90,824 research outputs found
[Report to Chief J. E. Curry, by an unknown author #1]
No full textReport to Chief J. E. Curry, by an unknown author. The report contains a list of officers who gave depositions to the United States Attorney
[Report to Chief J. E. Curry, by an unknown author #2]
No full textReport to Chief J. E. Curry, by an unknown author. The report contains a list of officers who gave depositions to the United States Attorney
Repopulation of a coastal stream by brook trout and rainbow-trout after endosulfan poisoning
No full textPT: J; CR: BERGER BL, CITED INDIRECTLY GUNNING GE, 1968, PROG FISH CULT, V30, P92 OLMSTED LL, 1974, T AM FISH SOC, V103, P79 PETERSON RH, 1975, J FISH RES BOARD CAN, V31, P1757 PHILLIPS WEJ, 1965, ENDOSULFAN ITS EFFEC PHILLIPS WEJ, 1975, 14098 NAT RES COUNC RICKER WE, 1968, IBP3 INT BIOL PROGR SCHOETTGER RA, 1970, BUR SPORT FISH WILDL, V35, P1 SMITH MW, 1958, J FISH RES BOARD CAN, V15, P1403 SMITH MW, 1963, J FISH RES BOARD CAN, V20, P327; NR: 10; TC: 3; J9: PROGR FISH-CULT; PG: 4; GA: JS849Source type: Electronic(1
Quantitative estimates of fish abundance from boat electrofishing
No full textMultiple removals by boat electro-fishing were used to estimate fish populations in non-wadeable habitats in New Zealand lakes and rivers. Mean capture probability was 0.47±h0.10 (± 95% CI) from 35 population estimates made with 2-7 successive removals. The relationship between the population estimate from the Zippin method (Y)and the number of fish caught in the first removal (X) was significant (adjusted r2=0.84, P<0.001; Figure 2). The least-squares regression was Y = 1.55X 1.23. Mean density ± 95% confidence interval for 13 fishing occasions was 30±27 fish 100 m-
2. Mean biomass of fish for sites was 78±39 g m-2 (range 29 to 245 g m-2). Koi carp comprised the largest proportion of the fish biomass wherever they were present. The high biomasses of koi carp estimated in these results (mean 56±33 g m-2) suggest that they can reach problematic abundances in New Zealand. Bioniass of spawning koi carp can exceed 400 g m-2
Coos River Basin fish management plan
No full textprepared by Linda J. Wagoner, Kim K. Jones, Reese E. Bender, Jerry A. Butler, Darrell E. Demory, Thomas F. Gaumer, Joel A. Hurtado, William G. Mullarkey, Paul E. Reimers, Neil T. Richmond, Thomas J. Rumreich.This archived document is maintained by the State Library of Oregon as part of the Oregon Documents Depository Program. It is for informational purposes and may not be suitable for legal purposes.Includes bibliographical references (pages 122-124).Mode of access: Internet from the Oregon Government Publications Collection.Text in English
Effects of calcium channel blockers on pharmacologically induced contractions of rainbow trout (Oncorhynchus mykiss) intestine
No full textPT: J; CR: BERRIDGE MJ, 1988, P R SOC LONDON B, V234, P359 BRINK C, 1981, J PHARMACOL EXP THER, V217, P592 BURKA JF, 1989, ANIMAL DRUGS FOOD SA, P110 BURKA JF, 1989, CAN J PHYSIOL PHARM, V67, P477 BURKA JF, 1990, IN PRESS CAN J PHYSL, V68 BURNSTOCK G, 1958, BRIT J PHARMACOL CHE, V13, P216 BURNSTOCK G, 1959, Q J MICROSC SCI, V100, P199 CARPENTER JR, 1986, J PHARMACOL METHOD, V15, P283 COOK DA, 1977, FED PROC, V36, P2584 HOAR WS, 1967, LABORATORY COMPANION ISHIKAWA S, 1985, BRIT J PHARMACOL, V86, P789 JANIS RA, 1983, BIOCHEM PHARMACOL, V32, P3499 JANIS RA, 1983, J MED CHEM, V26, P775 KARAKI H, 1988, LIFE SCI, V42, P111 KITCHEN I, 1984, TXB INVITRO PRACTICA LEFF P, 1987, J PHARMACOL EXP THER, V240, P284 SAIDA K, 1983, BLOOD VESSELS, V20, P105 SCHWARTZ A, 1984, ANNU REV MED, V35, P325 SMALL RC, 1982, BRIT J PHARMACOL, V77, P45 SPEDDING M, 1988, ANN NY ACAD SCI, V522, P248 VANBREEMEN C, 1979, PHARMACOL REV, V30, P167 WOLOWYK MW, 1987, J THERM BIOL, V12, P87; NR: 22; TC: 2; J9: FISH PHYSIOL BIOCHEM; PG: 7; GA: EG368Source type: Electronic(1
Resistance to Loma salmonae in rainbow trout Oncorhynchus mykiss subsequent to recovery from an initial low-dose cohabitation exposure
No full textProtective response was investigated in rainbow trout Oncorhynchus mykiss that had recovered from a primary low-dose cohabitation challenge of Loma salmonae and were re-challenged via a high dose oral exposure under experimental conditions. Compared to uninfected control fish, the previously exposed trout had 82.6% and 86.0% fewer xenomas, and many of the recovered fish (47% and 55%) showed no signs of infection whereas 95% and 100% of the control fish developed xenomas on their gills in two experimental tanks. From the results we conclude that rainbow trout develop protective immunity following low-dose exposure to L. salmonae acquired through cohabitation. The relevance of this with respect to unexpected patterns of disease presentation at commercial salmon farming sites is discussed.PT: J; UT: BIOSIS:PREV201100298882Source type: Electronic(1
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Fish and wildlife resources of the John Day Basin, Oregon and their water requirements
No full textThe Basin Investigation Section, Oregon State Game Commission (now a part of the Environmental Management Section, Oregon Department of Fish and Wildlife) studied the John Day Basin to provide the State Water Resources Board with fish life water requirement data. Field studies were conducted during 1966, 1967, and 1970. Principal results of these studies are instream flow quantity recommendations shown in Appendixes 1 and 2. Additional information includes fish and wildlife resources, their abundance, distribution, value, and related information. Stream flow and temperature measurements recorded are tabulated in Appendix 4. Field work was conducted by Jim E. Lauman, Gregory J. Hattan, John D. Fortune, and Kenneth E. Thompson, aquatic biologists, and Ronald L. Lindland, student trainee.A report with recommendations to the Oregon State Water Resources Board.Keywords: Streamflow, Fish passage, Salmonids, Fish and wildlife, Fish production, Water resources, Salmo
CpG inclusion in feed reduces sea lice, Lepeophtheirus salmonis, numbers following re-infection
No full textLepeophtheirus salmonis infections in Atlantic salmon, Salmo salar, have been characterized by little to no hyperplastic response and a biphasic immune response that results in chronic inflammation with tissue repair as the infection progresses. We hypothesized that CpG administration with prior lice exposure would enhance epithelial inflammatory mechanisms and boost the Atlantic salmon immune response to L. salmonis, leading to greater protection against infection. We administered multiple exposures of L. salmonis to two groups of Atlantic salmon and compared responses against first-time exposed Atlantic salmon. Following re-exposure, CpG fed fish exhibited increased skin expression of interleukin (IL)-1β and IL-12 β compared to control previously exposed (CPE) and control first-time exposed (CFE) animals, respectively. This inflammatory enhancement occurred with significantly lower expression of matrix metalloproteinase-9 (MMP 9), both systemically (spleen) and locally (skin). Reduced MMP 9 expression was a hallmark of the re-infected fish (occurred in both tissues at both times). When significant differences were present in the skin or spleen, the two re-exposed groups showed greater similarity than with the first exposure group. Lice numbers on CpG fed fish were significantly lower than CFE fish at 7 days post-re-infection (dpri), and although they were not significantly different at 17 dpri, the trend of lower lice levels remained. CpG fed fish also showed nearly twofold greater protection than CPE when compared to the CFE group (48.5% vs. 27.0% reductions at 7 dpri and 27.2% vs. 13.1% reductions at 17 dpri, respectively). The enhanced protection of CpG oligodeoxynucleotide administration to previous exposure was consistent across all body surfaces and suggests that CpG can not only enhance innate responses to L. salmonis in Atlantic salmon, but also further stimulate adaptive responses
Review: fish microsporidia: immune response, immunomodulation and vaccination
No full textImmune response to fish microsporidia is still unknown and there are current research trying to elucidate the events involved in the immune response to this parasite. There is evidence suggesting the role of innate immune response and it is clear that adaptive immunity plays an essential part for eliminating and then mounting a solid resistance against subsequent microsporidian infections. This review article discusses the main mechanisms of resistance to fish microsporidia, which are considered under four main headings. 1) Innate immunity: the inflammatory tissue reaction associated with fish microsporidiosis has been studied at the ultrastructural level, providing identification of many of the inflammatory cells and molecules that are actively participating in the spore elimination, such as macrophages, neutrophils, eosinophilic granular cells, soluble factors and MHC molecules. 2) Adaptive immunity: the study of the humoral response is relatively new and controversial. In some cases, the antibody response is well established and it has a protective role, while in other situations, the immune response is not protective or it is depressed. Study of the cellular response against fish microsporidia is still in its infancy. Although the nature of the microsporidian infection suggests participation of cellular mechanisms, few studies have focused on the cellular immune response of infected fish. 3) Immunomodulation: glucans are compounds that can modulate the immune system and potentiate resistance to microorganisms. These compounds have been proposed that can interact with receptors on the surface of leukocytes that result in the stimulation on non-specific immune responses. 4) Vaccination: little is known about a biological product that could be used as a vaccine for preventing this infection in fish. In the Loma salmonae experience, one of the arguments that favor the production of a vaccine is the development in fish of resistance, associated to a cellular immune response. A recently proved spore-based vaccine to prevent microsporidial gill disease in salmon has recently shown its efficacy by considerably reducing the incidence of infection. This recent discovery would be first anti-microsporidian vaccine that is effective against this elusive parasite
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