919 research outputs found

    Not Evidence for Baumol’s Cost Disease. A Reply to Atanda and Reed (International Journal for Re-Views in Empirical Economics, 2020)

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    In this article Jochen Hartwig replies to Antanda's and Reed's replication study published earlier this year (IREE, 2020-1)

    Marx's reproduction schemes and the Keynesian multiplier: a reply to Sardoni

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    In a recent contribution to this journal, C. Sardoni takes issue with the identification by Trigg, in a 2006 publication, of a role for the Keynesian investment multiplier in Marx's schemes of reproduction. Indirectly, Sardoni also expresses his disagreement with Hartwig (by attributing one of his statements to Trigg). We appreciate the opportunity to defend our view against Sardoni's critique. This reply shows that a bridging point between Marx and Keynes can be established without recourse to microfoundations. As suggested by both Trigg, in 2006, and Hartwig, in 2004, the well known Harrod--Domar model of economic growth provides an interpretation of Marx's reproduction schemes that has the Keynesian multiplier as a constituent element. This note will further explore the assumptions underlying the interface between Marx and Keynes, in response to the challenging questions raised in Sardoni's contribution. Copyright The Author 2010. Published by Oxford University Press on behalf of the Cambridge Political Economy Society. All rights reserved., Oxford University Press.

    Gene loss and lineage specific restriction-modification systems associated with niche differentiation in the Campylobacter jejuni Sequence Type 403 clonal complex

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    Campylobacter jejuni is a highly diverse species of bacteria commonly associated with infectious intestinal disease of humans and zoonotic carriage in poultry, cattle, pigs, and other animals. The species contains a large number of distinct clonal complexes that vary from host generalist lineages commonly found in poultry, livestock, and human disease cases to host-adapted specialized lineages primarily associated with livestock or poultry. Here, we present novel data on the ST403 clonal complex of C. jejuni, a lineage that has not been reported in avian hosts. Our data show that the lineage exhibits a distinctive pattern of intralineage recombination that is accompanied by the presence of lineage-specific restriction-modification systems. Furthermore, we show that the ST403 complex has undergone gene decay at a number of loci. Our data provide a putative link between the lack of association with avian hosts of C. jejuni ST403 and both gene gain and gene loss through nonsense mutations in coding sequences of genes, resulting in pseudogene formation

    Effective Demand: Securing the Foundations: Olivier ALLAIN on Jochen HARTWIG (2007) and Mark HAYES (2007)

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    A panel session was organised at the 5th "Dijon" Post-Keynesian Conference (Roskilde University - 13th-14th May 2011) in order to confront three recent interpretations of Keynes's principle of effective demand: that of Hartwig (2007), Hayes (2007) and Allain (2009). Allain's comments on Hartwig and Hayes articles are developed in the present contribution

    Not Evidence for Baumol's Cost Disease. A replication study of Hartwig (Journal of Health Economics, 2008)

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    In his 2008 Journal of Health Economics paper, Jochen Hartwig claimed that Baumol's Cost Disease (BCD) theory could explain observed increases in health care expenditures in OECD countries. This paper replicates Hartwig's results and demonstrates that he tested the wrong hypothesis. When one tests the correct hypothesis, Hartwig's conclusions are not supported. Rather than providing evidence in favor of BCD, Hartwig's estimation procedures, when applied correctly, strongly reject BCD as an explanation for health expenditure increases for the OECD data he examined
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