4,860 research outputs found
Letter from J. Cogan to Hagan
No full textHolograph letter from J. Cogan, St. Joseph's College, Carbally Park, Ballinasloe (County Galway), to Hagan. Regretting he cannot attend the celebrations; informing him of their two new students Joseph Cunniffe and William Reid. Fr. Fair will also give good reference of them
Cogan, T E J, VX43247
No full textThis record was harvested from a previous catalogue system and will be withdrawn in 2025. Information in this record may be superseded or incomplete. Visit this record in UMA's new catalogue at: https://archives.library.unimelb.edu.au/nodes/view/377880Surname: COGAN
Given Name(s) or Initials: T E J
Military Service Number or Last Known Location: VX43247
Missing, Wounded and Prisoner of War Enquiry Card Index Number: 45344191694
Item: [2016.0049.10175] "Cogan, T E J, VX43247
Visual Hallucinations / Epileptic Seizures
No full textcurriculum_fellowHistory: 65 year old woman; 2-3 year history of episodes of warm surge going through the body beneath the sternum accompanied by appearance of female figure. Diagnosed by Dr. David Cogan as epileptic seizure. Disease/ Diagnosis: Epilepsy
Agraphia (with associated alexia); Agnosias
No full textRSagraphiaHistory: 32 year old male; partial pneumonectomy 3 weeks prior; post-operative difficulty finding objects and with memory; confabulates to circumvent memory deficit. Presenting Symptom: visual function has improved significantly since the earlier recording (5 days prior); memory remains profoundly deficient; spatial localization is the predominant visual problem at present. Disease/ Diagnosis: Dyslexia, Acquired; Agraphia; Agnosia. See also: http://ecclesvideo.med.utah.edu/NOVEL/Cogan/23-5.wm
Experimental Parkinson\u27s Disease and Hemiplegia (Monkey)
No full textBankiewicz KS, Oldfield EH, Plunkett RJ, Schuette WH, Cogan DG, Hogan N, Zuddas A, and Kopin IJ: Apparent Unilateral Visual Neglect in MPTP-hemiparkinsonian monkeys due to delayed initiation of motion. Brain Research. 541:98-102, 1991.History: MPTP induced. Left carotid infusion of MPTP with left sided Parkinsonism. Left sided visual neglect and anatomical changes in left substantia nigra and left retinal pigment epithelium. Disease/ Diagnosis: Parkinson Disease
Interview with Dr. John Cogan
No full textEconomicStudies_AnalysisDr. John Cogan was the spring, 2018 recipient of the Kirby Distinguished Visiting Professorship. During his visit to Texas A&M University, he was interviewed by Dr. Andrew J. Rettenmaier
Towards inclusion? Models of behaviour support in secondary schools in one education authority in Scotland
No full textThis article draws on data emerging from an evaluation of behaviour support strategies in secondary schools in an education authority in Scotland. The authors all work at the University of Glasgow. Jean Kane has research and teaching interests in the area of special educational needs; she offers consultancy to local authorities in the development of inclusive policies and practices in schools. Dr George Head has research and teaching interests in the area of social, emotional and behavioural difficulties and social inclusion; he is also an experienced teacher. Both Jean Kane and George Head are lecturers in the Faculty of Education. Nicola Cogan is a researcher at the Strathclyde Centre for Disability Research and has a background as a research psychologist in the health sector.
In Scotland, the growth of behaviour support provision is closely related to broader policy on social inclusion. It is argued in this article that new models of behaviour support can be developed in the light of previous and related experience in the development of inclusive support systems in schools. The authors present a typology of behaviour support, drawing upon their evaluation of provision, and discuss the characteristics of the types of support that emerge. Using data from exclusion statistics, pupil case studies and interviews with teachers, managers, pupils and parents, Jean Kane, George Head and Nicola Cogan explore the implications of their work for future developments in support for pupils who present difficult behaviours
Cogan Lid Twitch
No full textA 50-year-old woman presented with ptosis of her left eyelid for 6 months. Several exam findings including variable and fatigable ptosis, and Cogan lid twitch, raised suspicion for Myasthenia Gravis. Acetylcholine receptor binding, blocking, and modulating antibodies were negative, and single fiber EMG was normal. MRI brain/orbits without and with contrast was unremarkable. There was no evidence of thymoma on chest CT. Diagnosis of Myasthenia was ultimately confirmed with positive ice pack test, positive edrophonium (Tensilon) test, and suggestive history and exam findings. The patient was started on Pyridostigmine 60mg 5 times daily, to which she responded well, but stopped it a year later on her own with resolution of diplopia and ptosis. The video displays a Cogan lid twitch of the patient\u27s left eyelid. First described by Dr. David G. Cogan in 1965, the Cogan lid twitch (CLT) is a sign of Myasthenia Gravis and can be elicited by having the patient initially maintain downward gaze and then look up to the primary position. The lid will show a brief upward twitch. Cogan theorized that this sign could be explained by the quick fatigability and rapid recovery of the myasthenic levator palpebrae superioris. The downward gaze allows for relaxation of the muscle which allows for recovery once the patient looks straight ahead. As the elevation is only maintained for a brief period, it appears as a twitch [1]. The specificity of the CLT has been reported as 99% and sensitivity as 75% in one study [2]. [1] Cogan DG. Myasthenia Gravis: A Review of the Disease and a Description of Lid Twitch as a Characteristic Sign. Archives of Ophthalmology. 1965;74(2):217-221. doi:10.1001/archopht.1965.00970040219016 [2] Singman E; Matta N; Silbert DI. Use of the Cogan Lid Twitch to Identify Myasthenia Gravis. Journal of Neuro-Ophthalmology. 2011;31(3):239-240 doi:10.1097/WNO.0b013e3182224b92[1] Cogan DG. Myasthenia Gravis: A Review of the Disease and a Description of Lid Twitch as a Characteristic Sign. Archives of Ophthalmology. 1965;74(2):217-221. doi:10.1001/archopht.1965.00970040219016 [2] Singman E; Matta N; Silbert DI. Use of the Cogan Lid Twitch to Identify Myasthenia Gravis. Journal of Neuro-Ophthalmology. 2011;31(3):239-240 doi:10.1097/WNO.0b013e3182224b9
Atypisches Cogan Syndrom und Morbus Basedow: beiderseitige Ertaubung, Neuritis nervi optici und systemische Manifestationen bei einer Schwarzafrikanerin
No full textKasuistik: Wir berichten über den schubweisen, jahrelangen Krankheitsverlauf eines atypischen Cogan Syndroms einer 49-jährigen Kenianerin, die sich bei uns mit einer zunächst einseitigen akuten Ertaubung, Tinnitus und Gangunsicherheit bei vorbestehender mittelgradiger Innenohrschwerhörigkeit kontralateral vorstellte. Die Eigenanamnese gab Hinweise auf eine vor 17 Jahren durch Korticosteroide ausgeheilte Iritis. Weiterhin war die Patientin seit 9 Jahren wegen beiderseitig rezidivierenden Hörstürzen alio loco mehrmalig antiphlogistisch-rheologisch infundiert worden. Die akute Ertaubung führte zur stationären Aufnahme. Eine Tympanotomie mit Abdichtung des runden Fensters und die antiphlogistisch-rheologische Infusionstherapie blieben erfolglos. Das MRT-Schädel stellte eine Signalintensitätserhöhung okzipital links dar, die am ehesten einem ischämischen Herd entsprach und im Vergleich zum Vorbefund von 1998 neu war. Ein retrocochleäres Geschehen konnte ausgeschlossen werden. Das Routinelabor ergab keine wegweisenden Befunde. Die Luesserologie war negativ. Drei Monate später ertaubte die Patientin auf der Gegenseite. Weder operative noch konservative Therapien waren erfolgreich, so dass die Vorbereitungen zur Cochlea Implantation einseitig aufgenommen wurden. Ein Monat später beklagte die Patientin eine akute Sehverschlechterung ipsilateral, die durch eine Neuritis nervi optici erklärt werden konnte. Serologisch ergaben sich keine Hinweise auf eine Vaskulitis. Mit der neu aufgetretenen Augensymptomatik konnte erstmals die Diagnose eines Cogan Syndroms gestellt werden. Aufgrund der Gefahr einer Cochleaobliteration beim Cogan Syndrom entschlossen wir uns umgehend beiderseitig zu implantieren. Die Visusverschlechterung erholte sich ad integrum auf die hoch dosierte Kortikosteroidtherapie. Drei Monate postoperativ entwickelt die Patientin, trotz einer Prednisolon Erhaltungsdosis plötzlich eine Verschlechterung ihres Allgemeinzustandes mit Arthralgien der großen Gelenke. Gleichzeitig fielen im Serum um das 3- bis 5fach erhöhte freie Schilddrüsenhormonwerte auf bei massiv erniedrigtem TSH. Wir interpretieren den aktuellen Krankheitsverlaufverlauf als eine systemische Manifestation eines atypischen Cogan Syndroms, wobei eine hyperthyreote Stoffwechsellage in diesem Zusammenhang bisher nicht beschrieben wurde. Die mannigfaltigen Erscheinungsformen des atypischen Cogan Syndroms mit systemischem Verlauf erschweren die Erstdiagnosestellung. Darüber hinaus ist eine interdisziplinäre Strategieplanung hinsichtlich Diagnostik und Therapie unumgänglich
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