40,086 research outputs found
Taylor and Francis Dimensions Analysis for Impact Assessment Author Survey
No full textDimensions analysis for Taylor & Francis Impact Assessment Author Survey</p
Taylor-and-Francis_Impact-Assessment-of-Earth-and-Environmental-Sciences-Research-Author-Survey_Raw-Data_Figshare
No full textAnonymized responses dataset from the Taylor & Francis Impact Assessment of Earth & Environmental Sciences Research: Author Survey.In Spring 2020, Taylor & Francis surveyed authors from across our Earth & Environmental Sciences portfolio.We investigated what benefits publishing in our journals could impart on both the research and on the authors following publication, and we looked at to what extent global challenges, such as those expressed by the UN Sustainable Development Goals (SDGs), were shaping researcher ambitions.</div
An essay about the Francis Paudras Collection on Bud Powell by Peter Pullman
No full textThis is an essay about the Francis Paudras Collection on Bud Powell written by Peter Pullman, a jazz scholar and author of Wail: The Life of Bud Powell (Brooklyn: Bop Changes, 2012).One image file (pdf)This project was supported by a Recordings at Risk grant from the Council on Library and Information Resources (CLIR). The grant program is made possible by funding from The Andrew W. Mellon Foundation
Taylor & Francis post-publication author survey - submission decision factors - USA vs Global comparison
No full textThe Taylor & Francis post-publication author survey asks about the importance of various factors in authors' decisions to submit their article to a specific journal. This summary table and chart provides a snapshot of responses for the full year 2019, comparing the USA with the global average.</p
Alu/Alu recombination of disease-causing genomic Alu elements.
No full text<p><b>(A)</b> A schematic of the genomic AARP constructs used in this study. Two genomic Alu elements (Alu Sz and Sx) that have been shown to cause disease through Alu/Alu recombination within the MLL gene [<a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#pgen.1005016.ref022" target="_blank">22</a>] were cloned into the AARP reporter cassette and integrated into HEK293FRT cells (see <a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#pgen.1005016.s001" target="_blank">S1 Fig</a>, <a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#sec012" target="_blank">Materials and Methods</a>). <b>(B)</b> The average number of puro<sup>r</sup> colonies is plotted for MLL-AARP (18% sequence divergence) and 20%-AARP HEK293FRT cells. Data from at least three independent experiments using at least three independently isolated clones for each AARP cell line are averaged. Error bars denote standard error and statistical significance is shown using one-way ANOVA. <b>(C)</b> Graphic representation of the location of previously reported genomic Alu/Alu recombination junctions [<a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#pgen.1005016.ref002" target="_blank">2</a>,<a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#pgen.1005016.ref018" target="_blank">18</a>,<a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#pgen.1005016.ref023" target="_blank">23</a>–<a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#pgen.1005016.ref032" target="_blank">32</a>]. The Alu/Alu recombination product is divided into three segments of equal length (100 bp). The number of Alu/Alu recombination junctions expected and observed in each 100 bp interval is shown. An asterisk (*) marks an interval in which p<0.05 significance as determined by a chi-square test for observed vs. expected.</p
Using Altmetric data to encourage author engagement at Taylor & Francis
No full textJames Hardcastle, Head of Business Development at Wizdom.ai and former Senior Manager in the Research & Analytics department at Taylor & Francis, shares how Taylor & Francis use books data from Altmetric to encourage author engagement
Francis Lee Utley (interview)
No full textThis interview is included in the American Folklore Society Oral History Project held at the Archive of Folk Culture, American Folklife Center, Library of Congress, Washington, D.C. This item consists of oral history interviews with folklorist Francis Lee Utley conducted in 1973 by Patrick B. Mullen and Richard Reuss for the American Folklore Society Oral History Project. This collection consists of 2 sound tape reels : analog, 3 3/4 ips, 2 track, mono. ; 7 in. Originally recorded on July 19, 1973 by Patrick B. Mullen on a 7-inch reel, 3 3/4 ips, 2 track at an unidentified location; and on November 3, 1973 by Patrick B. Mullen and by Richard Reuss at the annual meeting of the American Folklore Society in Nashville, Tennessee on a Sony audiocassette. Sound recordings are first generation copies on two sound tape reels, 7 in. Biography/History note: Francis Lee Utley was born May 25, 1907 in Watertown, Wisconsin, and died March 8, 1974. He was a folklorist, medievalist, linguist, educator, and author who earned his M.A. in 1934 and Ph.D. in 1936 in literature at Harvard University. He taught at Ohio State University and the University of California at Berkeley, and was president of the American Folklore Society from 1951-1952
The distribution of Alu/Alu recombination junctions in AARP HEK293FRT cells.
No full text<p><b>(A)</b> A schematic of the Alu/Alu recombination product indicates, in purple, a 5’ section of Alu1 (containing sequence divergence relative to Alu2) and, in pink, Alu 2 (Alu Ya5 consensus sequence). Arrows represent PCR primers used to amplify Alu/Alu recombination products (<a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#pgen.1005016.s021" target="_blank">S2 Table</a>). <b>(B)</b> The distribution of Alu/Alu recombination junctions in 3%-, 5%-, and 10%-AARP HEK293FRT cells as determined by PCR and sequence analysis of DNA repair products from isolated puro<sup>r</sup> colonies. The Alu/Alu recombination product is divided into three segments of equal length (100 bp), which each contain the same extent of sequence divergence. The number of Alu/Alu recombination junctions expected and observed in each 100 bp interval is shown. An asterisk (*) marks an interval in which p<0.05 significance as determined by a chi-square test for observed vs. expected. <b>(C-E)</b> Alu/Alu recombination junctions mapped individually for the 3%- <b>(C)</b>, 5%- <b>(D)</b>, and 10%- <b>(E)</b> AARP HEK293FRT cells. The Alu/Alu recombination product was divided into segments of equal length according to the intervals of homology in which the Alu/Alu recombination junction can be mapped for each diverged AARP construct. The number of Alu/Alu recombination junctions observed is plotted on the y-axis for each interval of homology.</p
Human Alu Dataset
No full textHuman Alu dataset for "A two-state model of tree evolution and its applications to Alu retrotransposition
The Alu/Alu recombination system.
No full text<p><b>(A)</b> Schematic of the Alu/Alu recombination reporter. Cells harboring the reporter were neomycin resistant (neo<sup>r</sup>) and sensitive to puromycin (puro<sup>s</sup>) or blasticidin (blast<sup>s</sup>) or eGFP negative (eGFP<sup>-</sup>), depending on the reporter cassette used (AARP, AARB or AARG). Cells were grown in the presence of neomycin to eliminate cells that underwent spurious recombination before induction of DNA double-strand breaks (DSBs). DSBs were induced by transfection of an I-SceI endonuclease expression vector. <b>(B)</b> The reporter cassette was inserted using the Flp-In system (see <a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#pgen.1005016.s001" target="_blank">S1 Fig</a>, <a href="http://www.plosgenetics.org/article/info:doi/10.1371/journal.pgen.1005016#sec012" target="_blank">Materials and Methods</a>) into a single genomic location in each cell line, which allows variants of the vector (i.e. different Alu elements) to be inserted in the same chromosomal location. <b>(C)</b> Repair of an I-SceI-induced DNA DSB may result in a deletion of the sequence between the two Alu elements after which the cell becomes neomycin sensitive (neo<sup>s</sup>) and express the puromycin resistance (puro<sup>R</sup>), blasticidin resistance (blast<sup>R</sup>) or eGFP (eGFP<sup>+</sup>) gene. (<b>D and E</b>) I-SceI-induced DNA DSBs in the AARP <b>(D)</b> and AARB <b>(E)</b> HEK293FRT cells result in Alu/Alu recombination. Cells were transiently transfected with either I-SceI expression vector or empty vector (pUC19) control. Puromycin resistant (puro<sup>r</sup>) or blasticidin resistant (blast<sup>r</sup>) colonies were counted and are shown graphically. Error bars denote standard error and statistical significance was shown using one-way ANOVA. Below the graphs are representative flasks showing puro<sup>r</sup> or blast<sup>r</sup> colonies following I-SceI-induced DNA DSBs. (F) I-SceI-induced DNA DSBs in 0%-AARG HEK293FRT cells result in eGFP<sup>+</sup> cells. The percentage of eGFP<sup>+</sup> cells are shown graphically. Error bars represent standard error and statistical significance was shown using Student’s t-test. Representative flow plots from fluorescence activated cell sorting (FACS) analysis following transfections with I-SceI or empty vector pUC19 are shown below the graphs.</p
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