1,723,813 research outputs found
Linked collectors and determiners for: The importance of correct labelling of types: an example in Tortricidae (Lepidoptera) and its rectification.
No full textNatural history specimen data linked to collectors and determiners held within, "The importance of correct labelling of types: an example in Tortricidae (Lepidoptera) and its rectification". Claims or attributions were made on Bionomia by volunteer Scribes, <a href="http://bionomia.net/dataset/e143db18-f7fe-4f11-9457-edbf383c0e5f">https://bionomia.net/dataset/e143db18-f7fe-4f11-9457-edbf383c0e5f</a> using specimen data from the dataset aggregated by the Global Biodiversity Information Facility, <a href="https://gbif.org/dataset/e143db18-f7fe-4f11-9457-edbf383c0e5f">https://gbif.org/dataset/e143db18-f7fe-4f11-9457-edbf383c0e5f</a>. Formatted as a Frictionless Data package
Pathogenicity of four strains of staphylococci isolated from chickens with clinical tenosynovitis
No full textFour strains of staphylococci of different phage patterns (two principal and two minor phage types) associated with naturally occurring tenosynovitis were studied for virulence in chickens and tropism for tendon tissue. There were significant differences in virulence between the principal and minor phage types of Staphylococcus aureus. Infection with the principal phage types produced a generalized septicaemic disease and no specific tropism of the bacteria for tendon tissue although there were gross and histopathological changes in tendons and tendon sheaths indistinguishable from those in natural tenosynovitis. However, the pathogenesis of the experimental infections was different from that considered to occur in natural cases and provided further indirect evidence for the secondary nature of the staphylococcal infection in tenosynovitis..RE: 26 ref.; SC: ZA; CA; VE; 0V; 7A; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0307-9457&isbn=&volume=12&issue=2&spage=213&pages=213-220&date=1983&title=Avian%20Pathology&atitle=Pathogenicity%20of%20four%20strains%20of%20staphylococci%20isolated%20from%20chickens%20with%20clinical%20tenosynovitis.&aulast=Kibenge&pid=%3Cauthor%3EKibenge%2c%20F%20S%20B%3bWilcox%2c%20G%20E%3bPass%2c%20D%20A%3C%2Fauthor%3E%3CAN%3E19842232875%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Bacterial and viral agents associated with tenosynovitis in broiler breeders in Western Australia
No full textSeveral outbreaks of clinical tenosynovitis in poultry were investigated. Staphylococcus aureus was recovered from tendon tissue of from 52.98% of affected chickens in different outbreaks. It was also present in the liver and heart blood, and seemed to contribute to the high mortality observed following occurrence of clinical tenosynovitis in individual chickens. Other bacteria were recovered less often. Adenoviruses and reoviruses were also recovered from tendon tissue of chickens with a concurrent bacterial infection. Although the pathology observed in clinically affected chickens suggested a bacterial aetiology, it seemed that the outbreaks were due to an early reovirus infection which normally remained subclinical, and that secondary infection with bacteria, particularly S. aureus, was responsible for the development of clinical signs..RE: 24 ref.; SC: ZA; CA; VE; 0V; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0307-9457&isbn=&volume=11&issue=3&spage=351&pages=351-359&date=1982&title=Avian%20Pathology&atitle=Bacterial%20and%20viral%20agents%20associated%20with%20tenosynovitis%20in%20broiler%20breeders%20in%20Western%20Australia.&aulast=Kibenge&pid=%3Cauthor%3EKibenge%2c%20F%20S%20B%3bRobertson%2c%20M%20D%3bWilcox%2c%20G%20E%3bPass%2c%20D%20A%3C%2Fauthor%3E%3CAN%3E19822209448%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Experimental reovirus infection in chickens: observations on early viraemia and virus distribution in bone marrow, liver and enteric tissues
No full textThe nature of the viraemia and the tissue distribution of reovirus were studied in the early phase after oral infection of 1-day-old SPF, White Leghorn chicks with the R2 strain of avian reovirus. A range of tissues collected up to 3 weeks after infection was titrated for viral content. Virus was present in the plasma, erythrocyte and mononuclear fractions of the blood within 30 h after inoculation (p.i.) and was widely distributed in tissues, including the bone marrow by 3 to 5 days p.i. A greater part of the viraemia was associated with plasma, virus in the blood mononuclear fraction being detected only occasionally. There was more infective virus in duodenum than liver, and the highest virus titres were found in cloacal swabs taken 1 to 5 days p.i. It was also evident that virus reached the liver shortly after infection ( http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0307-9457&isbn=&volume=14&issue=1&spage=87&pages=87-98&date=1985&title=Avian%20Pathology&atitle=Experimental%20reovirus%20infection%20in%20chickens%3a%20observations%20on%20early%20viraemia%20and%20virus%20distribution%20in%20bone%20marrow%2c%20liver%20and%20enteric%20tissues.&aulast=Kibenge&pid=%3Cauthor%3EKibenge%2c%20F%20S%20B%3bGwaze%2c%20G%20E%3bJones%2c%20R%20C%3bChapman%2c%20A%20F%3bSavage%2c%20C%20E%3C%2Fauthor%3E%3CAN%3E19862272282%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Identification of serotype II infectious bursal disease virus proteins
No full textPolyacrylamide gel electrophoresis (PAGE) of serotype II IBDV (OH and MO strains) purified from infected Vero cells resolved a previously undetected major viral polypeptide, VP2. The molecular weight (MW) of VP2 was different between the two strains of serotype II. It was 43.5 kDa in strain OH and 44 kDa in strain MO. This was higher than the MW of VP2 in SAL strain of serotype I IBDV which was 41 kDa. VPX (50 kDa), VP3 (33 kDa) and VP4 (30.5 kDa) were similar in both serotype II virus strains but were also of higher MW than VPX (48 kDa), VP3 (32 kDa) and VP4 (30 kDa) of SAL virus. VP1 (80 kDa) had the same MW in both serotypes..RE: 25 ref.; SC: ZA; CA; VE; 0V; 7A; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0307-9457&isbn=&volume=17&issue=3&spage=679&pages=679-687&date=1988&title=Avian%20Pathology&atitle=Identification%20of%20serotype%20II%20infectious%20bursal%20disease%20virus%20proteins.&aulast=Kibenge&pid=%3Cauthor%3EKibenge%2c%20F%20S%20B%3bDhillon%2c%20A%20S%3bRussell%2c%20R%20G%3C%2Fauthor%3E%3CAN%3E19882212161%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Rapid passage of avian reovirus in one-day-old chicks: clinical and virological findings
No full textTwo avian reoviruses, strain Reo-25 and isolate W3-492 were given by mouth to eight one-day-old chicks. 3-7 days after inoculation, the liver, spleen, pancreas, caecal tonsil and duodenum were collected, weighed and titrated in cell culture for viral content. Tissue homogenates were passaged several times in day-old chicks. Reo-25 virus was passaged at 3-day and W3-492 virus at 3- and 7- or 14-day intervals. For both Reo-25 and W3-492 viruses, pathological effects and virus yields in tissues decreased with continued passages. In direct comparisons of reovirus W3-492 before chick passage (PO) and after four passages at 7-day intervals (P4) using standardised amounts of virus for inoculation of chickens, no major differences in pathological effects were observed. P4 virus could be recovered from duodenal tissue at 28 days and from liver tissue at 14 days. PO virus could be recovered from duodenal tissue at 14 days and from liver tissue at 10 days..RE: 25 ref.; SC: ZA; CA; VE; 0V; 7A; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0307-9457&isbn=&volume=16&issue=2&spage=213&pages=213-225&date=1987&title=Avian%20Pathology&atitle=Rapid%20passage%20of%20avian%20reovirus%20in%20one-day-old%20chicks%3a%20clinical%20and%20virological%20findings.&aulast=Kibenge&pid=%3Cauthor%3EKibenge%2c%20F%20S%20B%3bDhillon%2c%20A%20S%3C%2Fauthor%3E%3CAN%3E19872296778%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Pathogenesis of rotavirus infection in turkey poults
No full textThe pathogenesis of rotavirus infection was examined after experimental infection of conventional and specific-pathogen-free (SPF) turkey poults. In six experiments birds were exposed to turkey rotavirus isolates Tu-1 or Tu-2 or the chicken isolate Ch-1 at 7, 10 or 42 days of age. Poults were examined between 1 and 24 days after infection. Between 2 and 5 days watery droppings were observed in conjunction with remarkable paleness of the intestinal tract which was grossly observable. Maximum viral replication occurred between 2 and 5 days, during which period viral antigen could be demonstrated in the epithelial cells of the duodenum, jejunum, ileum and colon. Sporadically, virus antigen-positive cells were seen in the caecum. As early as 4 to 5 days rotavirus antibodies could be detected by indirect immunofluorescence assays. Remarkable leukocyte infiltration of the lamina propria, vacuolation of the epithelial cells and scalloping of the villous surface at the tips were observed in the intestine of infected birds. Infection caused impairment of absorption of D-xylose from the intestinal tract at 2 and 4 days..RE: 28 ref.; MS: 2 fig.; SC: ZA; CA; VE; 0V; 7A; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0307-9457&isbn=&volume=15&issue=3&spage=421&pages=421-435&date=1986&title=Avian%20Pathology&atitle=Pathogenesis%20of%20rotavirus%20infection%20in%20turkey%20poults.&aulast=Yason&pid=%3Cauthor%3EYason%2c%20C%20V%3bSchat%2c%20K%20A%3C%2Fauthor%3E%3CAN%3E19862284428%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Reovirus-induced tenosynovitis in chickens: the effect of breed
No full textThe effect of breed of chicken on infection with an arthrotropic avian reovirus (strain R2) was studied by oral or footpad inoculation of day-old chicks of the SPF light-hybrid, commercial White Leghorn egg-layer, and commercial Ross I broiler breeds, observed to 12 weeks of age. Although most inoculated birds of all three breeds developed swelling of one or both legs below the hock joint at 3 to 4 weeks of age, gross lesions of tenosynovitis became progressively more severe and extended above the joints only in broilers, whereas in most orally-infected SPF and commercial light chickens gross lesions were intermittently severe and regressed with time. Cloacal virus shedding continued up to 2 weeks in the lighter breeds and 3 weeks after infection in broilers. From a small proportion of infected chickens, reovirus was also recovered from heart, pancreas and caecal tonsils. In all breeds, the tissue in which virus persisted longest was the hock joint/tendon. There was a poor correlation between isolation of virus and the presence of gross lesions in chickens of 12 weeks of age, especially in broilers. Virus-neutralization tests demonstrated that seroconversion in the lighter breeds occurred mainly at 3 weeks, and in broilers at 4 weeks after infection. In all three breeds the footpad infection resulted in poorer growth than in the control and oral-infection groups. Oral infection had no apparent effect on growth rate. The greater susceptibility of broilers to reovirus infection is discussed..RE: 30 ref.; SC: ZA; CA; VE; 0V; 7A; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0307-9457&isbn=&volume=13&issue=3&spage=511&pages=511-528&date=1984&title=Avian%20Pathology&atitle=Reovirus-induced%20tenosynovitis%20in%20chickens%3a%20the%20effect%20of%20breed.&aulast=Jones&pid=%3Cauthor%3EJones%2c%20R%20C%3bKibenge%2c%20F%20S%20B%3C%2Fauthor%3E%3CAN%3E19842247265%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
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