1,720,984 research outputs found
New perspectives in the treatment of advanced heart failure and life-threatening ventricular tachyarrhythmias in Becker patients with dilated cardiomyopathy
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P dispersion: a new electrocardiographic parameter to evaluate the risk of atrial arrhythmias in Emery-Dreifuss muscular dystrophies
No full textEffects of growth hormone therapy in Becker patients with dilated cardiomyopathy. Preliminary results
No full textEvaluation of the effectiveness of the permanent biventricular pacing or implantable cardioverter-defibrillator in Becker patients with dilated cardiomyopathy and advanced heart failure
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Cardiomyopathies: diagnosis of types and stages
No full textPrimary cardiomyopathies have as dominant feature the involvement of heart muscle itself. They are not the result of other diseases and should be defined as diseases of heart muscle not consequent to disorders of other parts of the cardiovascular apparatus. Most of them are consequent to genetic defects and can be subdivided into three major groups: isolated, associated with skeletal muscle diseases, associated with neurological disorders. Primary cardiomyopathies show an evolution from mild to more severe stages. Four types of cardiomyopathies are classically described: dilated, hypertrophic, restrictive and arrhythmogenic. However, from a clinical point of view, it is possible to distinguish seven stages: pre-clinical, prevalently arrhythmogenic, prevalently pseudo-hypertrophic, spotty fibrotic, restrictive, dilated and refractory heart failure. In the course of their evolution, cardiomyopathies can shift from a clinical picture to another, consequently requiring frequent examinations of patients in order to adjust their treatment
ST-segment displacement in Duchenne muscular dystrophy: myocardial necrosis or apoptosis?
No full textAn electrocardiographic pattern resembling myocardial infarction is a rare condition in Duchenne muscular dystrophy. We report the case of a Duchenne boy, aged 12 years and 7 month, who, during a programmed examination, showed electrocardiographic signs of ST segment elevation, without symptoms usually accompanying myocardial infarction (chest pain, dyspnoea, sweating). The biological markers of myocardial damage became positive on the 2nd day and recovered on the 5th day. Clinical features of this uncommon pattern are described, with the retrospective evaluation of similar cases from personal records. The differential diagnosis between myocardial necrosis and apoptosis is discussed
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