20,110 research outputs found
Hemigyrus Brunner von Wattenwyl 1893
No full textGenus Hemigyrus Brunner von Wattenwyl, 1893 Type species: Hemigyrus (Hemigyrus) amplus Brunner von Wattenwyl, 1893Published as part of Xie, Hui-Cong, Wang, Han-Qiang, Zong, Jing-Song, Li, Kai & He, Zhu-Qing, 2022, Taxonomic revision of the genus Hemigyrus Brunner von Wattenwyl, 1893 (Orthoptera: Tettigoniidae: Pseudophyllinae), pp. 97-115 in Zootaxa 5092 (1) on page 101, DOI: 10.11646/zootaxa.5092.1.5, http://zenodo.org/record/586967
Hemigyrus (Hemigyrus) amplus : Brunner von Wattenwyl 1893
No full text2.2 Hemigyrus (Hemigyrus) amplus Brunner von Wattenwyl, 1893 Figs.7A–H Hemigyrus amplus: Brunner von Wattenwyl, 1893, Annali del Museo Civico di Storia Naturale di Genova Serie 2, 13 (33): 175; Brunner von Wattenwyl, 1895, Monographie der Pseudophylliden, 45: 47; Kirby, 1906, A Synonymic Catalogue of Orthoptera (Orthoptera Saltatoria, Locustidae vel Acridiidae), 2: 297; Beier, 1954, Revision der Pseudophyllinen, 144; Jin & Xia, 1994, Journal of Orthoptera Research, 3: 24. Hemigyrus (Hemigyrus) amplus: Ingrisch & Shishodia, 1998, Mitteilungen der Schweizerischen Entomologischen Gesellschaft, 71: 367; Shishodia, Chandra & Gupta, 2010, Records of the Zoological Survey of India, 314: 315.Published as part of Xie, Hui-Cong, Wang, Han-Qiang, Zong, Jing-Song, Li, Kai & He, Zhu-Qing, 2022, Taxonomic revision of the genus Hemigyrus Brunner von Wattenwyl, 1893 (Orthoptera: Tettigoniidae: Pseudophyllinae), pp. 97-115 in Zootaxa 5092 (1) on page 106, DOI: 10.11646/zootaxa.5092.1.5, http://zenodo.org/record/586967
Hemigyrus (Hemigyrus) acutifolius : Brunner von Wattenwyl 1895
No full text2.4 Hemigyrus (Hemigyrus) acutifolius Brunner von Wattenwyl, 1895 Figs. 9A–H Hemigyrus acutifolius: Brunner von Wattenwyl, 1895, Verhandlungen der Kaiserlich-Königlichen Zoologisch-Botanischen Gesellschaft in Wien, 45: 48; Kirby, 1906, A Synonymic Catalogue of Orthoptera (Orthoptera Saltatoria, Locustidae vel Acridiidae), 2: 297; Beier, 1954, Revision der Pseudophyllinen, 146; Liana, 1999, Bulletin of the Museum and Institute of Zoology PAS, 2: 62. Hemigyrus (Hemigyrus) acutifolius: Gorochov & Voltshenkova, 1998, Zoologicheskii Zhurnal, 77 (7): 860.Published as part of Xie, Hui-Cong, Wang, Han-Qiang, Zong, Jing-Song, Li, Kai & He, Zhu-Qing, 2022, Taxonomic revision of the genus Hemigyrus Brunner von Wattenwyl, 1893 (Orthoptera: Tettigoniidae: Pseudophyllinae), pp. 97-115 in Zootaxa 5092 (1) on page 107, DOI: 10.11646/zootaxa.5092.1.5, http://zenodo.org/record/586967
Brunner syndrome associated MAOA mutations result in NMDAR hyperfunction and increased network activity in human dopaminergic neurons
No full textMonoamine neurotransmitter abundance affects motor control, emotion, and cognitive function and is regulated by monoamine oxidases. Among these, Monoamine oxidase A (MAOA) catalyzes the degradation of dopamine, norepinephrine, and serotonin into their inactive metabolites. Loss-of-function mutations in the X-linked MAOA gene have been associated with Brunner syndrome, which is characterized by various forms of impulsivity, maladaptive externalizing behavior, and mild intellectual disability. Impaired MAOA activity in individuals with Brunner syndrome results in bioamine aberration, but it is currently unknown how this affects neuronal function, specifically in dopaminergic (DA) neurons. Here we generated human induced pluripotent stem cell (hiPSC)-derived DA neurons from three individuals with Brunner syndrome carrying different mutations and characterized neuronal properties at the single cell and neuronal network level in vitro. DA neurons of Brunner syndrome patients showed reduced synaptic density but exhibited hyperactive network activity. Intrinsic functional properties and α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor (AMPAR)-mediated synaptic transmission were not affected in DA neurons of individuals with Brunner syndrome. Instead, we show that the neuronal network hyperactivity is mediated by upregulation of the GRIN2A and GRIN2B subunits of the N-methyl-d-aspartate receptor (NMDAR), resulting in increased NMDAR-mediated currents. By correcting a MAOA missense mutation with CRISPR/Cas9 genome editing we normalized GRIN2A and GRIN2B expression, NMDAR function and neuronal population activity to control levels. Our data suggest that MAOA mutations in Brunner syndrome increase the activity of dopaminergic neurons through upregulation of NMDAR function, which may contribute to the etiology of Brunner syndrome associated phenotypes.</p
Duodenal Adenocarcinoma of Brunner Gland Origin: A Case Report
No full textWe report a case of adenocarcinoma originating from the duodenal Brunner glands in a 47-year-old female patient. The lesion was 0.8 cm in extent and located at the posterior wall of the first part of the duodenum. Histologically, the tumor showed transition from non-neoplastic Brunner glands through dysplastic epithelium into adenocarcinoma. The carcinoma cells were strongly positive for MUC6 protein, which is an epithelial marker for the Brunner glands. Tumor protein p53 was overexpressed in the carcinoma cells, but not in the non-neoplastic or dysplastic epithelium. Dystrophic calcification was predominant. This is the first case report of duodenal adenocarcinoma of Brunner gland origin in Korea
FIGURE 13 in Taxonomic revision of the genus Hemigyrus Brunner von Wattenwyl, 1893 (Orthoptera: Tettigoniidae: Pseudophyllinae)
No full textFIGURE 13. Distribution of Hemigyrus in the world.Published as part of Xie, Hui-Cong, Wang, Han-Qiang, Zong, Jing-Song, Li, Kai & He, Zhu-Qing, 2022, Taxonomic revision of the genus Hemigyrus Brunner von Wattenwyl, 1893 (Orthoptera: Tettigoniidae: Pseudophyllinae), pp. 97-115 in Zootaxa 5092 (1) on page 114, DOI: 10.11646/zootaxa.5092.1.5, http://zenodo.org/record/586967
FIGURE 1 in Taxonomic revision of the genus Hemigyrus Brunner von Wattenwyl, 1893 (Orthoptera: Tettigoniidae: Pseudophyllinae)
No full textFIGURE 1. The phylogenetic relationship among Hemigyrus species inferred from the COI fragments. The SH-aLRT value and the bootstrap value are indicated.Published as part of Xie, Hui-Cong, Wang, Han-Qiang, Zong, Jing-Song, Li, Kai & He, Zhu-Qing, 2022, Taxonomic revision of the genus Hemigyrus Brunner von Wattenwyl, 1893 (Orthoptera: Tettigoniidae: Pseudophyllinae), pp. 97-115 in Zootaxa 5092 (1) on page 100, DOI: 10.11646/zootaxa.5092.1.5, http://zenodo.org/record/586967
Dataset to support the article "High-resolution 𝜙-OFDR using phase unwrap and nonlinearity suppression"
No full textThis dataset is used for realizing high resolution of phase-sensitive Optical Frequency Domain Reflectometer. It is associated with the research paper:
Guo Z, Yan J, Han G, Yu Y, Greenwood D and Marco J (2023) "High-Resolution φ-OFDR Using Phase Unwrap and Nonlinearity Suppression". Journal of Lightwave Technology, 41 (9), 2885-2891. (https://doi.org/10.1109/JLT.2023.3236775).
The data is presented as an excel file:
High_resolution_OFDR_using_phase_unwrap_and_nonlinearity_suppression.xlsx
This work was funded by High Value Manufacturing Catapult and the Engineer and Physical Sciences Research Council - EPSRC EP/V000624/1. The author Gaoce Han would like to acknowledge the China Scholarship Council for sponsoring.</span
Brunner syndrome associated MAOA dysfunction in human dopaminergic neurons results in NMDAR hyperfunction and increased network activity
No full textBackground Monoamine neurotransmitter abundance affects motor control, emotion, and cognitive function and is regulated by monoamine oxidases. Amongst these, monoamine oxidase A (MAOA) catalyzes the degradation of dopamine, norepinephrine, and serotonin into their inactive metabolites. Loss-of-function mutations in the X-linked MAOA gene cause Brunner syndrome, which is characterized by various forms of impulsivity, maladaptive externalizing behavior, and mild intellectual disability. Impaired MAOA activity in individuals with Brunner syndrome results in bioamine aberration, but it is currently unknown how this affects neuronal function.Methods We generated human induced pluripotent stem cell (hiPSC)-derived dopaminergic (DA) neurons from three individuals with Brunner syndrome carrying different mutations, and used CRISPR/Cas9 mediated homologous recombination to rescue MAOA function. We used these lines to characterize morphological and functional properties of DA neuronal cultures at the single cell and neuronal network level in vitro.Results Brunner syndrome DA neurons showed reduced synaptic density but hyperactive network activity. Intrinsic functional properties and α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor (AMPAR)-mediated synaptic transmission were not affected by MAOA dysfunction. Instead, we show that the neuronal network hyperactivity is mediated by upregulation of the GRIN2A and GRIN2B subunits of the N-methyl-D-aspartate receptor (NMDAR), and rescue of MAOA results in normalization of NMDAR function as well as restoration of network activity.Conclusions Our data suggest that MAOA dysfunction in Brunner syndrome increases activity of dopaminergic neurons through upregulation of NMDAR function, which may contribute to Brunner syndrome associated phenotypes.</p
Phaneroptera nigroantennata Brunner
No full textPhaneroptera nigroantennata Brunner von Wattenwyl, 1878 (Plate 3 a-f) Phaneroptera nigroantennata Brunner, 1878, Monogr. Phaneropt.: 215 (Holotype, M, origi. descrip., Type locality: Eurasia: Japan; Type depository: Museum für Naturkunde der Humboldt-Universität, Berlin, Germany). Phaneroptera nakanoensis Matsumura & Shiraki, 1908, Jour. Coll. Agric. Tohoku Imp. Univ. 3 (1): 21; Ragge, 1957, Proc. R. Ent. Soc. London (B) 26: 123 (syn.). Redescription: Male stridulatory file sinuate, with about 130 distinct densely spaced stridulatory teeth, which are becoming larger mediad (Plate 3 c). Material examined: China: Zhejiang Prov.: 4 males, 3 females, Tianmushan[Mt.], 1947. VIII. 16 –XI. 15, Coll. Unknown (IZAS); China: Hubei Prov.: 1 male, 1 female, Honghua, 860m, 1981. VIII.22, 1 male, 1 female, Dajiuhu, 1800m, 1981. VIII. 4, Coll. Han Yinheng (IZAS); 1 female, China: Anhui: Huangshan [Mt.], Wenquan, 1980. X. 26, Coll. Du (MSIE); 1 male, China: Hunan Prov.: Dayong, 1985, Coll. Zhang Mingcai (MSIE); 2 males, China: Anhui Prov.: Huoshan, Foziceng, 1964. IX. 16, Coll. Jin Gentao (MSIE); 2 males, 1 female, China: Anhui Prov.: Huangshan, 500m – 650m, Coll. Jin Gentao (MSIE). Distribution: China (including Taiwan Island); Japan (Ragge 1956; Huang 2004).Published as part of Liu, Chun-Xiang, 2011, Phaneroptera Serville and Anormalous gen. nov. (Orthoptera: Tettigoniidae: Phaneropterinae) from China, with description of two new species, pp. 60-68 in Zootaxa 2979 on page 65, DOI: 10.5281/zenodo.20536
- …
