27 research outputs found
Square micro-magnets arrays.
<p>Plan-view (a) optical and (b) magneto-optical images of square micro-magnets (100×100 µm<sup>2</sup>) separated by 100 µm. In the latter case, a uniaxial Magneto-Optic Indicator Film placed upon the micro-magnet array serves to reveal the direction of the z-component of the stray field pattern produced above the micro-magnet array, following out-of-plane magnetization in a magnetic field. Side-view of micro-magnets (c).</p
Characteristic Features of Major BRD Pathogens.
<p>Characteristic Features of Major BRD Pathogens.</p
Time course of the mean clinical scores for steers infected with viral pathogens and mock infected controls.
<p>Infection with (A) BRSV occurred on day 0 and clinical sign scores are presented for the next seven days. Mean (n = 4) scores were significantly different for both treatment versus day effect and for treatment versus mock infected controls (p<0.005); Bonferroni post hoc test p<0.05. (B) IBR infection occurred on day 0 and mean clinical sign scores are presented for the next six days. Mean (n = 4) scores were highly significantly different from mock infected controls; and treatment versus day effect was significant (Bonferroni test p<0.05). (C) BVDV infection on day 0 was followed until day 15 and mean clinical scores are presented for infected and mock infected controls. The effect of infection (p = 0.0013) and day (p = 0.0071) were significant. Mean clinical sign scores were significantly different for infected and control steers only on day 8 (Bonferroni p<0.058).</p
Histopathology of the lung from <i>Mycoplasma bovis</i> infected steer.
<p>(A) necrosuppuratve bronchiolitis with peribronchiolar mixed leukocytic infiltrate (arrow), (B) Immunohistochemistry of lesion demonstrating positive staining for <i>Mycoplasma bovis</i> in the bronchiolar luminal exudate (brown color and arrow).</p
Scoring system for Clinical Signs.
<p>*0–3 indicates magnitude, then x indicated weight factor</p><p>Scoring system for Clinical Signs.</p
Bacterial isolations from pharyngeal swabs.
<p>To evaluate the presence of the BRDC bacterial pathogens in the posterior pharynx of steers before and after infection, sheathed swabs were taken at intervals and at necropsy. The number of steers from each pathogen infection group (x axis) that had each of the bacterial pathogens isolated from the upper respiratory tract either during infection or at necropsy is shown (y axis).</p
Numbers of Up- and Down-Regulated Differentially Expressed Genes and Isoforms for each Challenge Group in Contrast to Controls.
<p>Numbers of Up- and Down-Regulated Differentially Expressed Genes and Isoforms for each Challenge Group in Contrast to Controls.</p
Pathogens, doses, and animals.
<p>NA = not applicable.</p><p><sup>1</sup>The pilot study was conducted in summer 2011. The optimized study was conducted in summer 2012.</p><p><sup>2</sup>California Animal Health and Food Safety Laboratory, clinical isolate.</p><p><sup>3</sup>Euthanized early, tissues not sampled.</p><p><sup>4</sup>For each pathogen in the optimized study there were 4 control steers for clinical sign analysis (a total of 24 steers); these were not necropsied. Tissue samples and pathology data was obtained from 3 additional numbered viral controls and 3 additional bacterial controls.</p><p>Pathogens, doses, and animals.</p
Heatmap showing the Jensen–Shannon (JS) divergence between challenge groups estimated from FPKM values for all genes.
<p>Heatmap showing the Jensen–Shannon (JS) divergence between challenge groups estimated from FPKM values for all genes.</p
Differentially Expressed Genes with the Largest Expression Variances by Challenge Group.
<p>Differentially Expressed Genes with the Largest Expression Variances by Challenge Group.</p
