28 research outputs found

    A 0.6in CRT as a miniature display device

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    Photocatalytic degradation of chlorinated propenes using TiO2

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    The photocatalytic degradation of chlorinated propenes using TiO2 was investigated by FTIR spectroscopy. The chlorinated propenes were degraded to HCl, CO2, CO, H2O, and HCOOH during UV irradiation. During the degradation of 3-chloro-1-propene, the concentrations of CO2, CO, and HCOOH increased just after starting the irradiation. The onset of the HCl formation was delayed. On the other hand, the onset of the HCOOH formation was delayed during the degradation of 1-chloro-1-propene. During the degradation of 2-chloro-1-propene, the rate of the HCOOH production was slower than that during the degradation of 3-chloro-1-propene although the HCl production was not delayed. These results indicated that HCOOH was produced by the degradation of the double-bonded carbon bonding to two H atoms during the initial stage. The chlorinated compounds were preferentially produced from the double-bonded carbon bonding to the Cl atom and rapidly degraded to HCl, CO2, and CO during the initial stage. The residual part was degraded in the latter steps. Furthermore, it is suggested that the Cl atom on one of the double-bonded C atoms of the propenes was transferred to the other C atom before the degradation. Consequently, the double-bonded carbon bonding to two H atoms in 2-chloro-1-propene was chlorinated, then degraded to HCl, CO2, and CO during the initial stage.ArticleRESEARCH ON CHEMICAL INTERMEDIATES. 41(10):7641-7654 (2015)journal articl

    Morphology of mitochondria and cell respiration,pt.2.

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    With the purpose to elucidate the relation between the enzyme activity and the morphology of mitochondria the author carried out histochemical and biochemical investigations of cytochrome oxidase and succinic dehydrogenase activities of liver cells obtained at various intervals after the oral administration of CCl4, to male rats. And the data were compared with those reported in the first report. In the normal liver histochemically demonstrable cytochrome c oxidase activity and succinic dehydrogenase activity can be seen in parenchymal cells. In both cases the cells lying in the peripheral area show a more intense activities than those in the central part of liver lobules. The activity of cytochrome c oxidase falls markedly 5 to 6 hours after the CCl4, administration, while the activity of succinic dehydrogenase is retained almost at normal level for about 20 hours. Quantitative estimation of the succinic dehydrogenase activity of tissue homogenate revealed a transient rise in the activity 90 minutes after the CCl4, administration, and thereafter the values have been kept in almost normal level by 20 hours though a gradually fall has been seen in this period with a marked degree at 22nd hour. Taking the changes of minute structure occurring at each stage into consideration, which have been reported in the previous paper, the author concludes that the activity of succinic dehydrogenase is closely correlated with the maintenance of double membraneous structure of mitochondria, but the activity of cytochrome c oxidase is reduced by the swelling of mitochondria.</p

    Morphology of mitochondria and cell respiration,pt.1.

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    To reveal the mechanism of liver damage by taking CCl4 the author observed the liver tissues from rats at 1.5, 5, 6, 10, 17, 20, and 22 hours after the CCl4 administration, both by light microscope and electron-microscope. 1. Light microscope observation revealed the swelling of liver cells in the carly stage, the appearance of centrolobular fatty degeneration, focal degeneration area and the appearance of balloon cells, with the circulatory disturbances in accompanying stages and hemorrhage in the later stage. 2. Electron-microscope observation revealed the swelling of mitochondria, appearance of the files of thin ER's in the early stage and the regeneration and degeneration of mitochondria with an increase of microbodies in number. Fat droplets are developed from small ones probably from some microbodies without correlation with mitochondria. 3. From these observations the author is of the opinion that CCl4 arrests the cells at first inducing the swelling of cells and their mitochondria, but later the degenerative changes will become severe being complicated by the anoxia which is induced by the circulatory disturbances caused by the compression of vessels with the swollen cells.</p

    Corrigendum to “The 2016 update of the International Study Group (ISGPF) definition and grading of postoperative pancreatic fistula: eleven years after.” Surgery 2017. Mar; 161 (3):584–591. Epub Dec 28, 2016 (Surgery (2017) 161(3) (584–591), (S0039606016307577), (10.1016/j.surg.2016.11.014))

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    The authors regret that the name of author Charles R. Vollmer MD is incorrect in the final published version. The correct name Charles Vollmer. The authors would like to apologise for any inconvenience caused. Below is the correct order of authors: Claudio Bassi, MDa, Giovanni Marchegiani, MDa, Christos Dervenis, MD,b, Micheal Sarr, MDc, Mohammad Abu Hilal, MDd, Mustapha Adham, MDe, Peter Allen, MDf, Roland Andersson, MDg, Horacio J. Asbun, MDh, Marc G. Besselink, MDi, Kevin Conlon, MDj, Marco Del Chiaro, MDk, Massimo Falconi, MDl, Laureano Fernandez-Cruz, MDm, Carlos Fernandez-del Castillo, MDn, Abe Fingerhut, MDo, Helmut Friess, MDp, Dirk J Gouma, MDi, Thilo Hackert, MDq, Jakob Izbicki, MDr, Keith D. Lillemoe, MDn, John P. Neoptolemos, MDs, Attila Olah, MDt, Richard Schulick, MDu, Shailesh V. Shrikhande, MDv, Tadahiro Takada, MDw, Kyoichi Takaori, MDx, William Traverso, MDy, Charles Vollmer, MDz, Christopher L. Wolfgang, MDaa, Charles J. Yeo, MDbb, Roberto Salvia, MDa, Marcus Buchler, MDq, from the International Study Group on Pancreatic Surgery (ISGPS
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