97 research outputs found
Donadoni E., Velo L. G., Fabian L., 2015, “Ri-ciclare spazi e forme della mobilità” in Donadoni, Fabian, Munarin (a cura di) Re-cycle Veneto, Venezia: Aracne Editrice Srl, pp. 115-139 - ISBN 978-88-548-8642-1
No full textDonadoni E., Velo L. G., Fabian L., 2016 “Ri-Ciclare la mobilità in Veneto” Officina n° 10 gen-feb 2016, pp. 8-13 - ISSN 2384-9029
No full textDonadoni E., Fabian L., Munarin S. (a cura di), 2015, Re-cycle Veneto, Venezia: Aracne Editrice Srl - ISBN 978-88-548-8642-1
No full textDonadoni E., 2016 “BIKE (Bicycle Instruments, Knowledge and Enterprise). La mobilità urbana dopo il picco del petrolio” in L. Fabian, V. De Ferrario (a cura di), Veneto Futuri, Venezia: Mimesis, pp. 53-69 - ISBN 978-88-5753-814-3.
No full textDonadoni E., Velo L. G., Fabian L., 2013 “Be-Cycle: spazi e forme della mobilità tra ferro e acqua nella città diffusa” in Urbanistica Informazioni, VIII Giornata di Studi INU: Una politica per le città italiane”, n° 257, Roma: INU Edizioni, pp. 27- 32 - ISSN 0392-5005
No full textINTRASPINAL DEGENERATIVE ATROPHY CAUSED BY SCIATIC-NERVE LESIONS PREVENTED BY ACETYL-L-CARNITINE
No full textPeripheral nerve lesions cause retrograde changes in the spinal cord. involving initially the descending serotoninergic pathways and later the substance P sensory input and methionine-enkephalin interneurons. Within 48 h after sciatic nerve resection there is a significant increase of 5-hydroxyindoleacetic acid in the lumbar spinal cord with no changes of serotonin metabolism in the cell body areas. The immunocytochemical analysis of the spinal cord shows that 20 days after nerve lesion there is a loss of substance P-positive boutons in the laminae I and II of the dorsal horn in the lumbar segment, Such a morphological change is correlated by radioimmunoassay for substance P and methionine-enkephalin, that reveals a significant loss of both peptides. Treatment with acetyl-L-carnitine prevents the early 5-hydroxyindoleacetic acid increase and the reduction of peptide content observed 20 days after lesioning the sciatic nerve. These data suggest that treatment with acetyl-L-carnitine exerts a neuroprotective activity preventing the retrograde changes triggered by peripheral nerve lesions
PEPTIDE ALTERATIONS IN AUTONOMIC DIABETIC NEUROPATHY PREVENTED BY ACETYL-L-CARNITINE
No full textAutonomic neuropathy and gastrointestinal problems are among the most common complications of diabetes. In this report it is shown that a possible correlation between the two disorders might exist, since diabetes causes a profound alteration of the peptidergic innervation of the gut. It is reported that 14 weeks after diabetes induction with alloxan the levels of substance P and methionine-enkephalin are markedly reduced throughout the intestine, while vasoactive intestinal polypeptide content is dramatically increased. Therefore the enteric innervation of diabetic animals is completely disorganized, with some systems undergoing atrophy and others undergoing hypertrophy. Treatment of diabetic animals with acetyl-L-carnitine prevents the onset of the marked peptide changes described above. The results suggest a potential for acetyl-L-carnitine in the treatment of autonomic neuropathies
Deceptive Actions to Improve the Attribution of Rationality to Playing Robotic Agents
No full textPlay is a common activity providing not only pleasure but also physical and cognitive development. In the quest for new playing experiences, there is an increasing tendency to develop robots playing with people. Making believable playing robots able to keep human players engaged and satisfied by the playing experience is the main challenge. In this work, we investigate the possibilities of a playful interaction between a human player and a mobile robot. In particular, this paper focuses on the applicability of deception as a means to support engagement and the attribution of rationality to playing robotic agents. By analyzing the interaction situation between the human and robot players, by identifying the need for deception, and by deciding whether and how to deceive, we aim at increasing self-reported engagement and fun, which are also related to the perception of the robotic opponent as smart enough to compete at an appropriate level. Experiments were conducted on a sample of 78 subjects facing two different deceptive behaviors and a basic behavior without any deception. All participants responded to a post-interaction questionnaire from which it was possible to observe a positive acceptance of the perception of the robot as a rational agent aimed at winning. In general, deception was perceived by most of the players as one of the robot’s abilities, when actuated, and contributed to the reported fun
“Cooperazione e inclusione sociale al servizio del turismo lento: la rigenerazione territoriale secondo il progetto TWIN”, 2022, in Chiara Belingardi, Gabriella Esposito De Vita, Laura Lieto, Giusy Pappalardo, Laura Saija (a cura). Atti della XXIV Conferenza SIU, “Dare Valore ai valori in urbanistica ”, volume 5. Agire collettivo e rapporto tra attori nel governo del territorio, Brescia 23-24 giugno 2022,
No full textNITRIC OXIDE-SENSITIVE PROTEIN ADP-RIBOSYLATION IS ALTERED IN RAT DIABETIC NEUROPATHY
No full textEndogenous ADP-ribosylation of proteins was studied in retina crude extract, membrane and cytosolic fractions of control and diabetic rats. ADP-ribosyltransferase activity is present in all cellular fractions, but protein ADP-ribosylation is reduced in diabetic rat retina. At least 6 proteins are labelled in the crude extract fraction and a similar number in the membrane preparation of control animals. In these preparations from diabetic retina, only two bands were labelled, the 85 K and 36K for the crude extract, and the 97 K and 39 K for membranes. Labelling of 36 K and 39 K proteins was much less than in controls, In the cytosolic preparations of controls, two proteins of 85 K and 39 K are ADP-ribosylated, while in diabetic rat retina cytosol, only the 85 K is labelled. Treatment of diabetic rats with insulin normalized plasma glucose levels and prevented the alterations of the extent of ADP-ribosylation for the 38 K cytosolic, 39 K membrane and 36 K crude extracts proteins, but it failed to affect the other bands. These results suggest a hyperactivity of endogenous ADP-ribosylases in diabetic rat retina, so that the protein sites for ADP-ribosylation are no longer available. Since insulin treatment prevents the onset of neuropathy and of retinal G protein impairment (Abbracchio et al., J Neurosci Res 29:196-220, 1991) in diabetic rats and, in this study, normalizes ADP-ribosylation of 39 K, 38 K and 36 K proteins, we suggest that the abnormal endogenous ADP-ribosylation of these proteins might play a role in the onset of diabetic neuropathy. (C) 1995 Wiley-Liss, Inc
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