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CSF and serum IgG and albumin in schizophrenics
No full textCSF and serum concentrations of IgG and albumin were determined by single radial immunodiffusion in 25 schizophrenics, 22 endogenously depressed subjects and 37 controls. No significant difference of the mean values was found among the 3 groups but, considering the homogeneity of variance, IgG and albumin concentrations of CSF were distributed less homogeneously in the schizophrenics than in the other groups. This was attributed to the behaviour of a relatively small number of cases. The possible meaning of this finding are discussed, also taking into consideration the hitherto conflicting data from the literature
[The general intelligence factor: psychometric study. Approach to the problem of mental deterioration].
No full textThe authors have measured the correlation coefficient between W.B. and P.M. scores in 80 normal subjects, comparable for age and school. A mutual relationship between the two tests, connected by the hypothetical general intelligence factor "g", was really demonstrated. Correlation coefficient was positive and significative between W.B. total scores, excepted verbal scores, and P.M., the highest one between subtests 8, 9 and P.M. The intra-tests correlation between W.B. total scores and each subtest score was also evaluated: between sub-tests 8, 9 and W.B. total scores the highest one again. A new qualitative interpretation of mental deterioration in clinical neurology and psychiatry is suggested on the base of these results
[Neuropsychological aspects of schizophrenic mental deterioration].
No full textThe authors have administered a test of intelligence (WAIS) and a test of attention (Color Naming) to a group of patients affected by chronic schizophrenic impairment. The scores give shape to a picture of mental deterioration, especially characterized by a decrease of attentive power. This results validates the possibility of a neuropsychological approach to the etiopatogenesis of schizophrenia and supports the hypothesis that an impairment of mental synthesis power may explain both clinical and psychometric features of the disease
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