682 research outputs found
The Making of a Book: The Debby Atwell Project: A Lessonn in Stewardship
No full textVideo accompanying the book, Many Friends, by the children of Peaks Island Elementary School, under the guidance of author/illustrator Debby Atwell. Videography by Don Perry. Music by Peaks Island musicians. Spring 2001.This bookmaking project and the videotaping were funded by a grant from MBNA to the Peaks Island Branch of the Portland Public Library with additional support from Peaks Island Lions Club, Peaks Island School, Peaks Island School PTA, and the Friends of the Peaks Island Library.Running time: 20 minutes.https://digitalcommons.portlandlibrary.com/peaks_vhs/1001/thumbnail.jp
Control of familial and renal cardiac diseases in English bull terriers: How to repair a damaged breed?
No full textControl recommendations are presented for four genetic or familial diseases that cause significant morbidity and mortality in affected English Bull Terriers. Bull Terrier polycystic kidney disease is an autosomal dominant disease diagnosed by detecting a minimum of three renal cysts, with cysts present in both kidneys, and similarly affected family members to confirm the inherited nature of the cysts. Bull Terrier hereditary nephritis is an autosomal dominant disease diagnosed in otherwise normal animals with urinary protein: creatinine ratios persistently >0.3 and no significant urinary sediment, a family history of the disease, and characteristic glomerular basement membrane lesions. Mitral valve myxomatous degeneration and left ventricular outflow tract obstruction in Bull Terriers are familial diseases diagnosed by auscultating characteristic murmurs in affected animals. Excluding animals with these clinical signs from the breeding pool will reduce the prevalence rates of these diseases, however maintenance of an effective population size is also important. Providing breeders with information on genetics, including the risks associated with inbreeding and the benefits of outcrossing, is likely to improve canine breeding practices, thus increasing fitness and fecundity of these purebred dogs
Memorial Sketches of Rev. George B. Atwell
No full textThe Rev. George B. Atwell was a Baptist minister in Connecticut until his death in 1879. There is a frontispiece of him at the age of 85. His serious look sets him a long way from Aesop playing with kids in the street! This book is composed of fifteen chapters written by his children as biographical sketches of periods of his life. During his pastoral duty, he wrote a series of original fables, a few of which were published. He meant to title the collection Pearls for the Poor, Contained in Proverbs and Parables, in Which Fact Is Drawn from Fable. (In fact, Pearls for the Poor still is at the bottom of the cover of this book.) Chapter X, Pearls, offers a good selection of these fables (89-126). Some of the fable subjects include the wrangling of vowels and consonants (90), an argument between the sword and the plough (94), the refusal to share of a man who has stood on another's shoulders to get grapes (95), and an argument between snow and ice (98). The latter takes a good turn when the sun dissolves both, and they find themselves becoming one. There is something humorous about The Short Man and Long Shadow (93). A man sensitive about his height notices at dawn that he now casts a long shadow. Wondering whether the shadow reflects reality, he decides to wait until sunset to check again whether his shadow has become long. Atwell enjoys punning, as when the river tells the fire that he has two banks, from which the fire will receive a check for all that is demanded of him. A turn of the screw worthy of the fable tradition occurs when a hen being sued for custody of her children has a fox as a lawyer, who pleads brilliantly and then demands the children as his fee (109). Atwell likes to pun and play with letters and sounds as when U and I square off in Woman's Rights (116).This is a hardbound book (hard cover)Told by John Walle
Some factors affecting diagnostic accuracy
No full textDiagnosis involves a complex and overlapping series of steps, each of which may be a source of error and of variability between clinicians. This variation may involve the ability to elicit relevant information from the client or animal, in the accuracy, objectivity and completeness of relevant memory stores, and in psychological attributes including tolerance for uncertainty and willingness to engage in constructive self-criticism. The diagnostic acumen of an individual clinician may not be constant, varying with external and personal factors, with different clients and cases, and with the use made of tests. In relation to clients, variations may occur in the ability to gain their confidence, to ask appropriate questions and to evaluate accurately both verbal and nonverbal responses. Tests may introduce problems of accuracy, validity, sensitivity, specificity, interpretation and general appropriateness for the case. Continuing effectiveness as a diagnostician therefore requires constant attention to the maintenance of adequate and up-to-date skills and knowledge relating to the animals and their diseases and to tests, and of sensitive interpersonal skills
Possible reasons for variation in Ixodes holocyclus toxicity
No full textTick paralysis caused by Ixodes holocyclus affects an estimated 20,000 domestic animals each year along the eastern coast of Australia (Stone 1988). Animals are presented with clinical signs ranging from mild paresis to ascending flaccid paralysis and varying degrees of respiratory and cardiac compromise. Mortality rates are significantly increased in animals presented with respiratory compromise compared to those animals without respiratory compromise, regardless of the degree of flaccid paralysis (Atwell et al 2001). Anecdotal evidence of ticks (collected from different sites in northern New South Wales) causing different clinical signs and mortality in hyper-immune dogs used for serum production, suggests that there may be a variation in toxin production and toxin content between individual ticks (Warne, N 2002 pers comm). This literature review suggests that two possible contributing factors to toxin variation may be the genetic variation within the I. holocyclus species and the variation in the host's response to tick feeding
[3]Ferrocenophanes with a tetramethyldisiloxane bridge: synthesis and molecular structure
No full textSiemeling U, Krallmann R, Jutzi P, Neumann B, Stammler H-G. [3]Ferrocenophanes with a tetramethyldisiloxane bridge: synthesis and molecular structure. Monatshefte für Chemie. 1994;125(5):579-586.6,6,8,8-Tetramethyl-7-oxa-6,8-disila[3]-ferrocenophan 2 entsteht aus dem Di(alkoxysilyl)ferrocen (H4C5SiMe2OR)2Fe (R=CH2CH2OCH2CH2OCH2CH2OMe) durch Hydrolyse und anschließende intramolekulare Disiloxan-Bildung. 2,2',3,3',4,4',5,5',6,6,8,8-Dodecamethyl-7-oxa-6,8-disila[3]ferrocenophan 3 wurde durch Luftoxidation von 2,2',3,3',4,4',5,5',6,6,7,7-Dodecamethyl-6,7-disila[2]ferrocenophan erhalten. Die Kristallstrukturen beider Verbindungen wurden durch Einkristall-Röntgenstrukturanalyse bestimmt (2:a=8.5330(10), b=15.610(3), c=18.774(5)Å, [alpha]=70.68(2), [beta]=77.94(2), [gamma]=75.150(10)°, V=2259.8(8)Å3, Z=6, Raumgruppe P1, R=0.045, Rw =0.044; 3:a=12.388(3), b=9.924(3), c=19.136(10)Å, [beta]=105.11(3)°, V=2271.2(15)Å3, Z=4, Raumgruppe P21/c, R=0.076, Rw =0.060). Wegen der Flexibilität der Disiloxan-Brücke sind 2 und 3 ungespannte Moleküle
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