramos to Dr. Hector P. Garci
ESCAMILLA M., Rodolfo (Gral.)
No full textCarta del Corl. Angel Camargo al Gral. PEC. Presenta al Corl. Rodolfo Escamilla M. para tratarle algunos asuntos (no especifica de que naturaleza). Recomendación del Gral. Lázaro Cárdenas ante el Gral. Joaquín Amaro, Secretario de Guerra y Marina para que se le confiera al Gral. Escamilla alguna comisión
Rodolfo Tanzi, l'ospedale e la società cittadina nei secoli XII e XIII
No full textIl saggio ricostruisce le origini e i primi sviluppi dell'ospedale Rodolfo Tanzi di Parma, i rapporti instaurati con le istituzioni e con la società locale, il modello di religiosità laicale proposto, per comprendere le ragioni che costituirono i felici presupposti per la duratura affermazione dell’ente nel sistema assistenziale cittadino
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A letter from Rodolfo Vega Rodriguez to Dr. Hector P. Garcia.
No full textA letter from Rodolfo Vega Rodriguez to Dr. Hector P. Garcia, asking for his help with his disability claim
Frontal executive function, apathy and behaviour in parkinson's disease
No full textThe pattern of cognitive deficits in Parkinson's disease is variable, but often includes executive impairment similar to that seen in patients with frontal lesions, as well as episodic memory impairment, visuospatial dysfunction and impaired verbal fluency. That could derive from the not irrelevant "new" role of the basal ganglia in determining cognitive process refinement. We discuss a huge number of pathological observations, starting from neuro-anatomical and biochemical networks, from basal ganglia to thalamus and cortical cortex
Freezing in-on and parkinson's disease: Cognition and behavior
No full textFreezing of gait (FOG) refers to transient episodes, usually lasting seconds, in which a patient is unable to initiate or continue locomotion, occurring on a background of relatively good ability to move and is best described by patients as "feet get glued to the ground." Freezing of gait is common in Parkinson's disease, with increasing prevalence as the disease progresses, but it has been commonly reported in pathologically proven progressive supranuclear palsy and vascular parkinsonism. Two types of freezing of gait have been recognized in patients affected by Parkinson's Disease, taking L-Dopa. The most common is an "off" -freezing of gait, which can be improved with L-Dopa or dopaminergic treatment, such as apomorphine. "Off" -freezing appears during an "off" state, when the patient is generally bradykinetic and rigid. In contrast, "on" -freezing is characterized by a worsening of symptoms as the dose of L-Dopa is increased, and by a general improvement, as the dose is decreased or, better said, modulated. The onfreezing of gait is related to abnormal execution of complex motor tasks such as repetitive, simultaneous, or sequential motor acts. Different Authors suggested that the primary underlying abnormality might be related to the inability to deliver, or hold a pre-programmed, continuous, and complex motor act, in response to an established and correct internal plan of action. Therefore, we hypothesized that PD- on freezing patients might be clinically well differentiated from the other clinical subtypes of PD and might present a specific cognitive impairment, different from that presented by PD patients, without on-FOG. We discuss these hypothesis, based on clinical studies and follow-up
Parkinson's disease as a model of basal ganglia disruption
No full textThe assumption that the PD neurodegenerative process begins in the dopaminergic substantia nigra has been seriously challenged by recent publications. New different studies have provided evidence in support of a lower brainstem origin, predating involvement of the nigra. In addition to the prominent loss of nigro-striatal dopamine neurons, there is also degeneration of nor-epinephrine neurons in the locus coeruleus, cholinergic neurons in the nucleus basalis of Meynert, the dorsal motor nucleus of the vagus, and involvement of the spinal cord and peripheral autonomic system. There is dysfunction of multiple neurotransmitter systems and research is underway to determine if therapeutic restoration of these transmitters can provide benefit in PD. We discuss these biochemical and pathological aspects inside the most classic clinical assumption of PD as a dopaminergic depletion-induced diseas
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A letter from Rose Sanchez Bryant and Rodolfo Cantu to Dr. Hector P. Garcia.
No full textA letter from Rose Sanchez Bryant, Media Coordinator; and Rodolfo Cantu, Executive Director of P.A.D.R.E.S.; to Dr. Hector P. Garcia, thanking him for his participation in a recent rally
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