1,721,014 research outputs found
Detection of Myocardial Fibrosis, Edema and Hypoperfusion in Hypertrophic Cardiomyopathy By Magnetic Resonance Imaging
No full textFine-needle aspiration biopsy in primary adrenal tumors. Clinical radiological and histological correlations.
No full textUnusual antefemoral dissecting cyst.
Full text linkCystic collections in the popliteal space (Baker's cysts)
are frequently observed in inflammatory joint diseases.
The causal mechanism is generally regarded as
entrapment of synovial fluid from the articular space in
the gastrocnemio-semimembranosus bursa (Doppman,
1965; Freiberger & Kay, 1979; Wilson et al, 1938); back
flow is prevented by a valve effect at the level of their
connection.
These cysts can remain localised in the popliteal
space or can give rise to dissections and/or rupture.
This usually occurs between the calf muscles but
occasionally the cyst may extend into the thigh
In comparison with these posterior dissecting cysts,
the finding of an anterior dissecting cyst originating
from the suprapatellar pouch is very rare
Myocardial Crypt in an Asymptomatic Young Athlete: How to Interpret?
No full textMyocardial crypts are extensions of blood signal penetrating the compact myocardium and are considered in literature as either a distinctive cardiac magnetic resonance (CMR) imaging marker for hypertrophic cardiomyopathy or as benign congenital malformations. What if CMR reveals a myocardial crypt in the presence of an altered ECG in an asymptomatic, enlarged young athlete's heart? The illustrated case demonstrates that new insights in CMR can also require further diagnostic interventions, which might have deleterious consequences for the individual athlete due to the uncertain interpretation of some findings in the demanding new world of a rapidly developing diagnostic imaging technique
Structural abnormalities and functional implications of predischarge ST-segment elevation after acute myocardial infarction
No full textMyocardial structural correlates of late persistence of ST-segment elevation in the subacute stage of myocardial infarction
No full textExtension and Transmural Entity of Myocardial Necrosis Are Mayor Determinants of diastolic deceleration time Impairment After Acute Myocardial Function Treated With Primary Coronary Angioplasty.
No full textAlström Syndrome: Cardiac Magnetic Resonance findings.
No full textBACKGROUND: Alström Syndrome (ALMS) is an extremely rare multiorgan disease caused by mutations in ALMS1. Dilated cardiomyopathy (DCM) is a common finding but only one series has been investigated by Cardiac Magnetic Resonance (CMR).
METHODS: Eight genetically proven ALMS patients (ages 11-41) underwent CMR performed by standard cine steady state, T1, T2 and late gadolinium enhancement (LGE) sequences. Ejection fraction (EF), Diastolic Volume (EDV) and Systolic Volume normalized for body surface area (ESV), and mass indices were determined, as well as EDV/Mass ratio, an index expressing the adequacy of cardiac mass to heart volume. Regional fibrosis was assessed by LGE; diffuse fibrosis was measured by a TI scout sequence acquired at 5, 10 and 15min after gadolinium by comparing inversion time values (TI) at null time in ALMS and control group.
RESULTS: In one patient severe DCM was present with diffuse LGE. There were seven cases without clinical DCM. In these patients, EF was at lower normal limits or slightly reduced and ESV index increased; six patients had decreased mass index and EDV/Mass ratio. Mild regional non ischemic fibrosis was detected by LGE in three cases; diffuse fibrosis was observed in all cases, as demonstrated by shorter TI values in ALMS in comparison with controls (5min: 152±12 vs 186±16, p 0.0002; 10min: 175±8 vs 204±18, p 0.0012; 15min: 193±9 vs 224±16, p 0.0002).
CONCLUSIONS: Cardiac involvement in ALMS is characterized by progressive DCM, associated with systolic dysfunction, myocardial fibrosis and reduced myocardial mass
Comparison between noninvasive transthoracic coronary flow reserve and contrast cardiac magnetic resonance to identify myocardial recovery after anterir reperfused myocardial infarction
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