1,723,288 research outputs found
Phagocytosis of the inert suspended clay kaolin by the gills of rainbow trout, Salmo gairdneri Richardson
No full textRE: 18 ref.; SC: ZA; CA; VE; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0140-7775&isbn=&volume=9&issue=2&spage=147&pages=147-151&date=1986&title=Journal%20of%20Fish%20Diseases&atitle=Phagocytosis%20of%20the%20inert%20suspended%20clay%20kaolin%20by%20the%20gills%20of%20rainbow%20trout%2c%20Salmo%20gairdneri%20Richardson.&aulast=Goldes&pid=%3Cauthor%3EGoldes%2c%20S%20A%3bFerguson%2c%20H%20W%3bDaoust%2c%20P%20Y%3bMoccia%2c%20R%20D%3C%2Fauthor%3E%3CAN%3E19862279495%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Nodular gill disease: a unique form of proliferative gill disease in rainbow trout, Salmo gairdneri Richardson
No full textIn an outbreak of proliferative gill disease in laboratory rainbow trout fingerlings lesions were multifocal among filaments and uniquely severe in the distal region of affected filaments, forming nodules. This contrasted with the more common forms of proliferative gill disease in Ontario, such as bacterial gill disease, in which lesions are diffuse along and among filaments. The cause is unknown, although cells with a very distinctive morphology were closely associated with the lesions..RE: 22 ref.; SC: ZA; CA; VE; 0V; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0140-7775&isbn=&volume=8&issue=6&spage=511&pages=511-522&date=1985&title=Journal%20of%20Fish%20Diseases&atitle=Nodular%20gill%20disease%3a%20a%20unique%20form%20of%20proliferative%20gill%20disease%20in%20rainbow%20trout%2c%20Salmo%20gairdneri%20Richardson.&aulast=Daoust&pid=%3Cauthor%3EDaoust%2c%20P%20Y%3bFerguson%2c%20H%20W%3C%2Fauthor%3E%3CAN%3E19862272504%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
The pathology of chronic ammonia toxicity in rainbow trout, Salmo gairdneri Richardson
No full textTwo groups of rainbow trout fingerlings were exposed for 90 days to 0.2 and 0.4 mg/l of un-ionized ammonia, respectively. Several fish exposed to the higher concentration soon developed clinical signs suggestive of a neurological dysfunction but subsequently recovered. No lesion attributable to ammonia was seen in the gills of any of the fish. This raises questions about the precise role of ammonia in the production of gill diseases in intensively cultured trout..RE: 25 ref.; RN: 7664-41-7; SC: ZA; CA; VE; EC; 0V; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0140-7775&isbn=&volume=7&issue=3&spage=199&pages=199-205&date=1984&title=Journal%20of%20Fish%20Diseases&atitle=The%20pathology%20of%20chronic%20ammonia%20toxicity%20in%20rainbow%20trout%2c%20Salmo%20gairdneri%20Richardson.&aulast=Daoust&pid=%3Cauthor%3EDaoust%2c%20P%20Y%3bFerguson%2c%20H%20W%3C%2Fauthor%3E%3CAN%3E19842244566%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Potential for recovery in nodular gill disease of rainbow trout, Salmo gairdneri Richardson
No full textNodular gill disease characterized by severe multifocal epithelial hyperplasia was diagnosed in a group of rainbow trout fingerlings. Gills of a large proportion of fish transferred from the affected tank to smaller aquaria showed a marked decrease in severity of the lesions after 2 months. Repair of hyperplastic lesions may occur in various forms of proliferative gill disease, provided that the basement membrane lining the filaments and lamellae remains intact..RE: 11 ref.; SC: ZA; CA; VE; 0V; 0ISource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0140-7775&isbn=&volume=9&issue=4&spage=313&pages=313-318&date=1986&title=Journal%20of%20Fish%20Diseases&atitle=Potential%20for%20recovery%20in%20nodular%20gill%20disease%20of%20rainbow%20trout%2c%20Salmo%20gairdneri%20Richardson.&aulast=Daoust&pid=%3Cauthor%3EDaoust%2c%20P%20Y%3bFerguson%2c%20H%20W%3C%2Fauthor%3E%3CAN%3E19862285526%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
[South African Rwys & Harbours, Engine Drawing Card, Sketch No. 7775]
No full textThis engine drawing card was created for South African Railways & Harbours, Class 14-3-34 1/4 E. Section H-14 1/4 E. Sketch 7775. Copy Spec. 25F-375
Uptake and tissue distribution of liposomes after intraperitoneal administration to rainbow trout, Oncorhynchus mykiss (Richardson): a preliminary report
No full textLiposomes were prepared from phosphotidylcholine and cholesterol at a molar ratio of 4:1. One group of 10 fish received an intraperitoneal injection of multilamella liposomes (MLV), while another group received large unilamellar liposomes (LUV). Approx. 20 micro Ci of tritiated cholesterol had been incorporated per 50 micro moles of lipid, and radioactive counts from each liposome preparation averaged 280 000 cpm/10 micro l of the liposome suspension before injection. The fish were killed 24 h after injection and the various tissues examined for radioactivity. Uptake of LUVs was significantly greater than that of MLVs. The spleen, and anterior and posterior kidney were primary sites of localization. Liver had the next greatest uptake, followed by gills, heart and fat; muscle tissue had the lowest uptake. It is suggested that LUVs would provide an appropriate delivery system for vaccines, as well as for certain antimicrobial agents..RE: 13 ref.; SC: CA; VE; 0I; ZA; 0VSource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0140-7775&isbn=&volume=13&issue=4&spage=329&pages=329-332&date=1990&title=Journal%20of%20Fish%20Diseases&atitle=Uptake%20and%20tissue%20distribution%20of%20liposomes%20after%20intraperitoneal%20administration%20to%20rainbow%20trout%2c%20Oncorhynchus%20mykiss%20%28Richardson%29%3a%20a%20preliminary%20report.&aulast=Power&pid=%3Cauthor%3EPower%2c%20C%20A%3bMarkham%2c%20R%20J%20F%3bDonald%2c%20A%20W%3C%2Fauthor%3E%3CAN%3E19902209901%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Pathology of bacterial gill disease: ultrastructure of branchial lesions
No full textThe range of branchial lesions associated with bacterial gill disease (BGD) in rainbow trout, Oncorhynchus mykiss, was investigated through the ultrastructural examination of 23 separate outbreaks of the disease condition within commercially reared stocks. Diseased branchial tissues had a large and diffusely distributed monomorphic population of filamentous bacteria which were strictly epicellular. Although bacterial colonization was restricted to the branchial cavity, it was neither site nor cell specific: epithelial and chloride cells of the lamellae, filaments and lining tissues of the branchial cavity were all uniformly affected. The bacteria possessed an extensive glycocalyx which appeared to facilitate adhesion to the apices of the microridged sub-unit modification of the cell surface. Sites of colonization were accompanied by a diffuse pattern of cellular degeneration and necrosis that was generally restricted to the outer layers of epithelium. The polarity and nature of these changes suggest that the mechanism of interaction between the bacteria and host cells includes progressive hydropic changes as a sequel to primary membrane damage and consequent increased cell permeability. These cellular changes were accompanied by the range of stereotypical responses of the gill to damage frequently reported for BGD including lamellar fusion, epithelial hyperplasia, and squamous and mucous cell metaplasia, in addition to lamellar spongiosis..RE: 42 ref.; SC: CA; VE; 0I; ZA; 0VSource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0140-7775&isbn=&volume=14&issue=1&spage=1&pages=1-20&date=1991&title=Journal%20of%20Fish%20Diseases&atitle=Pathology%20of%20bacterial%20gill%20disease%3a%20ultrastructure%20of%20branchial%20lesions.&aulast=Speare&pid=%3Cauthor%3ESpeare%2c%20D%20J%3bFerguson%2c%20H%20W%3bBeamish%2c%20F%20W%20M%3bYager%2c%20J%20A%3bYamashiro%2c%20S%3C%2Fauthor%3E%3CAN%3E19912256144%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Pathology of bacterial gill disease: sequential development of lesions during natural outbreaks of disease
No full textThe sequential pattern of bacterial colonization and the sequential development of branchial lesions during typical outbreaks of bacterial gill disease (BGD) was studied among 4 groups of commercially reared rainbow trout fingerlings, Oncorhynchus mykiss. During a 5-month monitoring regime, before the onset of natural outbreaks of BGD, gill morphology of examined fish remained unaltered. Bacterial colonization of the gills was immediately preceeded by several gill changes which were widely present in fish from all groups under study. These changes, which were detectable at the ultrastructural level only, included cytoplasmic blistering and degeneration of the microridges of the superficial filament epithelium, in addition to slight topographical irregularity of the filament tips suggestive of mild hyperplasia. Bacterial colonization began at these altered filament tips before spreading to more proximal regions of the filament and adjacent lamellar surfaces. Explosive increases in proportional morbidity and mortality coincided with the development of the following gill lesions: extensive bacterial colonization of lamellar surfaces, lamellar epithelial hydropic degeneration and necrosis, and lamellar oedema. In contrast, gill lesions such as lamellar fusion, epithelial hyperplasia and various metaplastic responses were detected either as subacute (3-5 days) or chronic (7-14 days) changes. The role of epithelial necrosis as an important event during BGD is discussed as a possible mechanism leading to development of sub-acute and chronic gill lesions..RE: 37 ref.; SC: CA; VE; 0I; ZA; 0VSource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0140-7775&isbn=&volume=14&issue=1&spage=21&pages=21-32&date=1991&title=Journal%20of%20Fish%20Diseases&atitle=Pathology%20of%20bacterial%20gill%20disease%3a%20sequential%20development%20of%20lesions%20during%20natural%20outbreaks%20of%20disease.&aulast=Speare&pid=%3Cauthor%3ESpeare%2c%20D%20J%3bFerguson%2c%20H%20W%3bBeamish%2c%20F%20W%20M%3bYager%2c%20J%20A%3bYamashiro%2c%20S%3C%2Fauthor%3E%3CAN%3E19912256145%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
Loma salmonae-associated growth rate suppression in rainbow trout, Oncorhynchus mykiss (Walbaum), occurs during early onset xenoma dissolution as determined by in situ hybridization and immunohistochemistry
No full textExperimental infection of rainbow trout juveniles with L. salmonae at a water temperature of 15 degrees C yielded detectable parasite DNA within the gills by the second week after exposure (AE) and detectable spore-wall antigen within developing xenomas by week 3 AE, as determined by in situ hybridization and monoclonal antibody (Mab) based immunohistochemistry, respectively. The microsporidian was most commonly located within endothelial cells of lamellar basal channels. Whereas the onset of xenoma formation appeared to be relatively synchronous, as expected from previous studies, xenoma dissolution followed an unexpected biphasic pattern with peaks at weeks 4 and 9 AE. The onset of significant growth rate suppression, at week 4 AE in exposed fish, was temporally associated with the appearance of gill lesions which, in turn, were centred about sites of premature xenoma dissolution. The latter was determined by the detection of spore-wall antigen within lesions. Co-habitant control fish began developing xenomas by week 10, indicating the infective potential of those spores released from the principal fish during early xenoma dissolution. It is concluded that although infection with L. salmonae significantly affects fish growth rates, the time-course of this suppression is limited and growth rate recovery starts before the resolution of the infection..RE: 24 ref.; SC: 0I; ZA; VE; CA; BE; PA; 0VSource type: Electronic(1) http://upei-resolver.asin-risa.ca?sid=SP:CABI&id=pmid:&id=&issn=0140-7775&isbn=&volume=21&issue=5&spage=345&pages=345-354&date=1998&title=Journal%20of%20Fish%20Diseases&atitle=Loma%20salmonae-associated%20growth%20rate%20suppression%20in%20rainbow%20trout%2c%20Oncorhynchus%20mykiss%20%28Walbaum%29%2c%20occurs%20during%20early%20onset%20xenoma%20dissolution%20as%20determined%20by%20in%20situ%20hybridization%20and%20immunohistochemistry.&aulast=Speare&pid=%3Cauthor%3ESpeare%2c%20D%20J%3bDaley%2c%20J%3bMarkham%2c%20R%20J%20F%3bSheppard%2c%20J%3bBeaman%2c%20H%20J%3bSanchez%2c%20J%20G%3C%2Fauthor%3E%3CAN%3E19992201414%3C%2FAN%3E%3CDT%3EJournal%20article%3C%2FDT%3
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