75,627 research outputs found

    Butler, L M, 1501976

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    This record was harvested from a previous catalogue system and will be withdrawn in 2025. Information in this record may be superseded or incomplete. Visit this record in UMA's new catalogue at: https://archives.library.unimelb.edu.au/nodes/view/375148Surname: BUTLER Given Name(s) or Initials: L M Military Service Number or Last Known Location: 1501976 Missing, Wounded and Prisoner of War Enquiry Card Index Number: 47915187556 Item: [2016.0049.07456] "Butler, L M, 1501976

    Butler Residence

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    Drawing from a set of 3 sheets of construction drawings of the Butler residence project, showing right side and front elevations.Pencil on vellu

    Butler groups of infinite rank

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    AbstractFor a fixed proper subgroup R (of type t) of the group Q of rational numbers, a torsion-free group A is called an R-group if it satisfies Bext1(A, R) = 0, where Bext stands for the set of balanced extensions. Those R-groups whose nonzero elements are of types ≤t are investigated. In the constructible universe L, these R-groups (up to cardinality ℵω) turn out to coincide with those A for which the group Ǎ = A ⊗ R0 is a Butler group; here R0 denotes the largest subgroup of R of idempotent type t0 ≤ t. This claim is false in models of set theory in which Shelah's Proper Forcing Axiom holds

    Author correction: obesity and ethnicity alter gene expression in skin

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    Daniel Butler was omitted from the author list in the original version of this Article. The Author contributions section now reads: “J.M.W. designed, conducted, and contributed to the writing of the manuscript, prepared Fig. 1. S.G. evaluated and did statistical analysis on the skin and fat samples, prepared Figs. 2–9. J.O.A. evaluated and contributed to writing the manuscript. D.B prepared and sequenced DNA libraries for the skin microbiota data, and wrote the applicable parts of the methods section. C.M. analyzed and wrote up the skin microbiota data, prepared Fig. 10. All authors have read the manuscript and approved its contents. D.D. analyzed and wrote up the skin microbiota data. S.Z. ran and analyzed the skin metabolite data. J.S. assisted in design, analysis and wrote up the skin metabolite data. J.K. assisted in analysis write up of skin and fat data. J.L.B. assisted in analysis, interpretation and writing of the manuscript. P.R.H. designed, analyzed, interpreted the data, and was the primary author of the manuscript.” This has been corrected in the PDF and HTML versions of the Article, and in the accompanying Supplementary Information file.</p

    An investigation into changes in immunogenic proteins associated with cold temperature adaptation in L. monocytogenes

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    Listeria monocytogenes can survive under different stress conditions and its virulence appears to increase when exposed to stress factors used to control its growth. The objective of this study was to identify immunogenic proteins (IP) of L. monocytogenes associated with adaptation to cold temperature. L. monocytogenes cultures were grown at 12 and 37°C (control). Bacterial cells were collected during late exponential growth phase. The proteins were extracted, purified, quantified by BCA method, in-gel trypsin digested and analyzed by nLC-MS/MS. Proteins were identified with at least 2 peptides per protein against the L. monocytogenes Uniprot database. The IP were predicted by an in silico analysis approach. The proteins were sublocalized (Cello v2.4, SignalP 3.9) considering for the immunogenic prediction (VirulentPred) of only the non-cytosolic proteins. The IP were clusterized by STRING v11.05, applying a minimum required interaction score equal to 0.7. As result, two main different networks were determined at 12 and 37°C. In response to low temperature, proteins associated with cell motility (FliM), chemotaxis (lmo0723) and oxidative stress (sod, trxB) were identified. Moreover the above mentioned proteins are absent at 37°C. At optimal growth condition, L. monocytogenes codified for several virulence factors associated to adherence of epithelial cells (Iap) and their invasion to colonize the host gastrointestinal tract (lmo1422 and lmo0129). These results were similar to previous literature reports. the datasets obtained can be useful for further studies on listeriosis pathogenesis and role of environmental stress in determining the virulence factors expression of different strains

    On a class of locally Butler groups

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    summary:A torsionfree abelian group BB is called a Butler group if Bext(B,T)=0Bext(B,T) = 0 for any torsion group TT. It has been shown in [DHR] that under CHCH any countable pure subgroup of a Butler group of cardinality not exceeding ω\aleph_\omega is again Butler. The purpose of this note is to show that this property has any Butler group which can be expressed as a smooth union α<μBα\cup_{\alpha < \mu}B_\alpha of pure subgroups BαB_\alpha having countable typesets

    Review of "Le Temps des �����criveuses���: L���Oeuvre pionni��re des ��pistoli��res au XVIIe si��cle" by Larry W. Riggs, Butler University.

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    Nathalie Freidel. Le Temps des �����criveuses���: L���Oeuvre pionni��re des ��pistoli��res au XVIIe si��cle. Paris: Classiques Garnier, 2021. 290pp. 32���. Review by Larry W. Riggs, Butler Universit

    Almost Butler groups

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    summary:Generalizing the notion of the almost free group we introduce almost Butler groups. An almost B2B_2-group GG of singular cardinality is a B2B_2-group. Since almost B2B_2-groups have preseparative chains, the same result in regular cardinality holds under the additional hypothesis that GG is a B1B_1-group. Some other results characterizing B2B_2-groups within the classes of almost B1B_1-groups and almost B2B_2-groups are obtained. A theorem of stating that a group GG of weakly compact cardinality λ\lambda having a λ\lambda -filtration consisting of pure B2B_2-subgroup is a B2B_2-group appears as a corollary
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