1,720,993 research outputs found

    sj-docx-3-wso-10.1177_17474930231203133 – Supplemental material for Iatrogenic cerebral amyloid angiopathy: A multinational case series and individual patient data analysis of the literature

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    Supplemental material, sj-docx-3-wso-10.1177_17474930231203133 for Iatrogenic cerebral amyloid angiopathy: A multinational case series and individual patient data analysis of the literature by Slaven Pikija, Janja Pretnar-Oblak, Senta Frol, Branko Malojcic, Thomas Gattringer, Kinga Rak-Frattner, Dimitre Staykov, Andrea Salmaggi, Riccardo Milani, Jozef Magdic, Sarah Iglseder, Eugen Trinka, Theo Kraus, Andreea Toma, Jacopo C. DiFrancesco, Payam Tabaee Damavandi, Natalia Fabin, Anna Bersano, Patricia de la Riva Juez, Ines Albajar Gomez, Benedetta Storti and Simon Fandler-Höfler in International Journal of Stroke</p

    sj-tif-2-wso-10.1177_17474930231203133 – Supplemental material for Iatrogenic cerebral amyloid angiopathy: A multinational case series and individual patient data analysis of the literature

    No full text
    Supplemental material, sj-tif-2-wso-10.1177_17474930231203133 for Iatrogenic cerebral amyloid angiopathy: A multinational case series and individual patient data analysis of the literature by Slaven Pikija, Janja Pretnar-Oblak, Senta Frol, Branko Malojcic, Thomas Gattringer, Kinga Rak-Frattner, Dimitre Staykov, Andrea Salmaggi, Riccardo Milani, Jozef Magdic, Sarah Iglseder, Eugen Trinka, Theo Kraus, Andreea Toma, Jacopo C. DiFrancesco, Payam Tabaee Damavandi, Natalia Fabin, Anna Bersano, Patricia de la Riva Juez, Ines Albajar Gomez, Benedetta Storti and Simon Fandler-Höfler in International Journal of Stroke</p

    sj-docx-1-wso-10.1177_17474930231203133 – Supplemental material for Iatrogenic cerebral amyloid angiopathy: A multinational case series and individual patient data analysis of the literature

    No full text
    Supplemental material, sj-docx-1-wso-10.1177_17474930231203133 for Iatrogenic cerebral amyloid angiopathy: A multinational case series and individual patient data analysis of the literature by Slaven Pikija, Janja Pretnar-Oblak, Senta Frol, Branko Malojcic, Thomas Gattringer, Kinga Rak-Frattner, Dimitre Staykov, Andrea Salmaggi, Riccardo Milani, Jozef Magdic, Sarah Iglseder, Eugen Trinka, Theo Kraus, Andreea Toma, Jacopo C. DiFrancesco, Payam Tabaee Damavandi, Natalia Fabin, Anna Bersano, Patricia de la Riva Juez, Ines Albajar Gomez, Benedetta Storti and Simon Fandler-Höfler in International Journal of Stroke</p

    Supplemental material for Therapeutic hypothermia for acute ischaemic stroke. Results of a European multicentre, randomised, phase III clinical trial

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    Supplemental Material for Therapeutic hypothermia for acute ischaemic stroke. Results of a European multicentre, randomised, phase III clinical trial by H Bart van der Worp, Malcolm R Macleod, Philip MW Bath, Raj Bathula, Hanne Christensen, Bridget Colam, Charlotte Cordonnier, Jacques Demotes-Mainard, Isabelle Durand-Zaleski, Christian Gluud, Janus Christian Jakobsen, Bernd Kallmünzer, Rainer Kollmar, Derk W Krieger, Kennedy R Lees, Dominik Michalski, Carlos Molina, Joan Montaner, Risto O Roine, Jesper Petersson, Richard Perry, Nikola Sprigg, Dimitre Staykov, Istvan Szabo, Geert Vanhooren, Joanna M Wardlaw, Per Winkel, Stefan Schwab and the EuroHYP-1 investigators in European Stroke Journal</p

    Posterior Reversible Encephalopathy Syndrome

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    Posterior reversible encephalopathy syndrome (PRES) is characterized by headache, altered mental status, visual disturbances, and seizures. Radiological features typically include edema of the posterior cerebral regions, especially of the parietooccipital lobes. Atypical imaging features, such as involvement of anterior cerebral regions, deep white matter, and the brain stem are also frequently seen. Vasoconstriction is common in vascular imaging. Different conditions have been associated with PRES, but toxemia of pregnancy, solid organ or bone marrow transplantation, immunosuppressive treatment, cancer chemotherapy, autoimmune diseases, and hypertension are most commonly described. The pathophysiology of PRES is unclear and different hypotheses are being discussed. Posterior reversible encephalopathy syndrome is best managed by monitoring and treatment in the setting of a neurointensive care unit. The prognosis is usually benign with complete reversal of clinical symptoms within several days, when adequate treatment is immediately initiated. Treatment of severe hypertension, seizures, and withdrawal of causative agents represent the hallmarks of specific therapy in PRES. Delay in diagnosis and treatment may lead to permanent neurological sequelae. Therefore, awareness of PRES is of crucial importance for the intensivist. </jats:p
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