1,721,277 research outputs found
Lettre de M. Albertini sur les fouilles d'Ampurias (Espagne)
No full textCagnat René. Lettre de M. Albertini sur les fouilles d'Ampurias (Espagne). In: Comptes rendus des séances de l'Académie des Inscriptions et Belles-Lettres, 53ᵉ année, N. 12, 1909. pp. 939-943
Une fresque de l'économie française : J.-M. Albertini, L'économie française
No full textSouriac René. Une fresque de l'économie française : J.-M. Albertini, L'économie française. In: Revue géographique des Pyrénées et du Sud-Ouest, tome 50, fascicule 1, 1979. Commerce. pp. 136-138
Note de M. Albertini directeur du service des antiquités de l'Algérie
No full textNote de M. Albertini directeur du service des antiquités de l'Algérie . In: Comptes rendus des séances de l'Académie des Inscriptions et Belles-Lettres, 68ᵉ année, N. 1, 1924. pp. 81-83
Note de M. Albertini directeur du service des antiquités de l'Algérie
No full textNote de M. Albertini directeur du service des antiquités de l'Algérie . In: Comptes rendus des séances de l'Académie des Inscriptions et Belles-Lettres, 68ᵉ année, N. 1, 1924. pp. 81-83
Ruolo inibitorio esercitato dal vago sull'attività elettromiografica del diaframma nel maiale neonato ed adulto
No full textIn anesthetized (Sodium Thiopental mg 15-25/kg i.v.), tracheostomized pigs, aging 2-90-180 days, was evaluated vagal mechanism regulating the time of the diaphragmatic inspiratory discharge. Inspiratory time was evaluated on flow trace (phasic TI) and on EMG of the costal and crural portion of diaphragm (TIEMG). Our results show that the time of the diaphragmatic EMG discharge reflects the output of the respiratory centers, modulated by volume/time dependent vagal inhibitory control. The inibitory vagal control is more marked in newborn (TIEMG vs TI-25%) than in adult pig (TIEMG vs TI-14%)
Effetti vascolari e respiratori della NG-Nitro-L-arginina metil estere (L-NAME), inibitore della NO sintasi
No full textTo evaluate if circulatory and respiratory effects due to Platelet Activating Factor (PAF) administration are modulated by releasing of endogenous nitric oxide (NO), in six anesthetized, paralyzed and mechanically ventilated Large White pigs L-NAME (10 mg/kg iv), an inhibitor of NO synthase, was used. PAF (50 ng/kg iv) in control conditions caused bronchoconstriction, evidenced by an increase in resistances and elastances of the respiratory system, pulmonary hypertension and a transient systemic hypotension. L-NAME, administered in control conditions, caused an increase in systemic and pulmonary arterial pressure and a modest increase in resistances and elastances of the respiratory system. After PAF, L-NAME caused strengthened bronchoconstriction and pulmonary hypertension, without modifying systemic hypotension. These results show that endogenous NO counterbalances respiratory and pulmonary vascular effects of PAF, while it is not involved in the systemic hypotension PAF-dependent
La meccanica respiratoria del maiale in funzione del flusso e del volume inspirato
No full textIn 6 Large White pigs were evaluated the changes of resistances and elastances of total respiratory system, of lung and chest wall as function of flow and volume, using the partitioning method. By this method is possible to distinguish the total resistances in flow resistance of airways (Rint) and in deltaR, espression of viscoelastic tissues property. The total respiratory resistances decrease increasing flow, while increase linearly with volume. The dynamic and static elastance is linear with volumes above o.o5 l
I vasodilatatori endoteliali controbilanciano l’azione dell’endotelina-1
No full textIn 10 anaesthetized and mechanically ventilate Large White pigs we analysed the activity of Endothelin-1 (ET-1) and the involvement of prostacyclin (PGI2) and nitric oxide (NO) as modulators of ET-1 activity. Parameters evaluated were: systemic (MAP) and pulmonary arterial pressure (MPAP), heart rate (HR), cardiac output (CO) and systemic (SVR) and pulmonary vascular resistances (PVR). Results showed that ET-1 (100 pmol/kg i.v.) induces a biphasic response both on pulmonary and systemic vascular bed. This was characterised by an early and transient hypotension, followed by an hypertensive activity lasting about 20'. Blocking NO by NG-nitro-L-arginine methyl ester (L-NAME 5 mg/kg i.v.), or PGI2 by indomethacin (3 mg/kg i.v.), the hypotensive activity ET-1-dependent on systemic vascular bed, at difference to pulmonary one, disappears. This data show that ET-1 at systemic level induces the release of NO and PGI2, while at pulmonary level its hypotensive activity is probably due to other vasoactive metabolites
Effetti dell’ossido d’azoto (NO) endogeno ed inalato in un modello sperimentale di ipertensione polmonare e broncocostrizione
No full textIn 12 anesthetized, paralyzed and mechanically ventilated pigs, the effects of both endogenous and inhaled nitric oxide (NO, 80 ppm for 6 min), have been evaluated in control conditions and after administration of 50 ng/kg i.v. of platelet activating factor (PAF). To study the effects of endogenous NO, NG-nitro-L-arginine methyl ester (L-NAME, 10 mg/kg i.v.), an inhibitor of NO synthesis, was used. In control conditions, the block of endogenous NO increased pulmonary arterial pressure (PAPM), without changing systemic arterial pressure (PAM) or resistances (Rrs) and compliance (Crs) of the respiratory system. PAF administration causes a pulmonary hypertension and a prompt and brief decrease in systemic arterial pressure. L-NAME administration strengthened the effects of PAF on Rrs, Crs and PAM, while caused a lesser increase in pulmonary arterial pressure. These data show that PAF-dependent systemic hypotension is not caused by endogenous NO and that PAF administration favors the release of NO, which counterbalances the respiratory effects. Inhaled NO, in all experimental conditions, did not modify PAM, while in control conditions caused a decrease in PAPM. Inhalation of NO counterbalanced the respiratory and circulatory effects caused by PAF administration
Valutazione del ruolo dell’ossido d’azoto sulla fatica diaframmatica nel suino
No full textIn 12 anaesthetized and mechanically ventilated Large White pigs, divided into two grous, we analyzed the effect of nitric oxide (NO) on the diaphragmatic fatigue induced by phrenic nerve stimulation. The first group was treated with NG-nitro-L-arginine methyl ester (L-NAME, 5 mg/kg i.v.) to block endogenous NO synthesis. The second group was treated with sodium nitroprusside (SNP, 0.57 mg/kg i.v.) as NO donor. L-NAME decreased the diaphragmatic contraction during endurance test (stimulation for 30 sec at 10 Hz and 15 V)and increased mean systemic arterial pressure (PAM) and vascular resistance (RVS). SNP also caused an impairment of diaphragmatic function, but this was associated with a decrease in PAM and RVS. Also if we can not exclude a NO effect on diaphragmatic activity, probably the decreased resistance to fatigue after phrenic nerve stimulation by us observed, is due to the reduction of diaphragmatic blood flow
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