232 research outputs found
Amos Stagg Biography
This is a brief biography of Springfield College faculty member and alumnus Alonzo Amos Stagg. An All-American Yale player, Amos Alonzo Stagg (1862-1965) brought football to the YMCA Training College (now Springfield College) and coached the institution’s first team in 1891. This document is most likely written and created by someone at Springfield College, but the exact author is unknown.For more information on Amos Alonzo Stagg, see: https://springfield.as.atlas-sys.com/agents/people/661Paper is fragile
Letter to Amos Alonzo Stagg from the New York Athletic Club not dated
Letter to Amos Alonzo Stagg from the New York Athletic Club. The letter is not dated and the author's name is not readable. The letter states the regrets that a team cannot be formed in time to play in early October, but possible later in the season. The author addresses Stagg as "Lonny." The letter is part of a series of letters received by Stagg regarding arrangements to play Springfield College in Football.For more information on Amos Alonzo Stagg, see: https://springfield.as.atlas-sys.com/agents/people/661Brackets and question marks in the text field represent words or phrases that were not readable due to the authors handwriting. The envelope for this item exists. To see envelope, click here: http://cdm16122.contentdm.oclc.org/cdm/compoundobject/collection/p15370coll2/id/14464/rec/
Letter to Amos Alonzo Stagg from the New York Athletic Club not dated
Letter to Amos Alonzo Stagg from the New York Athletic Club. The letter is not dated and the author's name is not readable. The letter states the team has an open date on October 24, 1891 since a game with the Princeton Football team cannot be played at that time. The money offered for the game is $100 or one-third of the take. The author addresses Stagg as "Lonny." The letter is part of a series of letters received by Stagg regarding arrangements to play Springfield College in Football.For more information on Amos Alonzo Stagg, see: https://springfield.as.atlas-sys.com/agents/people/661Brackets and question marks in the text field represent words or phrases that were not readable due to the authors handwriting. The envelope for this item is not available
Letter to Amos Alonzo Stagg from Weslyan University dated September 23, 1891
Letter to Amos Alonzo Stagg from the Weslyan University Foot Ball Association dated September 23, 1891 asking if October 10, 1891 is free for a game and offering $50 from the receipts. The author of the letter is thought to F.W. Taskaberry, but the writing is hard to read and this transcription might be inaccurate. The letter is part of a series of letters received by Stagg regarding arrangements to play Springfield College in Football.For more information on Amos Alonzo Stagg, see: https://springfield.as.atlas-sys.com/agents/people/661Brackets and question marks in the text field represent words or phrases that were not readable due to the authors handwriting. The envelope for this item exists. To see envelope, click here
Ruimte rond (grotere) bedden
Rapport van de STAGG-studiegroep 'Vloeroppervlakten in relatie tot kosten'Architectur
Amos Alonzo Stagg\u27s Contributions to Athletics
This thesis is a collection of Mr. Stagg\u27s contributions to athletics covering a period of the last sixty-six years, partly as a participant, but largely as a coach, director of physical education, and as a leader of men.
Herein the author wishes to bring tribute to this great man who has contributed more to the field of athletics and the development of manhood than could be written hare in the form of facts and figures.
His achievements and inventions in the various phases of athletics speak for themselves and will go down in the archives representing this field of endeavor.
As a true Christian leader Mr. Stagg has created an enviable character that has left its imprint on many thousands of men whom he has guided through college and university careers. Through this truly great influence on men, his ideals and code of ethics will be felt throughout the world for many centuries to come
Author response
We previously demonstrated that network level functional connectivity in the human brain could be related to levels of inhibition in a major network node at baseline (Stagg et al., 2014). In this study, we build upon this finding to directly investigate the effects of perturbing M1 GABA and resting state functional connectivity using transcranial direct current stimulation (tDCS), a neuromodulatory approach that has previously been demonstrated to modulate both metrics. FMRI data and GABA levels, as assessed by Magnetic Resonance Spectroscopy, were measured before and after 20 min of 1 mA anodal or sham tDCS. In line with previous studies, baseline GABA levels were negatively correlated with the strength of functional connectivity within the resting motor network. However, although we confirm the previously reported findings that anodal tDCS reduces GABA concentration and increases functional connectivity in the stimulated motor cortex; these changes are not correlated, suggesting they may be driven by distinct underlying mechanisms
Motion of quantum vortex lines near realistic rough boundaries
We numerically solve the three--dimensional, time--dependent Gross--Pitaevskii equation to model a superfluid flowing over a realistic rough boundary. Our model for the boundary is based on the atom--force microscope image of a NbTi vibrating wire used to generate turbulence in actual experiments. We find that near the boundary a dense region of quantum vortices is created, which forms a kind of `superfluid boundary layer'
Transcranial magnetic stimulation: From neurophysiology to pharmacology, molecular biology and genomics
Noninvasive plasticity paradigms, both physiologically induced and artificially induced, have come into their own in the
study of the effects of genetic variation on human cortical plasticity. These techniques have the singular advantage that
they enable one to study the effects of genetic variation in its natural and most relevant context, that of the awake
intact human cortex, in both health and disease. This review aims to introduce the currently available artificially induced
plasticity paradigms, their putative mechanisms—both in the traditional language of the systems neurophysiologist and
in the evolving (and perhaps more relevant for the purposes of stimulation genomics) reinterpretation in terms of
molecular neurochemistry, and highlights recent studies employing these techniques by way of examples of applications.Noninvasive plasticity paradigms, both physiologically induced and artificially induced, have come into their own in the study of the effects of genetic variation on human cortical plasticity. These techniques have the singular advantage that they enable one to study the effects of genetic variation in its natural and most relevant context, that of the awake intact human cortex, in both health and disease. This review aims to introduce the currently available artificially induced plasticity paradigms, their putative mechanismsĝ€"both in the traditional language of the systems neurophysiologist and in the evolving (and perhaps more relevant for the purposes of stimulation genomics) reinterpretation in terms of molecular neurochemistry, and highlights recent studies employing these techniques by way of examples of applications. © The Author(s) 2010
CASP, a novel, highly conserved alternative-splicing product of the CDP/cut/cux gene, lacks cut-repeat and homeo DNA-binding domains, and interacts with full-length CDP in vitro.
Human CDP/cut and its murine counterpart, cux1/CDP are homeodomain repressor proteins in the family of Drosophila Cut. Northern blot analysis reveals complex alternative splicing, including forms too small to encode the full 1505 amino acid protein. We have characterized a CDP/cut alternatively spliced cDNA (CASP) of 3.4 kb. Human CASP, a predicted 678 amino acid polypeptide, shares 400 amino acids with CDP, but has an alternate N terminal exon of 20 aa, and the C-terminal 258 amino acids diverge from CDP/cut entirely. As the unique C-terminus of CASP lacks the three ‘cut-repeats’ and homeodomain of CDP/cut, we predict it does not bind DNA. Murine CASP, 96% similar to human, shares these features. Database searches identify homologs in chicken (86% identical to human CASP) and yeast (29% identical to human). Murine CASP mRNA is ubiquitous in mouse tissues and in tissue-culture cell lines. We generated a specific antiserum against the unique C-terminus of CASP, and used this reagent to demonstrate that CASP protein is expressed as an approx. 80 kDa protein in human and murine cells. Co-translation of in vitro-translated CDP and CASP mRNA, followed by immunoprecipitation with specific anti-CASP IgG, shows that CASP polypeptide can form a complex with CDP. Studies of the intron/exon structure of the murine cux/CDP/mCASP locus (&100 kb) reveal that the unique 3∞ exons of CASP are interposed between cut-repeats 2 and 3 of the cux gene. We speculate that a primordial CASP-like gene captured a cut-repeat-homeobox gene to give rise to the eukaryotic Cut/CDP family of proteins. © 1997 Elsevier Science B.V
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