101,481 research outputs found
Applying analytical and numerical methods for the analysis of microcrystalline silicon solar cells
Brammer T, Stiebig H. Applying analytical and numerical methods for the analysis of microcrystalline silicon solar cells. Solar Energy Materials & Solar Cells. 2006;90(18-19):3021-3030
The application of grating couplers in thin-film silicon solar cells
Stiebig H, Senoussaoui N, Brammer T, Müller J. The application of grating couplers in thin-film silicon solar cells. Solar Energy Materials & Solar Cells. 2006;90(18-19):3031-3040
Thin-film solar cells with periodic grating coupler
Senoussaoui N, Krause M, Müller J, Bunte E, Brammer T, Stiebig H. Thin-film solar cells with periodic grating coupler. Thin solid films. 2004;451-452:397-401
Letter, [Author unclear] to Paulina T. Merritt
Handwritten letter to Paulina Merritt from an unknown author, October 1, 1876.
Defect density and recombination lifetime in microcrystalline silicon absorbers of highly efficient thin-film solar cells determined by numerical device simulations
Brammer T, Stiebig H. Defect density and recombination lifetime in microcrystalline silicon absorbers of highly efficient thin-film solar cells determined by numerical device simulations. J. Appl. Physics. 2003;94(2):1035-1042.The absorber layers of microcrystalline silicon thin-film solar cells with p-i-n structure deposited by plasma-enhanced chemical vapor deposition at 200degreesC are characterized regarding the defect density and the recombination lifetime. The characterization is based on a comparison of experimentally determined solar cell characteristics with results from numerical device simulations. Evaluation of the dark reverse saturation current indicates a strong dependence of the recombination lifetime tau on the hydrogen dilution during the deposition. Close to the transition region to amorphous growth, where the highest solar cell efficiencies are observed, tau is maximum within the crystalline deposition regime and equals around 80 ns. The aspect of a spatially varying defect density within the absorber layer is also addressed by numerical simulations. The results from the analysis of the dark current are compared with electron spin resonance data determined on single layers, which allows conclusions to be drawn regarding the capture cross section of the dominant recombination site in microcrystalline silicon. (C) 2003 American Institute of Physics
Avaliação de acessos de Aegilops tauschii quanto ao recrescimento das raízes em solução nutritiva após exposição ao alumínio.
Área: Melhoramento, Aptidão Industrial e Sementes
C-H bonds are not elongated by coordination to lanthanide metals: Single-crystal neutron diffraction structures of (C5Me5)Y(OC6H3(t)Bu2)CH(SiMe3)2 at 20 K and (C5Me5)LaCH(SiMe3)22 at 15 K [2]
Análise citogenética de híbridos entre Triticum durum x Aegilops tauschii com potencial uso em programas de melhoramento genético de trigo.
Editores técnicos: Joseani Mesquita Antunes, Ana Lídia Variani Bonato, Márcia Barrocas Moreira Pimentel
Handwritten biographical information on Paulina T. McClung Merritt
A handwritten biography of Paulina T. McClung Merritt by an unknown author, 1892.
Heterogeneous and tissue-specific regulation of effector T cell responses by IFN-gamma during Plasmodium berghei ANKA infection.
IFN-γ and T cells are both required for the development of experimental cerebral malaria during Plasmodium berghei ANKA infection. Surprisingly, however, the role of IFN-γ in shaping the effector CD4(+) and CD8(+) T cell response during this infection has not been examined in detail. To address this, we have compared the effector T cell responses in wild-type and IFN-γ(-/-) mice during P. berghei ANKA infection. The expansion of splenic CD4(+) and CD8(+) T cells during P. berghei ANKA infection was unaffected by the absence of IFN-γ, but the contraction phase of the T cell response was significantly attenuated. Splenic T cell activation and effector function were essentially normal in IFN-γ(-/-) mice; however, the migration to, and accumulation of, effector CD4(+) and CD8(+) T cells in the lung, liver, and brain was altered in IFN-γ(-/-) mice. Interestingly, activation and accumulation of T cells in various nonlymphoid organs was differently affected by lack of IFN-γ, suggesting that IFN-γ influences T cell effector function to varying levels in different anatomical locations. Importantly, control of splenic T cell numbers during P. berghei ANKA infection depended on active IFN-γ-dependent environmental signals--leading to T cell apoptosis--rather than upon intrinsic alterations in T cell programming. To our knowledge, this is the first study to fully investigate the role of IFN-γ in modulating T cell function during P. berghei ANKA infection and reveals that IFN-γ is required for efficient contraction of the pool of activated T cells
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