47 research outputs found
Neopostmodrinism, or, Dieser Rasen ist kein Hundeklo, or, Gub²rzub² number 6, or, The incognita of Rita\u27s deep time coexisting within central discoveries of the thermodynamic dichotomy of western thought : observed impregnant meanings & transhistorical justifications
No full text[102] pages : illustrations . Alternate Titles: Neopostmodrinism; Neopostmodernism; Dieser Rasen ist kein Hundeklo. Half title: Neopostmodernism, or, Dieser Rasen ist kein Hundeklo, or, Gabberjab number 6. Series: Hamady, Walter. Interminable gabberjabb ; 6. No. 6 of the author\u27s Interminable gabberjabb series. Limited edition of 125 copies. Signatures: [1]⁴ [2]¹² [3]⁸ [4]¹⁰ [5]⁸ [6]¹⁰. Page count runs from [1]₁b to [6]₁₀a. [2]₉ and [2]₁₀, though joined to form a double leaf, have printing on one of the facing surfaces.https://digitalcommons.risd.edu/specialcollections_artistsbooks/1501/thumbnail.jp
Effects of workload variation on the energy distribution in a mobile platform
No full textContinuous advancements in integrated circuit design and the constant development of a diverse set of computing applications, paved the way for versatile mobile devices that we use on daily basis. The dependency of these devices on battery as the main power source imposed strict constraints on the energy consumption of applications that run on these mobile devices. In this paper, we present a component-wise energy consumption breakdown of a modern platform under a variety of workloads and benchmarks. This study provides useful insights into the energy consumption profile of various hardware components, and the manner in which the operating system uses them. It is a crucial step in pointing future research towards the power-hungry components and providing a better understanding of the system's behavior, featuring a baseline to analyze the tradeoffs that system designers can use to optimize the upcoming platforms given the physical considerations and the practical constraints. Our results demonstrate a substantial variation in the energy contribution of various components as well as the total system energy depending on the nature of the workload. © 2013 Elsevier B.V. All rights reserved.Chinn, 2003, INTEL TECHNOLOGY J; Gatti Franco, 2002, CASES; Geer D, 2005, COMPUTER, V38, P11, DOI 10.1109-MC.2005.160; Hamady Faisal, 2011, P INT C EN AW COMP 2; Kothuru Hemanth JS, 2010, P 6 ANN GRASP S WICH; Mahesri Aqeel, 2004, WORKSH POW AW COMP S; Parkhurst Jeff, 2006, P 2006 IEEE ACM INT; Robert Graybill, 2002, POWER AWARE COMPUTIN; Zedlewski John, 2003, FILE STORAGE TECHNOL0
Reversing the Brain Drain
No full textThe increase in the brain drain from poor countries to more affluent ones is having a dramatic negative impact on the prospects for the economic development of the affected countries. One strategy that is being used to fight it, is called the “return” strategy, and consists of enticing those expatriates who have left to return and contribute to the economic development of their country of origin.
This paper argues that human resource managers in the West African country of Senegal can play a pivotal role in their country’s strategy to attract and retain professional expatriates.
Using the Four Frames Model from Bolman and Deal (2003) as a theoretical framework, the author collected much of his data from a survey that was distributed to human resource managers in Senegal. The data was, then, analyzed from the human resource, political, and structural perspectives of the Bolman and Deal model.
This study finds that Senegalese human resource managers are by and large competent, eager, and willing to play their role in the strategy to attract Senegalese professional expatriates, but their effectiveness is being hindered by issues ranging from an unfair promotion system, to inadequate organizational structures, and to very difficult macroeconomic conditions.
Some of the entities that may wish to use the findings of this research include policymakers in the government of Senegal, Senegalese private sector executives, non-profit organizations and development agencies interested in human capacity building in developing countries, and researchers in the field of management
The use of probiotics to prevent diarrhea in young children attending child care centers: a review
No full textThe incidence of diarrheal disease in children has been reduced because of public health measures, improved hygiene, and a better understanding of nutrition. However, it remains a particular problem where young children come into close contact with other children, such as in child day care centers. Probiotics are defined as products that contain an adequate dose of live microbial agents that have been shown in target-host studies to confer a health benefit. They have been used for the treatment and prevention of many diseases, but particularly of gastrointestinal diseases. Prebiotics are inactive food components, commonly oligosaccharides or polysaccharides, that stimulate growth of beneficial bacteria in the gastrointestinal tract and are commonly used in combination with probiotics. The initial searches identified 5860 articles from the PubMed database, but only 154 included the keyword "trial." Probiotics share the problem of limited systematic research with other traditional medications and foods, and only seven studies were included in the final analysis. A variety of probiotic organisms and prebiotics were used in the studies, and the end points were not standardized. However, examination of the six studies that used live cultures showed that five studies resulted in a decrease in either the number of episodes or the duration of diarrhea or both. However, the studies support a reduction of around 20% in diarrheal episodes or days of illness. Findings of this review have important implications for working parents. The regular use of a probiotic or probiotic/prebiotic combination will reduce the incidence and duration of diarrheal disease in children attending childcare centers (risk ratio, 0.72-0.82). Further research is needed to better define the most effective probiotic organisms and the optimal dosage
Correction to: Clinical and endovascular practice in interventional radiology: a contemporary European analysis
No full textUpon publication of this article (Kok et al., 2018), the authors noticed that author ‘Miguel Casares Santiago’ was spelt incorrectly. This has been corrected by means of this correction article
High-Fat Diet : Bacteria Interactions Promote Intestinal Inflammation Which Precedes and Correlates with Obesity and Insulin Resistance in Mouse
No full textBackground: Obesity induced by high fat (HF) diet is associated with inflammation which contributes to development of insulin resistance. Most prior studies have focused on adipose tissue as the source of obesity-associated inflammation. Increasing evidence links intestinal bacteria to development of diet-induced obesity (DIO). This study tested the hypothesis that HF western diet and gut bacteria interact to promote intestinal inflammation, which contributes to the progression of obesity and insulin resistance. Methodology/Principal Findings: Conventionally raised specific-pathogen free (CONV) and germ-free (GF) mice were given HF or low fat (LF) diet for 2-16 weeks. Body weight and adiposity were measured. Intestinal inflammation was assessed by evaluation of TNF-alpha mRNA and activation of a NF-kappa B-EGFP reporter gene. In CONV but not GF mice, HF diet induced increases in body weight and adiposity. HF diet induced ileal TNF-alpha mRNA in CONV but not GF mice and this increase preceded obesity and strongly and significantly correlated with diet induced weight gain, adiposity, plasma insulin and glucose. In CONV mice HF diet also resulted in activation of NF-kappa B-EGFP in epithelial cells, immune cells and endothelial cells of small intestine. Further experiments demonstrated that fecal slurries from CONV mice fed HF diet are sufficient to activate NF-kappa B-EGFP in GF NF-kappa B-EGFP mice. Conclusions/Significance: Bacteria and HF diet interact to promote proinflammatory changes in the small intestine, which precede weight gain and obesity and show strong and significant associations with progression of obesity and development of insulin resistance. To our knowledge, this is the first evidence that intestinal inflammation is an early consequence of HF diet which may contribute to obesity and associated insulin resistance. Interventions which limit intestinal inflammation induced by HF diet and bacteria may protect against obesity and insulin resistance
Effects of probiotics in patients with diabetes mellitus type 2 : study protocol for a randomized, double-blind, placebo-controlled trial
No full textBackground:
Low grade chronic inflammation is observed in patients with type 2 diabetes mellitus (T2DM). Endotoxin derived from gut bacteria may act as a potent inflammatory stimulant. Probiotics, which are believed to contain health promoting live microorganisms, may influence circulating endotoxin levels. Ingestion of live probiotic cultures may alter gut microbiota in a beneficial manner to reduce inflammation; no information is available whether or not they do so in patients with T2DM. Therefore, the aim of this study is to characterize the beneficial effects of probiotics on circulating endotoxin levels and other biomarkers related to systemic low-grade inflammation in patients with T2DM.
Methods:
One hundred and twenty consenting adult Saudi T2DM patients (naïve or newly diagnosed and without co-morbidities) will be enrolled in this clinical trial and randomized to receive daily placebo or probiotics (Ecologic®Barrier) for 26 weeks in a double-blind manner. Inflammatory and metabolic markers will be measured and fecal samples analyzed. Measurements/samples will be obtained at baseline and after 4, 8, 12/13 and 26 weeks of treatment.
Discussion:
It is expected that the probiotic product will induce beneficial changes in gut microbiota, reduce the systemic inflammatory state through altering systemic endotoxin levels and, as such, reduce the systemic inflammatory response observed in T2DM subjects.
Trial registration: ClinicalTrials.gov Identifier: NCT0176551
High-throughput clone library analysis of the mucosa-associated microbiota reveals dysbiosis and differences between inflamed and non-inflamed regions of the intestine in inflammatory bowel disease
No full textPeer reviewe
