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    Incoltorrida magna Perkins & Bergsten 2019, new species

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    <i>Incoltorrida magna</i>, new species <p>Figs. 3, 4, 10 (habitus); 21 (genitalia); 36 (map); 40 (habitat)</p> <p> <b>Type Material.</b> Holotype (male): <b>Antsiranana</b>, Diana: Antsaba: Galoko mountains, 3.4 km NW from Anstaba, S13.60931 E48.72129, aspirator, forceps, sieves: hygropetric rocks and pools, elev. 296 m, 28 xi 2012, elev. 296 m, 28 xi 2012, J. Bergsten, R. Bukontaite, J.H. Randriamihaja & T. Ranarilalatiana (MAD12-31) (NHRS). Paratypes (36): Same data as holotype (1 NHRS); <b>Antsiranana</b>, Anjiabe Ambony: Ambilobe: Antsabe stairways-like cascade with vertical (!) steps, exposed, extremely hot day, N: -13.60930 E: 48.72120, elev. 303 m, 23 xi 2004, Balke <i>et al</i>. (P25 MD16) (35 BMNH, NHRS, MCZ, PBZT / MBC; 2 DNA extractions, #’s BMNH 670732, BMNH 670733).</p> <p> <b>Differential Diagnosis.</b> This is the largest known <i>Incoltorrida</i> species: l/w ca. 2.62/1.58. The elytral serial punctures are very distinct, as is the transverse ridge that links costae #5 and #8. The pronotum lacks the basal ridges seen in <i>I. quintacostata</i>, <i>I. benesculpta</i>, and <i>I. madagassica</i>. It is much larger than <i>I. marojejy</i> (2.62 vs. 2.19); <i>I. marojejy</i> lacks the pronotal midlongitudinal carina that is present in <i>I. magna</i>, and the frons plate is shaped differently in the two species. The male genitalia (Fig. 21) distinctively differ from that of the other species in the genus. See also the diagnosis of <i>I. madagassica</i>.</p> <p> <b>Description</b>. Size: holotype (length/width, mm): body (length to elytral apices) 2.62/1.58; head width 0.50; pronotum 0.55/1.12; elytra 1.78/1.58. Dorsum dark brown to black, venter brown to reddish brown, legs brown to reddish brown except tarsi and femoral-tibial articulations black.</p> <p>Frontoclypeal shield narrowest at anterior 1/3, with sides of frons slightly arcuate; apical 1/3 is slightly wider than area of frontoclypeal suture.</p> <p>Short oblique carinae on anterior 1/3 of pronotum are well developed; a midlongitudinal carina is present, though not as developed as the oblique carinae; the area posterior to the carinae is transversely rounded and shows no indication of ridges or depressions, except for a basal impression in front of scutellum.</p> <p>Elytra quite convex, declivity steep. Each elytron with eight wide and high costae; #4 continuous, not interrupted by punctures; #5 weakly bisinuate; #6 and #7 distinct, though crossed by transverse ridge that connects #5 and #8. Second transverse ridge links #3 and #5, in nearly same plane as the #5-#8 ridge.</p> <p>Posterior 1/2 of metaventral tabella without distinctive, closely spaced transverse grooves; midlongitudinal groove in posterior 1/2 of metaventral tabella narrow and parallel-sided.</p> <p>Midlongitudinal carina of first abdominal ventrite not especially strong and extends only 1/2 length of ventrite.</p> <p>Male genitalia in lateral view distinctively arcuate; in ventral view widest at about apical 1/4, and then narrowed, with tips pointing slightly outward (Fig. 21).</p> <p> <b>Etymology</b>. Named in reference to the relatively large size.</p> <p> <b>Remarks</b>. DNA sequences of partial CO1 (Acc. Nos. FJ819703 (BMNH 670732) and FJ819702 (BMNH 670733)) and partial 28S (FJ 818159 (BMNH 670732) and FJ818158 (BMNH 670733)) published by Monaghan <i>et al.</i> (2009) are based on adult specimens, and are available in Genbank.</p>Published as part of <i>Perkins, Philip D. & Bergsten, Johannes, 2019, New Myxophagan water beetles from Madagascar (Coleoptera: Torridincolidae, Hydroscaphidae), pp. 57-96 in Zootaxa 4657 (1)</i> on page 76, DOI: 10.11646/zootaxa.4657.1.2, <a href="http://zenodo.org/record/3369801">http://zenodo.org/record/3369801</a&gt
    ZENODO

    Mitochondrial metabolism reveals a functional architecture in intact islets of Langerhans from normal and diabetic Psammomys obesus

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    The cells within the intact islet of Langerhans function as a metabolic syncytium, secreting insulin in a coordinated and oscillatory manner in response to external fuel. With increased glucose, the oscillatory amplitude is enhanced, leading to the hypothesis that cells within the islet are secreting with greater synchronization. Consequently, non-insulin-dependent diabetes mellitus (NIDDM; type 2 diabetes)-induced irregularities in insulin secretion oscillations may be attributed to decreased intercellular coordination. The purpose of the present study was to determine whether the degree of metabolic coordination within the intact islet was enhanced by increased glucose and compromised by NIDDM. Experiments were performed with isolated islets from normal and diabetic Psammomys obesus. Using confocal microscopy and the mitochondrial potentiometric dye rhodamine 123, we measured mitochondrial membrane potential oscillations in individual cells within intact islets. When mitochondrial membrane potential was averaged from all the cells in a single islet, the resultant waveform demonstrated clear sinusoidal oscillations. Cells within islets were heterogeneous in terms of cellular synchronicity (similarity in phase and period), sinusoidal regularity, and frequency of oscillation. Cells within normal islets oscillated with greater synchronicity compared with cells within diabetic islets. The range of oscillatory frequencies was unchanged by glucose or diabetes. Cells within diabetic (but not normal) islets increased oscillatory regularity in response to glucose. These data support the hypothesis that glucose enhances metabolic coupling in normal islets and that the dampening of oscillatory insulin secretion in NIDDM may result from disrupted metabolic coupling. <br/
    Southampton (e-Prints Soton)

    Molecular studies of complications in end stage renal disease : focus on expression and variations of candidate susceptibility genes

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    End stage renal disease (ESRD) is a complex multifactorial disorder, where both environmental and genetic factors are contributing to the phenotype. The most common complications seen in ESRD patients are malnutrition, inflammation and atherosclerosis and the coexistence of these complications is denoted the MIA syndrome, which is associated with inexplicably high mortality. However, due to big inter-individual differences not all patients have the same risk of developing the different complications of the MIAsyndrome.The objective of this thesis is to contribute to the development of an approach on how to use genetics as an instrument for identifying "high risk" ESRD patients at an early stage and to provide the tools that can help to develop more accurate and individually tailored treatment strategies in the future. With this aim, several susceptibility genes and proteins have been investigated with relation to the MIA syndrome and mortality in ESRD. They are presented in the following papers:Paper I; Myeloperoxidase (MPO) is suggested to be one link between inflammation, oxidative stress and endothelial dysfunction in uremic patients. The effect of a functional single nucleotide polymorphism (SNP) -463 G/A, that induces a 25-old transcription enhancement, was analyzed in relation to the MIA complications and oxidative stress in ESRD patients. The G/G genotype was associated with higher prevalence of inflammation and CVD, as well as with higher levels of an oxidative stress marker, a finding that replicates other studies.Paper II; The role of the anti-atherosclerotic protein adiponectin was studied in ESRD and the impact of variations of the adiponectin gene (ApM 1) on plasma adiponectin levels was investigated. In this study we found that plasma levels of adiponectin were markedly increased in ESRD patients. Additionally, patients with insulin dependent diabetes mellitus (DM) had even higher levels as compared to both non diabetic and non insulin dependent DM patients. Among the four SNPs that were studied (-11391G/A, -11377C/Q 45T/G, 276G/T) only the - 1377 C/C genotype was significantly associated with a lower prevalence of CVD as well as lower triglyceride levels. The low impact of the individual SNPs on plasma levels and presence of the MIA-complications shows the importance of analyzing and identifying risk-haplotypes that may have greater impact on the phenotype.Paper III; Since adiponectin levels are markedly elevated in ESRD, we analyzed the expression of ApM1 in fat tissue from patients with a high prevalence of MIA complications, patients who had few complications and healthy matched controls. We found that the ApM1 gene expression was markedly decreased in ESRD patients as compared to controls, but no significant difference was noted between the two groups of patients. The decreased ApM1 expression could be explained by a negative feedback regulation due to the high levels of circulating adiponectin.Paper IV; Plasma levels of interleukine-6 (IL-6) and C-reactive protein (CRP) are highly elevated in ESRD patients and predict mortality in both healthy and uremic individuals. Hence putative SNPs that may regulate plasma levels of these proteins were investigated (IL-6 SNPs: -597G/A, -174G/C, 5014A/G, Phe201Phe C/T and CRP SNPs: -286C/T/A, 1059G/C). We found that a genotype combination (-597G, 174G, 5014A) was associated with inflammation in ESRD patients. However, the lack evident impact on plasma levels, which is seen in other studies, may be explained by the strong effect of a promoter haplotype. Hence, further studies are necessary to evaluate how genotype combinations regulate the plasma levels of IL6 and CRP.Paper V; Resistin is a newly discovered protein that in rodents may promote insulin resistance. Resistin may have a direct proinflammatory effect on vascular endothelial cells, which could enhance the development of atherosclerosis. The role of resistin on insulin resistance was investigated in ESRD patients as well as the impact of a functional -180 C/G SNP on resistin plasma levels. We found that circulating levels of resistin were highly elevated in patients and that plasma levels correlated with glomerual filtrations rate. However, resistin did not seem to be associated with insulin resistance. Plasma resistin was associated with both adhesion molecules and inflammatory markers. Our findings suggest that resistin plasma levels should always be corrected for GFR in all study populations.The recent development in the field of genetics has made it possible to understand the impact of genotype in disease development and progress. It seems conceivable that in near future, prognostic or predictive multigene DNA assays will provide the nephrological community with a more precise approach for the identification of "high risk" ESRD patients and the development of accurate individual treatment strategies.List of scientific papersI. Pecoits-Filho R, Stenvinkel P, Marchlewska A, Heimburger O, Barany P, Hoff CM, Holmes CJ, Suliman M, Lindholm B, Schalling M, Nordfors L (2003). A functional variant of the myeloperoxidase gene is associated with cardiovascular disease in end-stage renal disease patients. Kidney Int Suppl. 84: S172-6. https://pubmed.ncbi.nlm.nih.gov/12694338II. Stenvinkel P, Marchlewska A, Pecoits-Filho R, Heimburger O, Zhang Z, Hoff C, Holmes C, Axelsson J, Arvidsson S, Schalling M, Barany P, Lindholm B, Nordfors L (2004). Adiponectin in renal disease: relationship to phenotype and genetic variation in the gene encoding adiponectin. Kidney Int. 65(1): 274-81. https://pubmed.ncbi.nlm.nih.gov/14675060III. Marchlewska A, Stenvinkel P, Lindholm B, Danielsson A, Pecoits-Filho R, Lonnqvist F, Schalling M, Heimburger O, Nordfors L (2004). Reduced gene expression of adiponectin in fat tissue from patients with end-stage renal disease. Kidney Int. 66(1): 46-50. https://pubmed.ncbi.nlm.nih.gov/15200411IV. Bergsten A, Stenvinkel P, Kockum I, Pecoits.Filho R, Barany P, Hoff C, Holmes C, Jonsson E, Lindholm B, Nordfors L, Heimburger O (2005). Genetic influence on inflammatory biomarkers in ESRD patients: a study of CRP and IL-6 single nucleotide polymorphisms. [Manuscript]V. Axelsson J, Bergsten A, Heimburger O, Barany P, Lonnqvist F, Lindholm B, Nordfors L, Alvestrand A, Stenvinkel P (2005). Elevated resistin levels in chronic kidney disease are associated with decreased glomerular filtration rate and inflammation, but not with insulin resistance. [Manuscript]</p
    KI Open Archive Karolinska Institutet

    Incoltorrida madagassica Steffan 1973

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    &lt;i&gt;Incoltorrida madagassica&lt;/i&gt; Steffan &lt;p&gt;Figs. 5, 6 (habitus); 14, 15, 19 (genitalia); 38 (map); 48, 49 (habitat)&lt;/p&gt; &lt;p&gt; &lt;i&gt;Incoltorrida madagassica&lt;/i&gt; Steffan 1973: 635.&lt;/p&gt; &lt;p&gt; &lt;b&gt;Type material:&lt;/b&gt; Holotype (male) with labels: &ldquo; HOLOTYPUS [red]// Madagascar (65) Cap Ambalasandra H. BERTRAND, 29.III.60 //MUS. ROY. AFR. CENTR. caput fehlt! TO-ma-03. mikr prap: 03.01. = genital// Incoltorrida madagassica STEFFAN 1973 HOLOTYPUS male [red].&rdquo; Now deposited, with permanent microslide of male genitalia, in MNHN; see remarks.&lt;/p&gt; &lt;p&gt; &lt;b&gt;Material examined (52):&lt;/b&gt; &lt;b&gt;Fianarantsoa&lt;/b&gt;, 20.7722S 47.1809E; Amoron&rsquo;i Mania, 3km south of Ambalamanakana next to RN7, Ankazomivady forest, hygropetric rocks and marsh with vegetation, elev. 1700 m, 1 xi 2014, J. Bergsten, T. Ranarilalatiana &amp; S. Holmgren (MAD 14-02) (3 NHRS). &lt;b&gt;Toamasina&lt;/b&gt;: Analanjirofo: S. side of Nosy Mangabe, Masoala National Park, hygropetric rocks by the sea, 15.5056S, 49.7571E, sea level, 20.II.2018, MAD 18- 60, J. Bergsten &amp; T. Ranarilalatiana (49 NHRS, BMNH, MCZ, PBZT / MBC).&lt;/p&gt; &lt;p&gt; &lt;b&gt;Differential Diagnosis.&lt;/b&gt; Similar in some characters to &lt;i&gt;I. magna&lt;/i&gt;; differing therefrom by smaller size (ca. 2.31 vs. 2.62 mm), less convex elytra with a more gradual posterior declivity, slightly narrower elytral costae, smaller elytral serial punctures, more sinuate fifth elytral costa, and especially differing by having two carinae and a distinct impression between the carinae on the basal 1/2 of the pronotum. The elytron of &lt;i&gt;I. madagassica&lt;/i&gt; has two transverse ridges, offset from one another, one linking costae #3 and #5 (located just behind the humeral umbo), and one linking costae #5 and #8 (located at the sinuation dip of costa #5). In &lt;i&gt;I. magna&lt;/i&gt; these two ridges are located in the same plane, not offset from one another. The male genitalia of &lt;i&gt;I. madagassica&lt;/i&gt; differ from that of &lt;i&gt;I. magna&lt;/i&gt; in being less arcuate in lateral view, and shaped differently in ventral view (Figs. 14, 15, 19, 21).&lt;/p&gt; &lt;p&gt; &lt;b&gt;Description&lt;/b&gt;. Size: holotype (length/width, mm): body (length to elytral apices) 2.31/1.29; head width 0.46; pronotum 0.50/0.96; elytra 1.51/1.29. Dorsum dark brown, venter brown to reddish brown, legs brown to reddish brown except tarsi and femoral-tibial articulations black.&lt;/p&gt; &lt;p&gt;Sides of frontoclypeal shield slightly arcuate toward midline at frontoclypeal suture, narrowest part slightly past midlength of shield, at frontoclypeal suture.&lt;/p&gt; &lt;p&gt;Short oblique carinae on the anterior 1/3 of the pronotum are well developed, but there is no indication of a midlongitudinal carina. Two short carinae and a distinct impression between carinae located on basal 1/2 of pronotum.&lt;/p&gt; &lt;p&gt;Elytron with eight costae; fourth costa interrupted by strong punctures; fifth costa strongly bisinuate; sixth and seventh costae very indistinct. Two transverse ridges, one linking costae #3 and #5 (located just behind the humeral umbo), and one linking costae #5 and #8 (located at the sinuation dip of costa #5).&lt;/p&gt; &lt;p&gt;Posterior 1/2 of metaventral tabella with closely spaced transverse grooves. Midlongitudinal groove in posterior 1/2 of metaventral tabella shallow, with ill-defined margins.&lt;/p&gt; &lt;p&gt;Midlongitudinal carina of first abdominal ventrite is strong and extends length of ventrite.&lt;/p&gt; &lt;p&gt;Male genitalia (Figs. 14, 15, 19) in lateral view slightly arcuate, aedeagal tips wide, apically rounded and with many pores (apparently without microsetae); in ventral view aedeagus nearly parallel-sided, with narrowed tips.&lt;/p&gt; &lt;p&gt; &lt;b&gt;Remarks.&lt;/b&gt; In the original description, Steffan (1973) stated that the holotype was deposited in the MNHN. However, MNHN collection staff were unable to find the type. Search at other museums determined that the holotype was still with the paratypes, at the MRAC. Having permission of that museum, the holotype (fragmentary; Fig. 6) and the permanent slide mounted male genitalia were transferred to the MNHN.&lt;/p&gt; &lt;p&gt;In the original description (Steffan 1973), it is noted by the author that the head of the holotype is missing (&ldquo;caput fehlt&rdquo;). However, both the head and the prothorax are missing (Fig. 6). The two paratypes, both females, are from a different locality than the holotype (&ldquo;Zentrales Hochland auf der Strecke Tananarie - Ambositra&rdquo;). These paratypes are larger than the holotype and have some sculptural differences; they probably represent a different species.&lt;/p&gt; &lt;p&gt;The original description gives &ldquo;Cap Ambalasandra&rdquo; as the type locality. Multiple searches online gave only one record with that geographical name, in a table in a paper on geological deposits. That locality is very near where the Mananara River enters the Indian Ocean (Fig. 38).&lt;/p&gt; &lt;p&gt;Thirty-three larvae were collected in association with adults at site MAD 18-60.&lt;/p&gt;Published as part of &lt;i&gt;Perkins, Philip D. &amp; Bergsten, Johannes, 2019, New Myxophagan water beetles from Madagascar (Coleoptera: Torridincolidae, Hydroscaphidae), pp. 57-96 in Zootaxa 4657 (1)&lt;/i&gt; on pages 64-67, DOI: 10.11646/zootaxa.4657.1.2, &lt;a href="http://zenodo.org/record/3369801"&gt;http://zenodo.org/record/3369801&lt;/a&gt
    ZENODO

    Figure 1 from: Wallin H, Kvamme T, Bergsten J (2017) To be or not to be a subspecies: description of Saperda populnea lapponica ssp. n. (Coleoptera, Cerambycidae) developing in downy willow (Salix lapponum L.). ZooKeys 691: 103-148. https://doi.org/10.3897/zookeys.691.12880

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    Figure 1 - Habitat of Saperda populnea lapponica ssp. n., Trysil: Ljørdalen, Norway with an accumulation of downy willow (Salix lapponum L.) on a boreal and elevated boggy meadow
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    ZENODO

    The EMPIR Project GIQS: Graphene Impedance Quantum Standard

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    METRICA Archivio istituzionale della ricerca - INRIM

    The euro at ten: the next global currency?

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    Over the first ten years of its existence, the euro has proved to be more than a powerful symbol of collective identity. It has provided price stability to previously inflation-prone countries; it has offered a shelter against currency crises; and it has by and large been conducive to budgetary discipline. The eurozone has attracted five new members in addition to the initial eleven, and many countries in Europe wish to adopt it. The euro has also been successful internationally. Even though research presented in this volume confirms that it has not rivaled the dollar's world currency status, it has certainly become a strong regional currency in Europe and the Mediterranean region. Some countries in the region have de facto adopted it, several peg to it, and many have become at least partially euroized. However, the euro's impressive first decade is likely to be followed by a much more difficult period. The present financial crisis is posing at least two important challenges: real economic adjustment within the euro area and maintenance of fiscal and financial stability without a central government authority capable of taking appropriate financial and fiscal decisions in difficult times. This book is the product of a joint conference held in 2008 by the Peterson Institute for International Economics and Bruegel. It is edited by Bruegel Director Jean Pisani-Ferry and then-PIEE Deputy Director and current Bruegel board member Adam Posen. The papers and remarks in this volume demonstrate that the euro has proved to be attractive as a fair weather currency for countries and investors well beyond its borders. But it remains to be seen whether it is equipped to also succeed as a stormy weather currency. Contributors: Joaquín Almunia, Maria Celina Arraes, Leszek Balcerowicz, C. Fred Bergsten, Lorenzo Bini Smaghi, Kristin J. Forbes, Linda S. Goldberg, C. Randall Henning, Mohsin S. Khan, Antonio de Lecea, Erkki Liikanen, Philippe Martin, Thomas Mayer, André Sapir, Dominique Strauss-Kahn, Lawrence H. Summers, and György Szapáry.
    Research Papers in Economics

    Study of Bc+→J/ψDs+ and Bc+→J/ψDs∗+ decays in pp collisions at √s = 13 TeV with the ATLAS detector

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    A study of Bc+→J/ψDs+ and Bc+→J/ψDs∗+ decays using 139 fb−1 of integrated luminosity collected with the ATLAS detector from s = 13 TeV pp collisions at the LHC is presented. The ratios of the branching fractions of the two decays to the branching fraction of the Bc+→ J/ψπ+ decay are measured: B(Bc+→J/ψDs+)/B(Bc+→J/ψπ+) = 2.76 ± 0.47 and B(Bc+→J/ψDs∗+)/B(Bc+→J/ψπ+) = 5.33 ± 0.96. The ratio of the branching fractions of the two decays is found to be B(Bc+→J/ψDs∗+)/B(Bc+→J/ψDs∗+) = 1.93 ± 0.26. For the Bc+→J/ψDs∗+ decay, the transverse polarization fraction, Γ±±/Γ, is measured to be 0.70 ± 0.11. The reported uncertainties include both the statistical and systematic components added in quadrature. The precision of the measurements exceeds that in all previous studies of these decays. These results supersede those obtained in the earlier ATLAS study of the same decays with s = 7 and 8 TeV pp collision data. A comparison with available theoretical predictions for the measured quantities is presented. [Figure not available: see fulltext.]
    Archivio della Ricerca - Università degli Studi di Siena
    Archivio istituzionale della ricerca - Università degli Studi di Udine
    Archivio della Ricerca - Università di Pisa
    Archivio della Ricerca - Università di Roma 3
    ePubs: the open archive for STFC research publications

    Validity of age at menarche self-reported in adulthood

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    Objective: To test the validity of age at menarche self-reported in adulthood and examine whether socioeconomic position, education, experience of gynaecological events and psychological symptoms influence the accuracy of recall.Design: Prospective birth cohort study.Setting: England, Scotland and Wales.Participants: 1050 women from the Medical Research Council National Survey of Health and Development, with two measures of age at menarche, one recorded in adolescence and the other self-reported at age 48 years.Results: By calculating the limits of agreement, kappa statistic and Pearson's correlation coefficients (r), we found that the validity of age at menarche self-reported in middle age compared with that recorded in adolescence was moderate (kappa = 0.35, r = 0.66, n = 1050). Validity was improved by categorising age at menarche into three groups: early, normal and late (kappa = 0.43). Agreement was influenced by educational level and having had a stillbirth or miscarriage.Conclusions: The level of validity shown in this study throws some doubt on whether it is justifiable to use age at menarche self-reported in middle age. It is likely to introduce error and bias, and researchers should be aware of these limitations and use such measures with caution
    Durham Research Online
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    UCL Discovery
    Explore Bristol Research

    Hydraena

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    Hydraena (Hydraenopsis) incertae sedis Hydraena regimbarti Zaitzev Figs. 115 (habitus), 117 (aedeagus), 282 (map) Hydraena marginicollis Régimbart 1903: 51 (nec H. marginicollis Kiesenwetter, 1849). Hydraena regimbarti Zaitzev (nomen novum), 1908 (catal.). Type Material. Lectotype (female): "Ft Dauphin Mad. (Alluaudi); MUSEUM PARIS COLL MAURICE REGIMBART 1908; TYPE [red]; marginicollis Reg. type; [female symbol]; A. d'Orchymont vid. Hydraena (s. str.) marginicollis Reg. [female symbol]; LECTOTYPE Hydraena marginicollis Régimbart 1903, design. P. D. Perkins 2015 [red]"; (MNHN). Specimens examined (87): Fianarantsoa, Atsimo Antsinanana: R.S. Manombo: Parcelle I, Rearatra, Piste 56: S 23.006183 E 47.7338833, GB nets and sieves, forest stream with pools, elev. 21 m, 23° 0' S, 47° 44' E, 14 xii 2013, J.H.Randriamihaja & T.Ranarilalatiana (MAD13-73) (1 NHRS); Ranohira (Fianarantsoa), right affl. of Riv. Ihazofotsy near ' Isalo Ranch', 750 m asl., elev. 750 m, 22° 26' S, 45° 21' E, 25 ix 2001, Gerecke & Goldschmidt (Md 95) (1 NMW); Mahajanga, Boeny: Ankarafantsika NP. S 16.31215 E 0 46.81523, 76 m. a.o. 29 XI 2009 Field # MAD09-02, 16° 18' S, 46° 48' E, J. Bergsten, N. Jönsson, T.Ranarilalatiana, HJ. Randriamihaja (MAD09-02) (7 NHRS); Boeny: Mahavavy Kinkony RS. S 16.01334 E 0 46.00376, elev. 24 m, 16° 1' S, 46° 0' E, 6 xii 2009, J. Bergsten, N. Jönsson, T. Ranarilalatiana, HJ. Randriamihaja (MAD09-33) (4 NHRS); Boeny: Mahavavy Kinkony RS. S 16.13337 E 0 45.95778, elev. 19 m, 16° 8' S, 45° 57' E, 4 xii 2009, J. Bergsten, N. Jönsson, T. Ranarilalatiana, HJ. Randriamihaja (MAD09-25) (10 NHRS); Boeny: Mahavavy Kinkony RS. S 16.14653 E 0 45.94926, elev. 9 m, 16° 9' S, 45° 57' E, 4 xii 2009, J. Bergsten, N. Jönsson, T. Ranarilalatiana, HJ. Randriamihaja (MAD09-24) (2 NHRS); Boeny: Mahavavy Kinkony RS. S 16.15890 E 0 45.93967, 11 m. a.o., 16° 10' S, 45° 56' E, 3 xii 2009, J. Bergsten, N. Jönsson, T. Ranarilalatiana, HJ. Randriamihaja (MAD09-21) (5 NHRS); Forêt de Tsimembo, 11.0 km 346° NNW Soatana, sifted litter (leaf mold, rotten wood) in tropical dry forest, elev. 50 m, 18° 59' S, 44° 26' E, 21– 25 xi 2001, Fisher, Griswold et al. (BLF 4508) (12 CAS); Boeny: Mahavavy Kinkony RS. S 16.14147 E 0 45.93661, water collecting hole, Field # MAD09-20.5 (next to Field # MAD09-20), elev. 12 m, 16° 8' S, 45° 56' E, 3 xii 2009, J. Bergsten, N. Jönsson, T. Ranarilalatiana, HJ. Randriamihaja (MAD09-20.5) (1 NHRS); Boeny: Mahavavy Kinkony RS. S 16.01334 E 0 46.00376, elev. 24 m, 16° 1' S, 46° 0' E, 6 xii 2009, J. Bergsten, N. Jönsson, T. Ranarilalatiana, HJ. Randriamihaja (MAD09-33) (5 NHRS); Melaky: Tsingy de Bemaraha NP. S 18.75804 E 0 44.71290, elev. 90 m, 18° 45' S, 44° 43' E, 17 xii 2009, J. Bergsten, N. Jönsson, T. Ranarilalatiana, HJ. Randriamihaja (MAD09-64) (2 NHRS); Parc National de Namoroka, 16.9 km 317° NW Vilanandro, sifted litter (leaf mold, rotten wood), tropical dry forest, elev. 100 m, 16° 24' S, 45° 18' E, 12–16 xi 2002, Fisher, Griswold et al. (BLF 6582) (1 CAS); Parc National Tsingy de Bemaraha, 3.4 km 93° E Bekopaka, Tombeau Vazimba, sifted litter, tropical dry forest, elev. 50 m, 19° 8' S, 44° 49' E, 6–10 xi 2001, Fisher, Griswold et al. (BLF 4232) (4 CAS); Sofia: Ambinanitelo, river Ambinanitelo next to road RN6 forest stream, S 15.23948 E 47.81591, GB nets and sieves; dried up river with side pools, elev. 43 m, 15° 14' S, 47° 49' E, 18 xi 2012, J. Bergsten, R. Bukontaite, J.H. Randriamihaja & T. Ranarilalatiana (MAD12-08) (6 NHRS); Sofia: Maropapango: river Maropapango next to road RN6, S 14.35419 E 48.01984; nets, sieves, aspirator: river with side pools, elev. 13 m, 14° 21' S, 48° 1' E, 18 xi 2012, J. Bergsten, R. Bukontaite, J.H. Randriamihaja & T. Ranarilalatiana (MAD12-10) (26 NHRS); Toliara, Andranomena, at black light, dry forest near open marsh, 19° 53' S, 45° 4' E, 30 iii 1990, W. E. Steiner, C. Kremen, V. Razafimahatratra (1 USNM); Betroka Reg., 1 st bridge on RN13 S of Intsoka, elev. 923 m, 23° 5' S, 46° 4' E, 7 i 2010, M. Ferroux & P. Obrdik (1 MMBC); Betroka (Tulear), right affl. of Riv. Mangoky NE of the villageponds near aquaeduct, elev. 830 m, 23° 16' S, 46° 0' E, 25 viii 2001, Gerecke & Goldschmidt (MD 47) (1 NMW); Menabe: Kirindy RS. S 20.07476 E 0 44.67075, elev. 49 m, 20° 4' S, 44° 40' E, 12 xii 2009, J. Bergsten, N. Jönsson, T. Ranarilalatiana, HJ. Randriamihaja (MAD09-45) (1 NHRS). Differential Diagnosis. Similar in size and plaques to H sinuatipes (ca. 1.41 mm); differing therefrom in the shape of the pronotum (posterior width less), the pronotum more coarsely punctate, the pronotum maculate instead of fasciate and, in males, the metatibiae being straight, without sinuate margins. The aedeagi of the two species differ markedly (Figs. 113, 117). Also refer to the remarks section. Description. Size: Lectotype (female) (length/width, mm): body (length to elytral apices) 1.41/0.61; head width 0.37; pronotum 0.34/0.49, PA 0.39, PB 0.44; elytra 0.89/0.61. Dorsum of head with frons and clypeus dark brown to piceous; pronotum with dark brown macula, diffusely margined, borders light brown to testaceous; elytra brown; legs light brown; maxillary palpi testaceous, distal 0.5 of last palpomere slightly darker. Frons punctures ca. 1–1.5xef, larger and denser near eyes than medially, interstices shining, 0.5–1xpd laterally, 2–5xpd medially. Clypeus very finely sparsely punctate, interstices shining medially, microreticulate laterally. Mentum very sparsely very finely punctulate, shining. Postmentum punctulate in median depression. Genae raised, shining, without posterior ridge. Pronotum with lateral margins slightly sinuate, posterior angles nearly right angles; anterior margin straight behind eyes, emarginate in middle; punctures on disc larger and deeper than those of frons, interstices weakly shining, 1–4xpd, punctures larger and denser at anterior and posterior; PF1 absent; PF2 shallow, oval; PF3 deep and large; PF4 shallow. Elytra weakly arcuate laterally; summit of posterior declivity at or slightly before midlength; lateral explanate margins narrow; serial punctures on disc ca. 1xpd largest pronotal punctures. Intervals not raised, shining, ca. 1xpd, as are interstices between punctures of a row. Apices in dorsal aspect conjointly rounded, in posterior aspect margins forming (male) shallow, (female) very shallow angle, if any, with one another. Ratios of P2 width and plaque shape (P2/w/l/s) ca. 1/0.25/1/2. P1 laminate; median carina sinuate in profile. P2 l/w ca. 2/1, sides slightly converging toward blunt apex, apex raised slightly above metaventral intercoxal process. Plaques very narrow lines, converging slightly anteriorly, widely separated, weakly raised, located at sides of shallow median depression. Metaventrite with very short longitudinal ridge on each side, extended posteriorly from margin of each mesocoxal cavity. AIS width at slightly arcuate posterior margin ca. 1.5x P2. Protibia (both sexes) very slightly arcuate, other tibiae straight, slender. Abdominal apex (male) with apicomedian asymmetrical notch. Remarks. The lectotype (female) was collected at Fort Dauphin. Two Hydraena species from that locality are present in the material studied, but both of these differ from the lectotype in several characters: H. furcula is much larger (ca. 2.17 vs. 1.41 mm) and the habitus and plaques differ markedly from those of H. regimbarti; H. elementaria is closer in size (ca. 1.51 mm), and the plaques are more similar to those of H. regimbarti, however the shape, foveae, and finely punctate pronotum differ from those of the lectotype. The specimens of H. regimbarti available include several localities in Toliara (Fig. 282), but none at Fort Dauphin.Published as part of Perkins, Philip D., 2017, Hydraenidae of Madagascar (Insecta: Coleoptera), pp. 1-264 in Zootaxa 4342 (1) on pages 125-127, DOI: 10.11646/zootaxa.4342.1.1, http://zenodo.org/record/104106
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