3,290 research outputs found
Measurements of chlorophyll fluorescence reveal mechanisms for habitat niche separation of Thalassia hemprichii and Halodule univervis in intertidal zone.
No full textEpigenetic activation of alpha 4, beta 2 and beta 6 integrins involved in cell migration in trichostatin A-treated Hep3B cells
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Downregulation of BRG-1 repressed expression of CD44s in cervical neuroendocrine carcinoma and adenocarcinoma.
No full textThe biomarkers of human papillomavirus infection in tonsillar squamous cell carcinoma-molecular basis and predicting favorable outcome.
No full text[[alternative]]The KT transition of YBCO films
No full text[[abstract]]我們想藉由觀察 YBaCuO 薄膜是否有 KT 變遷的現象,來瞭解薄膜厚度與
維度的關係。故我們製作二塊不同厚度 (240o,120o) 的薄膜,分別量測它
們的電阻率與 V-I 曲線, 並加以分析。其中240o 之 YBCO 薄膜由於厚度
較大,無法表現二維的行為。而120o 的薄膜其 V-I 曲線相關的次方定律
V=I,在 T<76.6K 時, n 滿足二維 Ginzberg- Landau 之線性關係式: n(
T)≒1+const(1-T/Tco)。以此直線與 n=1 之交點定出樣品的 T=79K。而
在n(T)約為5.1時, n 不再隨上述的線性關係式隨溫度下降,亦即發生了
KT 變遷中普遍躍遷的現象。我們分別以 n=3及 n=5.1 定出兩種不同的
Tkt, 來討論電阻率的結果。由電阻率的結果分析,無論那種決定 Tkt 的
方法在介於78.1及78.6K 間,其ln(R/Rn)與[(Tco-T)/(T-Tkt)]成線性關係
。亦就是出現 KT 理論中預測因二維渦流運動產生的特殊行為。所以其具
有二維的傳導現象。
In order to know the dimensionalities of YBCO films, we
measured the resistivities and I-V curves of two YBCO films(120
o,240o). According to our results,the film of 240o thickness
couldn't show the 2d behaviors.The power law of 120o film has
the universal jump phenomena and the resisti- vity behavior can
be explained by 2d free vortices mo- tion.We can say that the
YBCO film of 120o thickness is a 2d system.
In order to know the dimensionalities of YBCO films, we
PROTEIN-PHOSPHORYLATION AT A POSTRECEPTOR SITE CAN BLOCK DESENSITIZATION AND INDUCE POTENTIATION OF SECRETION IN CHROMAFFIN CELLS
No full textDesensitization or habituation to repeated or prolonged stimulation is a common property of secretory cells. Phosphorylation of receptors mediates some desensitization processes, but the relationship of phosphorylation to desensitization at postreceptor sites is not well understood. We have tested the effect of protein phosphorylation on desensitization in bovine chromaffin cells. To increase protein phosphorylation, we have used the protein phosphatase inhibitor okadaic acid at 12.5 nM, 100 muM 8-bromo-cyclic AMP to activate protein kinase A, and 10 nM phorbol 12,13-dibutyrate to activate protein kinase C. During repeated 6-s stimulation at 5-min intervals, catecholamine secretion from control cells decreases. Cells exposed to 8-bromocyclic AMP or okadaic acid alone show slightly decreased rates of desensitization. In cells pretreated with phorbol 12,13-dibutyrate, desensitization is blocked. Okadaic acid-treated cells stimulated in the presence of 8-bromocyclic AMP show potentiation of secretion with repeated stimulation. The protein kinase inhibitor 1-(5-isoquinolinylsulfonyl)-2-methylpiperazine (H7) increases the desensitization rate. Because these phenomena are observed during secretion evoked with elevated K+ as well as by a nicotinic agonist, the effect of phosphorylation is at a postreceptor site. In contrast to desensitization to the repeated stimulations, desensitization to prolonged stimulation with high K+ is not altered by the above protocols in chromaffin cells.X114sciescopu
Antiplatelet effect and selective binding to cyclooxygenase by molecular docking analysis of 3-Alkylaminopropoxy-9, 10-anthraquinone derivatives.
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