1,721,007 research outputs found
"Forced Behavior" as epileptic seizure: Description of a patient with neuronal migration disorder
No full textForced thinking and behavior can be true epileptic phenomena. Forced thinking, characterized by abrupt intrusion of thoughts, could be produced by an epileptic discharge in frontal or temporal lobes Epileptic forced thinking appears distinct from obsessive thoughts and compulsive urges. A male patient, teacher of optic pathophysiology, from the age of 40 showed short (2-3 minutes) episodes, characterized by a compulsive urge to speak technically as he would in his job. At the age of 43 he showed two nocturnal generalized seizures. An EEG was performed and it showed intercritical and critical anomalies on the left anterior regions. A magnetic resonance (MR) showed the presence of a neuronal migration disorder (sub-ependimal cotrical nodular heterotopia). Soon after, a therapy with carbamazepine 800 mg/die was started and all types of paroxystical manifestations disappeared. This case is interesting and intriguing because of the overlapping of epilepsy, forced behavior and neuronal migration disorders. Considering the anatomic systems involved in epilepsy and obsessive-compulsive disorder, we will discuss the differences and the common features of both disturbances
An object oriented approach to biofeedback applications for disabled people
No full textBled, Sloveni
MODIFICAZIONI DELL'ATTIVITA INTERCRITICA DURANTE VEGLIA E SONNO NOTTURNO IN UN PZ CON EPILESSIA MIOCLONICA GIOVANILE DI JANZ DI TIPO SCOTOSENSIBILE-NON FOTOSENSIBILE
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Modificazione delle performances cognitive dopo assunzione di carbamazepina in soggetti normali ed in pazienti con epilessia temporale di nuova diagnosi
No full textEstimation of cortical activity for non invasive EEG recordings for Brain Computer Interface applications
No full textEffetti sul sonno notturno e sulla sonnolenza diurna del trattamento cronico con Lamotrigina
No full textLamotrigine (LTG) is a new antiepileptic drug, currently used in medically intractable patients. We studied nocturnal sleep and daytime somnolence in 13 patients undergoing add-on therapy with LTG for seizures resistant to common antiepileptic drugs (AEDs). LTG caused a significant increase of REM sleep and a significant decrease of SWS, of the number of stage shifts and of the number of entries in REM. No significant changes were observed in daytime sleepiness and cognitive functions after 3 months of treatment. The increased stability of nocturnal sleep could play an important role in the anticonvulsant effect of LTG
Selection of task-related cortical EEG sources for Barain-Computer Interface Applications.
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