35 research outputs found
"Imeneo sotto il platano": un motivo bucolico in Claudiano (carm. min. 25)
No full textNelle scene del sonno di Venere e del dialogo tra Venere e Imeneo, l’Epitalamio
per Palladio e Celerina di Claudiano (carm. min. 25) si distingue dalla tradizione
epitalamica per l'atmosfera bucolica, evocata da una serie di riferimenti tematici
e formali al genere letterario. Le parole di Venere e Imeneo sembrano comunicare
un articolata riflessione dell'autore sulla poetica espressa nell'epitalamio: Imeneo
fungerebbe da portavoce di Claudiano, rivendicando la possibilità per il poeta di
adattare i toni bucolici alle esigenze encomiastiche del carme d occasione. È possibile
che lo sviluppo narrativo dell'episodio di Imeneo sia stato suggerito a Claudiano
dall'inno cletico del carme 61 di Catullo. Claudiano potrebbe aver sfruttato le
possibilità offerte dalla forma dell'epitalamio epico per fare dell'invocazione a Imeneo
un episodio organico al racconto.In the scenes of Venus’ sleep and of the dialogue between Venus and Hymenaeus, Claudian's
Epithalamium for Palladius and Celerina (carm. min. 25) stands out from the
epithalamic tradition for its bucolic atmosphere, which is summoned by several thematic
and stylistic references to the literary genre. The words of Venus and Hymenaeus seem to
communicate an articulate reflection of the author on the poetics of the epithalamium:
Hymenaeus may be acting as spokesman for Claudian, claiming the possibility for the poet
to adapt the bucolic modes to the encomiastic needs of the occasional poem. The narrative
development of Hymenaeus’ episode might have been suggested to Claudian by the cletic
hymn of Catullus 61. Claudian seems to have taken advantage of the possibilities offered
by the form of the epic epithalamium in order to make the invocation to Hymenaeus an
episode incorporated in the narrative
L'Insubria // consolata
No full textTitre uniforme : Agnesi, Maria Teresa (1720-1795). Compositeur. [L'Insubria consolata]L'auteur pris à la fin de la dédicace. - La date d'après la représentation : Milan, Théâtre ducal, 1766. - Deux p. de t.. - Dédicace à la princesse Maria Beatrice d'Este. - Rôles : Insubria (Ut 1), Imeneo (Ut 1), Genio estense (Ut 1), Genio aus[triaco] (Ut 4). - Vl 2, vla, bc, ob 2, tr 2, cor 2. - Illustrations et accolades ornées à la plume. - Rel. en parchemin vert recouvert d'un papier vert-bleu. - Pièce de titre au dos portant : "L'Insubria // consolata // cantata // Sra PinottiniPrésentation musicale : [Partition]Incipit : Quanto attender dovròAppartient à l’ensemble documentaire : RISM2Appartient à l’ensemble documentaire : RISMMssSérénades (musique vocale)Cantates profane
Author correction to Extracellular signal-regulated protein kinase mediates interleukin 17 (IL-17)-induced IL-8 secretion in Helicobacter pylori-infected human gastric epithelial cells [Infect. Immun. 72, 9, (2004) 5019-5026]
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Olive Tree Biophenols in Inflammatory Bowel Disease: When Bitter is Better
No full textThe current therapeutic scenario for inflammatory bowel diseases (IBD) involves aminosalicylates, corticosteroids, and immunomodulators, but concerns regarding their safety profiles and high costs heavily impact their widespread use. In recent years, the beneficial effects thatbiophenols—from fruit and vegetables—have on human health have been investigated. The antioxidant and anti-inflammatory properties of phenolic fraction, from olive leaves and fruits, have been suggested, and a potential application in gut inflammation has been supported by in vitro and IBD-animal models studies. In the present review, we first introduced the potential therapeutic role of olive tree biophenolsin chronic inflammatory disease. Then, we aimed to describe their most interesting application for gut inflammation, as the results of basic science studies and animal experimental models. Finally, the potential role of olive tree biophenols in the setting of human IBD is discussed
Normal Bone Mineral Density Associates with Duodenal Mucosa Healing in Adult Patients with Celiac Disease on a Gluten-Free Diet
No full textImpairment of bone mineral density (BMD) is frequent in celiac disease (CD) patients on a gluten-free diet (GFD). The normalization of intestinal mucosa is still difficult to predict. We aim to investigate the relationship between BMD and duodenal mucosa healing (DMH) in CD patients on a GFD. Sixty-four consecutive CD patients on a GFD were recruited. After a median period of a 6-year GFD (range 2–33 years), patients underwent repeat duodenal biopsy and dual-energy X-ray absorptiometry (DXA) scan. Twenty-four patients (38%) displayed normal and 40 (62%) low BMD, 47 (73%) DMH, and 17 (27%) duodenal mucosa lesions. All patients but one with normal BMD (23 of 24, 96%) showed DMH, while, among those with low BMD, 24 (60%) did and 16 (40%) did not. At multivariate analysis, being older (odds ratio (OR) 1.1, 95% confidence interval (CI) 1.03–1.18) and having diagnosis at an older age (OR 1.09, 95% CI 1.03–1.16) were associated with low BMD; in turn, having normal BMD was the only variable independently associated with DMH (OR 17.5, 95% CI 1.6–192). In older CD patients and with late onset disease, BMD recovery is not guaranteed, despite a GFD. A normal DXA scan identified CD patients with DMH; thus, it is a potential tool in planning endoscopic resampling
Helicobacter pylori and T Helper Cells: Mechanisms of Immune Escape and Tolerance
No full textHelicobacter pylori colonizes the gastric mucosa of at least half of the human population, causing a worldwide infection that appears in early childhood and if not treated, it can persist for life. The presence of symptoms and their severity depend on bacterial components, host susceptibility, and environmental factors, which allow H. pylori to switch between commensalism and pathogenicity. H. pylori-driven interactions with the host immune system underlie the persistence of the infection in humans, since the bacterium is able to interfere with the activity of innate and adaptive immune cells, reducing the inflammatory response in its favour. Gastritis due to H. pylori results from a complex interaction between several T cell subsets. In particular, H. pylori is known to induce a T helper (Th)1/Th17 cell response-driven gastritis, whose impaired modulation caused by the bacterium is thought to sustain the ongoing inflammatory condition and the unsuccessful clearing of the infection. In this review we discuss the current findings underlying the mechanisms implemented by H. pylori to alter the T helper lymphocyte proliferation, thus facilitating the development of chronic infections and allowing the survival of the bacterium in the human host
Enhanced Activation of Cyclooxygenase-2 Downregulates Th1 Signaling Pathway in Helicobacter pylori-infected Human Gastric Mucosa
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