52 research outputs found
Refaat Alareer’s “If I Must Die”: The Death of the Author, the Afterlife of the Tale
No full textThis article traces the performative role of tales and storytelling in late Refaat Alareer’s life, career, (creative) writing, activism, and death. It ultimately examines this performativity’s intensification and culmination in Alareer’s Saidian late-style poem “If I Must Die.” The article combines close textual analysis and comparative literary criticism to investigate the intricate relationship between storytelling, mortality, and resistance in Alareer’s poem. Paying attention to nuance, the article examines the poem’s intertextual connections with Claude McKay’s “If We Must Die” (1919) to demonstrate how Alareer transforms storytelling into a powerful mode of cultural survival and a means of transcending physical destruction and preserving collective memory in the face of systematic oppression. Additionally, the research traces Alareer’s evolving conceptualization of storytelling from a personal imperative to a collective form of resistance, culminating in “If I Must Die” whose stylistic and thematic treatment of his impending death anchor his poem locally/nationally, namely in Gaza/Palestine
Session 26: Community and collaboration: co-creating resources to promote opportunities for disabled and neurodivergent students
No full textFor many years, studies of the destinations of UK graduates illustrate the disadvantage experienced by disabled and neurodivergent graduates when it comes to employment (AGCAS 2024, 2022, 2021). Employment is recognised as a way in which people find purpose, self-confidence and economic wellbeing (Pratt et al. 2014); indeed, employment is a human right (Nolan & Gleeson 2017). For several years, colleagues in Student Futures have collaborated with disabled and neurodivergent students - those we see as experts by experience - to develop resources to support the success of LJMU students in the workplace. In this fourth year of collaboration, we have been particularly concerned with ensuring that our message reaches current disabled and neurodivergent students, so that they are aware of the resources and provision available to aid their success.
In this session, three students and two careers practitioners from Student Futures will describe the latest iteration of the co-creation internship project. A range of videos, social media posts and other resources were created, which will be shown during the session. In addition, the learning gained from this collaboration will be shared. For the interns, this has included the enjoyment of being able to choose the ways to promote initiatives to fellow students which they deem to be most effective. For the careers practitioners, working with the interns has led to a deeper understanding of the concerns and perspectives of these students, with implications for future practice. This session will describe the achievements of the project, outline recommendations for other staff and highlight the benefits of a deepened understanding and a sense of empowerment for all involved.
Community and collaboration: co-creating resources to promote opportunities for disabled and neurodivergent students PowerPoint. Only LJMU staff and students have access to this resource
Utility of the exercise electrocardiogram testing in sudden cardiac death risk stratification
No full textBackground Sudden cardiac death (SCD) remains a major public health problem. Current established criteria identifying those at risk of sudden arrhythmic death, and likely to benefit from implantable cardioverter defibrillators (ICDs), are neither sensitive nor specific. Exercise electrocardiogram (ECG) testing was traditionally used for information concerning patients' symptoms, exercise capacity, cardiovascular function, myocardial ischemia detection, and hemodynamic responses during activity in patients with hypertrophic cardiomyopathy. Methods We conducted a systematic review of MEDLINE on the utility of exercise ECG testing in SCD risk stratification. Results Exercise testing can unmask suspected primary electrical diseases in certain patients (catecholaminergic polymorphic ventricular tachycardia or concealed long QT syndrome) and can be effectively utilized to risk stratify patients at an increased (such as early repolarization syndrome and Brugada syndrome) or decreased risk of SCD, such as the loss of preexcitation on exercise testing in asymptomatic Wolff-Parkinson-White syndrome. Conclusions Exercise ECG testing helps in SCD risk stratification in patients with and without arrhythmogenic hereditary syndromes. © 2014 Wiley Periodicals, Inc.Adler A, 2012, HEART RHYTHM, V9, P901, DOI 10.1016-j.hrthm.2012.01.026; Atta S, 2012, J CLIN EXP CARDIOLOG, V3, P223; Bastiaenen R, 2013, HEART RHYTHM, V10, P247, DOI 10.1016-j.hrthm.2012.10.032; Bershader RS BC, 2007, HEART RHYTHM, V4, pS138; Calloe K, 2013, CIRC-ARRHYTHMIA ELEC, V6, P177, DOI 10.1161-CIRCEP.112.974220; Chattha IS, 2010, HEART RHYTHM, V7, P906, DOI 10.1016-j.hrthm.2010.03.006; Cohen MI, 2012, HEART RHYTHM, V9, P1006, DOI 10.1016-j.hrthm.2012.03.050; Elhendy A, 2002, AM J CARDIOL, V90, P95, DOI 10.1016-S0002-9149(02)02428-1; Engel G, 2004, CURR PROB CARDIOLOGY, V29, P365, DOI 10.1016-j.cpcardiol.2004.02.007; Frolkis JP, 2003, NEW ENGL J MED, V348, P781, DOI 10.1056-NEJMoa022353; Gimeno JR, 2009, EUR HEART J, V30, P2599, DOI 10.1093-eurheartj-ehp327; Goldberger Jeffrey J, 2008, Heart Rhythm, V5, pe1, DOI 10.1016-j.hrthm.2008.05.031; Haissaguerre M, 2008, NEW ENGL J MED, V358, P2016, DOI 10.1056-NEJMoa071968; HINDMAN MC, 1973, ANN INTERN MED, V79, P654; Horner JM, 2011, HEART RHYTHM, V8, P1698, DOI 10.1016-j.hrthm.2011.05.018; Josephson ME, 2000, ANN INTERN MED, V133, P901; Kentta T, 2012, HEART RHYTHM, V9, P1083, DOI 10.1016-j.hrthm.2012.02.030; Lahat H, 2001, AM J HUM GENET, V69, P1378, DOI 10.1086-324565; Laitinen PJ, 2001, CIRCULATION, V103, P485; Makimoto H, 2010, J AM COLL CARDIOL, V56, P1576, DOI 10.1016-j.jacc.2010.06.033; MARIEB MA, 1990, AM J CARDIOL, V66, P172, DOI 10.1016-0002-9149(90)90583-M; Meli AC, 2011, CIRC RES, V109, P281, DOI 10.1161-CIRCRESAHA.111.244970; Morshedi-Meibodi A, 2004, CIRCULATION, V109, P2417, DOI 10.1161-01.CIR.0000129762.41889.41; O'Neill JO, 2004, J AM COLL CARDIOL, V44, P820, DOI 10.1016-j.jacc.2004.02.063; Priori SG, 2002, CIRCULATION, V106, P69, DOI 10.1161-01.CIR.0000020013.73106.D8; Priori SG, 2013, EUROPACE, V15, P1389, DOI 10.1093-europace-eut272; Raju H, 2011, HEART RHYTHM, V8, pS41; Roux-Buisson N, 2012, HUM MOL GENET, V21, P2759, DOI 10.1093-hmg-dds104; Steinhaus DA, 2012, AM HEART J, V163, P125, DOI 10.1016-j.ahj.2011.09.016; Sy RW, 2011, CIRCULATION, V124, P2187, DOI 10.1161-CIRCULATIONAHA.111.028258; Tseng ZH, 2009, HEART RHYTHM, V6, P1315, DOI 10.1016-j.hrthm.2009.06.034; van der Werf C, 2010, CIRC-ARRHYTHMIA ELEC, V3, P96, DOI 10.1161-CIRCEP.109.877142; Watanabe J, 2001, CIRCULATION, V104, P1911; Zheng ZJ, 2001, CIRCULATION, V104, P2158, DOI 10.1161-hc4301.098254; Zipes DP, 2006, J AM COLL CARDIOL, V48, pe247, DOI DOI 10.1016-J.JACC.2006.07.0100
Pilot study for early prognosis of Azoospermia in relation to Y-STR Profiling
No full textAbstractBackgroundAzoospermia constitutes 20% of male infertility situations and affects 1% of the total male population (Jarvi et al., 2010). This condition is classified into three major types; pre-testicular, testicular and post-testicular Azoospermia (Sermondade et al., 2012). Genetic defects causing Azoospermia are due to chromosomal or non-chromosomal alterations on the Y-Chromosome (Lee et al., 2011). Initial diagnosis of Azoospermia is established when no spermatozoa are detected on microscopic examination of semen (World Health Organisation, 1999).ObjectiveTo evaluate the correlation of Y-STR Profiling results and the prevalence of Azoospermia, to help for early prognosis of Azoospermia before puberty.MethodsBuccal swab samples were taken from two groups of individuals (50 fertile and 50 Azoospermic patients), then DNA was isolated using QIAamp DNA Micro kit. DNA quantification was done using a Real-time PCR utilizing Quantifiler Kit. PCR was done using PowerPlex® Y PCR Amplification Kit, then amplified products were typed using a 3130 Genetic Analyzer.ResultsFive haplotypes in four different Y-STR loci were found to possess significantly higher occurrence percentages in Azoospermic than in fertile Saudi individuals, which can serve as a group of pre-diagnostic markers for early prognosis of Azoospermia in Saudi population.ConclusionThere was a significant correlation of Y-STR Profiling results and the prevalence of Azoospermia condition, which supports the idea of using Y-STR Profiling in early prognosis of Azoospermia
Under siege : remembering Leningrad, surviving Gaza
No full textHow does one communicate what life is like under siege? Much has been written to describe the besieged Gaza Strip yet Al-Shabaka Policy Member Ayah Abubasheer and Guest Author Esther Rappaport bring new insights and perspectives in this Roundtable. Ayah Abubasheer lives under the siege of Gaza at the present time; Esther Rappaport’s family lived under the siege of Leningrad during World War II. Ayah and Esther came to know each other through social media during the Summer 2014 attack on Gaza and first thought of writing this piece during this war. In their reflections and analysis of the two sieges they ably communicate the stark reality of life under siege. The reflections of each of the two authors are given in their own voice. They also provide some additional factual information and background, and this part of their discussion, conversation, and argument is presented in the voice of a “narrator”
Cardiac electrophysiology in Lebanon-part II
No full textSystematic national effort to improve cardiac electrophysiology practice in Lebanon is lacking, and the quality improvement program mainly relates to individual efforts along with regulations, which are set as a Road Map by the Lebanese Arrhythmia Working Group. Lebanon currently has fve electrophysiology laboratories. The Road Map mainly consists of creating a registry and a National Card for Electronic Device Holder, centralization of complex electrophysiology procedures in institutions where electrophysiologists are available, setting regulations to conform to international guidelines, and creating a National Arrhythmia Website and E-Journal. Most importantly, we emphasize that the practice of device checking must be performed by physicians with expertise and not by industry technicians. © the author(s), publisher and licensee Libertas Academica Ltd
Preparation and characterization of new in(iii), re(iii), and re(v) complexes with thenoyltrifluoroacetone and some bidentate heterocyclic ligands
No full textAll chemical used were of analytical grade and were used without
further purification. IR spectra were recorded in KBr pellets on a Perkin
Elmer FT-IR spectrometer. "Spectrum 1000". UV-Vis spectra in ethanolic
solutions were recorded on a Cecil 599 spectrophotometer using 1 cm
matched silica cells. 'H and , 3 C NMR spectra were measured in DMSO-d6
as solvent on a JEOL NMR 400 MHZ spectrometer. Elemental analyses
were carried out on a Perkin Elmer 2400 CHNSO elemental analyzer.
Iidium contents in the investigated indium complexes were determined by
the analytical laboratory at SABIC (R&D) (Saudi Arabia). TG measurements
were carried out using a Netzsch STA-429 thermal analyzer. The
weight loss was measured from ambient temperature up to 700 °C'at a
heating rate of 10 °C/min. X-ray diffraction patterns of the complexes were
recorded using a XRD 5000 Siemens diffractometer with Cu-target.Corresponding Author:
Dr. Refaat M. Mahfouz, Department of Chemistry, College of Sciences, King Saud University, P.O. Box 2455, Riyadh 11451, Saudi Arabia.
Email: [email protected]
Association of CASQ2 polymorphisms with sudden cardiac arrest and heart failure in patients with coronary artery disease
No full textBackground: Abnormal calcium handling plays a crucial role in arrhythmias, sudden cardiac arrest (SCA), and congestive heart failure (CHF). Calsequestrin 2 (CASQ2) mutations affect calcium release and initiate malignant ventricular arrhythmias (VAs) and SCA syndromes. Common single nucleotide polymorphisms (SNPs) in CASQ2 may be associated with SCA in patients with coronary artery disease (CAD). Objective: The purpose of this study was to examine the association of common CASQ2 SNPs with the risk of SCA in patients with CAD. Methods: CASQ2 SNPs (n = 14) were genotyped and analyzed in a case control study comparing 114 patients with CAD and SCA due to VA to 311 CAD controls without VA or SCA. Results: Multivariate logistic regression adjusting for age and CHF status identified an association between rs7521023 with SCA (odds ratio [OR] 2.72, 95percent confidence interval [CI] 1.44-5.13, P = .002). The substantial impact of CHF on SCA in the model (OR 26.6, 95percent CI 13.40-52.70, P .001) led us to further examine the relationship between CHF, SCA, and CASQ2 SNPs. We identified 2 CASQ2 variants (rs7521023: OR 0.4, 95percent CI 0.25-0.76, P = .003; rs6684209: OR 19.8, 95percent CI 3.63-108.2, P .001) associated with CHF after adjusting for SCA, age, gender, and hypertension. Conclusion: We observed association between a CASQ2 polymorphism and SCA due to VA in patients with CAD adjusting for CHF and independent associations between CASQ2 SNPs and CHF adjusting for SCA. Further investigation in independent cohorts is needed to confirm these findings. © 2014 Heart Rhythm Society. All rights reserved.Adzhubei IA, 2010, NAT METHODS, V7, P248, DOI 10.1038-nmeth0410-248; Aouizerat BE, 2011, BMC CARDIOVASC DISOR, P11; Conde L, 2006, NUCLEIC ACIDS RES, V34, pW621, DOI 10.1093-nar-gkl071; Dekker LRC, 2006, CIRCULATION, V114, P1140, DOI 10.1161-CIRCULATIONAHA.105.606145; di Barletta MR, 2006, CIRCULATION, V114, P1012, DOI 10.1161-CIRCULATIONAHA.106.623793; Faggioni M, 2012, AM J PHYSIOL-HEART C, V302, pH1250, DOI 10.1152-ajpheart.00779.2011; Fishman GI, 2010, CIRCULATION, V122, P2335, DOI 10.1161-CIRCULATIONAHA.110.976092; Friedlander Y, 1998, CIRCULATION, V97, P155; Gavin MC, 2011, HEART RHYTHM, V8, P704, DOI 10.1016-j.hrthm.2011.01.003; Halder I, 2008, HUM MUTAT, V29, P648, DOI 10.1002-humu.20695; Hoggart CJ, 2003, AM J HUM GENET, V72, P1492, DOI 10.1086-375613; Jouven X, 1999, CIRCULATION, V99, P1978; Knollmann BC, 2006, J CLIN INVEST, V116, P2510, DOI 10.1172-JCI29128; Morrison AC, 2010, CIRC-CARDIOVASC GENE, V3, P248, DOI 10.1161-CIRCGENETICS.109.895995; Price AL, 2006, NAT GENET, V38, P904, DOI 10.1038-ng1847; PULLINGER CR, 1995, J CLIN INVEST, V95, P1225, DOI 10.1172-JCI117772; Refaat M, 2009, HEART RHYTHM, V6, pS456; Sato Y, 1998, J BIOL CHEM, V273, P28470, DOI 10.1074-jbc.273.43.28470; Sotoodehnia N, 2009, HEART RHYTHM, V6, P1306, DOI 10.1016-j.hrthm.2009.06.013; Sotoodehnia N, 2006, CIRCULATION, V113, P1842, DOI 10.1161-CIRCULATIONAHA.105.582833; Spooner PM, 2009, J CARDIOVASC ELECTR, V20, P585, DOI 10.1111-j.1540-8167.2008.01419.x; Stephens M, 2001, AM J HUM GENET, V68, P978, DOI 10.1086-319501; Terentyev D, 2006, CIRC RES, V98, P1151, DOI 10.1161-01.RES.0000220647.93982.08; Tian C, 2008, HUM MOL GENET, V17, pR143, DOI 10.1093-hmg-ddn268; Tomaselli GF, 2004, CIRC RES, V95, P754, DOI 10.1161-01.RES.0000145047.14691.db; Tseng ZH, 2009, HEART RHYTHM, V6, P1745, DOI 10.1016-j.hrthm.2009.08.031; Tseng ZH, 2009, HEART RHYTHM, V6, P1315, DOI 10.1016-j.hrthm.2009.06.034; Tseng ZH, 2008, HEART RHYTHM, V5, P814, DOI 10.1016-j.hrthm.2008.03.016; Villard E, 2011, EUR HEART J, V32, P1065, DOI 10.1093-eurheartj-ehr105; Watanabe J, 2006, EUR J HEART FAIL, V8, P237, DOI 10.1016-j.ejheart.2005.08.003; Westaway SK, 2011, CIRC-CARDIOVASC GENE, V4, P397, DOI 10.1161-CIRCGENETICS.111.959916; Wong CH, 2009, FORENSIC SCI INT, V192, P53, DOI 10.1016-j.forsciint.2009.07.019; Zheng ZJ, 2001, CIRCULATION, V104, P2158, DOI 10.1161-hc4301.098254; Zipes DP, 1998, CIRCULATION, V98, P233411
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