1,720,988 research outputs found
ACUTE THROMBOSIS OF RENAL ARTERY: RESTORATION OF RENAL FUNCTION AFTER LATE REVASCULARIZATION
Manipulation of iron and energy status of cells as a tool fot triggering apoptosis. Key role for inactivation of mitochondrial ATP synthase
H2O2 induces apoptosis in heme-synthesizig erytrholeukemia cells with a mechanism indeoendent from nF-kB activation and mediated by decline in mitochondrial bioenergy
Inactivation of mitochondrial ATPsynthase through manipulation of iron status as a tool for triggering apoptosis
Endogenous pro-oxidant status and H2O2-mediated impairment of cell mitochondrial functions, ATP content and proliferative capacity
Down-regulation of the mitochondrial ATP synthase in Friend's erythroleukemia cells induced to differentiation
Different mechanisms of H2O2-dependent damage to mitochondrial F0F1ATPsynthase mediated by different intracellular iron chelates
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