1,721,047 research outputs found
Transesophageal Echo-Doppler for Study of Coronary Flow Reserve: Feasibility and Normalcy Criteria
No full textS-T Depression on Effort is Related to Heart Rate Response to Exercise in Patients with Essential Hypertension and no Coronary Artery Disease
No full textValutazione Regionale del Rilasciamento Ventricolare Sinistro Mediante Quantificazione Acustica in Codice di Colore
No full textExtravascular lung water: from genes to telemedicine
No full textExtravascular lung water is a crucial parameter for the management of many different pathological conditions, especially heart failure. Many modalities can be employed for its evaluation, each with different advantages and limitations. In a near future, we hope that extravascular lung water assessment could be performed by non-invasive wearable devices, allowing remote continuous monitoring of pulmonary congestion. Recently, it has been proposed that some specific genetic variations may be responsible for the heterogeneous individual response to the development of extravascular lung water. These data could be relevant to titrate and personalize both treatments and preventive interventions
The Coexistence of Myocardial Hypertrophy and Coronary Stenosis Has Additive Effect on Minimum Coronary Resistance in Man
No full textMechanisms of coronary flow reserve impairment in human hypertension - An integrated approach by transthoracic and transesophageal echocardiography
No full textThe purpose of this study was to investigate the different mechanisms responsible for an impairment of coronary vasodilator capacity in hypertensive subjects by an integrated echocardiographic approach, including transesophageal Doppler echocardiography, which allows noninvasive monitoring of coronary flow velocity in the left anterior descending artery during pharmacological vasodilation. The study population consisted of 17 healthy control subjects and 33 hypertensive subjects: 10 without hypertrophy, 16 with mild to moderate hypertrophy, and 7 with severe left ventricular hypertrophy. Mean systolic and diastolic flow velocities were monitored basally (together with indexes of myocardial oxygen demand, such as heart rate, myocardial inotropism, and left Ventricular wall stress) and during infusion of low-dose (0.56 mg/kg per 4 minutes) and high-dose (0.84 mg/kg per 9 minutes) dipyridamole. Coronary reserve was assessed as the ratio of mean diastolic velocity after high-dose dipyridamole and basal diastolic velocity, and minimum coronary resistance as the ratio of diastolic blood pressure and diastolic velocity after high-dose dipyridamole. Compared with the control group, in all hypertensive groups, coronary reserve was similarly decreased (3.54+/-0.84 versus 2.59+/-0.42, 2.29+/-0.46, and 2.43+/-0.71; P<.01) and minimum resistance increased (0.56+/-0.15 versus 0.75+/-0.31, 0.75+/-0.19, and 0.78+/-0.21 mm Hg . s(-1). cm(-1); P=NS). These results confirm that coronary reserve in hypertensive individuals is reduced even before the occurrence of left ventricular hypertrophy. The reduction in coronary reserve depends on both an increase in resting coronary flow and an impairment in maximal vasodilator capacity. An increase in resting flow is dependent on higher heart rate and wall stress in hypertensive subjects without ventricular hypertrophy and on increased myocardial mass in hypertensive subjects with hypertrophy. Hypertensive subjects with ventricular hypertrophy also demonstrated a significantly blunted response to low-dose dipyridamole
Digital Stress Echocardiography: Advantages in Terms of Diagnostic Accuracy and Echo-Lab Economy
No full textCoronary reserve in physiologic and pathologic left ventricular hypertrophy: a study by TEE Doppler
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Assessment of Coronary Flow Reserve by Transesophageal Doppler: Different Response to Adenosine or Dipyridamole
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