1,721,120 research outputs found
Spatial synoptic classification approach to projected heat vulnerability in California under future climate change scenarios
No full text"February 17, 2011."; "Sponsoring/monitoring agency report number, ARB/R11-914"--Report documentation page.; Includes bibliographical references (p. 100-106).; Final report.; Prepared by Kent State University, Dept. of Geography and University of Miami, Dept. of Geography and Regional Studies under contract no.Excessive heat significantly impacts the health of Californians during irregular but intense heat events. Through the 21st century, a significant increase in impact is likely, as the state experiences a changing climate as well as an aging population. To assess this impact, future heat-related mortality estimates were derived for nine metropolitan areas in the state for the remainder of the century. First, oppressive weather events were predicted for future years by first correlating past surface weather types with circulation patterns, and then predicting them in the future using projections of future atmospheric circulation at three levels. Second, we estimated heat-related mortality by initially determining historical weather-type mortality relationships for each metropolitan area. These were then projected into the future based on predicted weather types. Estimates account for several levels of uncertainty: for each metropolitan area, mortality values are produced for five different climate model-scenarios, three different population estimates (along with a no-growth model), and two different levels of acclimatization (along with no acclimatization). Results show a significant increase in heat events over the 21st century, with oppressive weather types potentially more than doubling in frequency, and with heat events of two weeks or longer becoming up to ten times more common at coastal locations. Major urban centers could have a greater than tenfold increase in heat-related mortality in the over 65 age group by the 2090s
Health effects of exposure to environmental tobacco smoke : the report of the California Environmental Protection Agency /
No full text"M307"--P. [4] of cover."August 1999"--P. [4] of cover.Includes bibliographical references.Mode of access: Internet
Assessment of California\u27s Largest Hazardous Waste Generators\u27 Source Reduction Planning Efforts
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Near-Source Ambient Air Monitoring of Nicotine as A Marker for Environmrntal Tobacco Smoke
Get PDFThis report presents the results of near-source ambient air monitoring of nicotine as a marker for environmental tobacco smoke (ETS) in California. The objective of the ETS study is to measure ambient outdoor ETS, to which the public is exposed, as part of an evaluation by the Air Resources Board of ETS as a possible toxic air contaminant
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Proposed Identification of Environmental Tobacco Smoke as a Toxic Air Contaminant
Get PDFThis report provides a comprehensive assessment of the effects of environmental tobacco smoke (secondhand smoke) as of mid-2005 produced by the California Air Resources Board and Office of Environmental Health Hazard Assessment as part of the process of identifying ETS as a toxic air contaminant.Part A of the report includes information about the chemical composition of secondhand smoke and exposure of the public to secondhand smoke, including outdoor exposures.Part B of the report includes information on the health effects of secondhand smoke and concludes that the following health effects are causally associated iwth ETS exposure:Developmental Effects: Fetal growth: Low birth weight and decrease in birth weight, Sudden Infant Death Syndrome (SIDS), Pre-term DeliveryRespiratory Effects: Acute lower respiratory tract infections in children (e.g., bronchitis and pneumonia), Asthma induction and exacerbation in children and adults, Chronic respiratory symptoms in children, Eye and nasal irritation in adults, Middle ear infections in children,Carcinogenic Effects: Lung cancer, Nasal sinus cancer, Breast cancer in younger, primarily premenopausal womenCardiovascular Effects: Heart disease mortality, Acute and chronic coronary heart disease morbidity, Altered vascular propertiesPart C: The public comments and responses are available in a separate report, available at http://repositories.cdlib.org/tc/surveys/CALEPA2005C
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Proposed Identification of Environmental Tobacco Smoke as a Toxic Air Contaminant, Part C: Public Comments and ARB/OEHHA Staff Responses
Get PDFThis report is part of a comprehensive assessment of the effects of environmental tobacco smoke (secondhand smoke) as of mid-2005 produced by the California Air Resources Board and Office of Environmental Health Hazard Assessment as part of the process of identifying ETS as a toxic air contaminant. Part C contains all the public comments and responses to these comments by the Air Resources Board and Office of Environmental Health Hazard Assessment.The main body of the report (Parts A and B) is available at http://repositories.cdlib.org/tc/surveys/CALEPA2005
CALIFORNIA ENVIRONMENTAL PROTECTION AGENCY
No full textIn the summer and fall of 2006, the Department of Pesticide Regulation (DPR) sampled water and sediment from 23 marinas to assess the geographical scope and severity of pollution stemming from the use of antifouling paint (AFP) pesticides in California. Copper, zinc, Irgarol, M1 (the primary breakdown product of Irgarol), and aquatic toxicity were selected as potential indicators of AFP pollution. The highest dissolved copper concentrations were observed in larger salt water marinas along California’s Central and South Coast, the lowest were seen in freshwater lake marinas. Copper and zinc concentrations were almost always higher in the marinas than in the adjacent local reference sites, indicating that significant sources of metals existed in the marinas. Concentrations of zinc never exceeded California’s water quality standards. In contrast, concentrations of dissolved copper in salt and brackish water marinas were frequently above California water quality standards established for the protection of aquatic life. Developmental toxicity tests on the copper-sensitive embryo of the mussel Mytilus galloprovincialis were conducted on 47 marina water samples. Eight of these samples showed a statistically significant toxic response and copper was the likely cause of the toxicity. Several copper toxicity models that account for copper bioavailability to aquatic organisms were used to predict toxicity for the 517 samples for which sitespecifi
California Environmental Protection Agency
No full textPlease include the attached comments on the Office ofEnvironmental Health Hazard Assessment's (OEHHA) draft CrVI public health goal (PHG) released for public comment in August 2009. We believe that these comprehensive comments address the many weaknesses in the proposed PHG requiring further evaluation and appropriate change to the proposed PHG at this time. We further believe that the now ongoing research at The Hamner Institutes will result in the provision of critical scientific findings which may have significant impacts on the level and scientific credibility of the PHG for CrVl proposed by OEHHA. Since the Hamner Institutes r~search will be completed within the coming year we ask that OEHHA consider wahing to finalize this PHG until OEHHA has considered the findings ofthat research. Thank you for the opportunity to comment.. Sincerely, f'\J \ ~:--···~-~--~
EVIDENCE ON DEVELOPMENTAL AND REPRODUCTIVE TOXICITY OF METHYL TERTIARY-BUTYL ETHER Reproductive and Cancer Hazard Assessment Section Office of Environmental Health Hazard Assessment California Environmental Protection Agency DRAFT
No full textHealth and Safety Code 25249.5 et seq.) requires that the Governor cause to be published a list of those chemicals “known to the state ” to cause cancer or reproductive toxicity. The Act specifies that “a chemical is known to the state to cause cancer or reproductive toxicity … if in the opinion of the state’s qualified experts the chemical has been clearly shown through scientifically valid testing according to generally accepted principles to cause cancer or reproductive toxicity. ” The lead agency for implementing Proposition 65 is the Office of Environmental Health Hazard Assessment (OEHHA) of the California Environmental Protection Agency. The “state’s qualified experts ” regarding findings of reproductive toxicity are identified as the members of the Developmental and Reproductive Toxicant (DART) Identification Committee of OEHHA’s Science Advisory Board (22 CCR 12301). The Local Drinking Water Protection Act of 1997 (SB1189 and AB592) requires that th
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