1,782,079 research outputs found
EDU 362: Subject Methods (Literature)
No full textEDU 362: SUBJECT METHODS (LITERATURE)Daystar Universit
Additional file 7: of MiR-362-5p promotes the malignancy of chronic myelocytic leukaemia via down-regulation of GADD45α
No full textqRT-PCR analysis of miR-362-5p in 293 T cell lines transfected with a miR-362-5p mimic (mimic) or a mimic control (mimic-ctrl). **P < 0.01. (DOC 44 kb
Participant 362 Community Conversations Artwork
No full textIn this poster art submission, Participant 362 describes their work as art that "shows coping methods to use during quarantine." The participant portrays ways people can remain busy while at home including exploring new hobbies, focusing on health and wellness, maintaining communication with others using technology, and partaking in safe forms of entertainment including playing games at home and watching television.New Jersey Department of Healt
Additional file 5: of MiR-362-5p promotes the malignancy of chronic myelocytic leukaemia via down-regulation of GADD45α
No full textRelative expression of miR-362-5p in Lv- miR-362-5p-inhibitor expressing K562 cells. qRT-PCR analysis of miR-362-5p in Lv-miR-362-5p-inhibitor (Lv-inhibitor) or Lv-ctrl expressing K562 cells. The expression levels of miR-362-5p were normalized to that of the U6 snRNA control. **P < 0.01. (DOC 58 kb
Additional file 6: of MiR-362-5p promotes the malignancy of chronic myelocytic leukaemia via down-regulation of GADD45α
No full textMiR-362-5p was reduced in lv-miR-362-5p-inhibitor tumors compare with lv-ctrl tumors. qRT-PCR analysis of miR-362-5p in Lv-miR-362-5p-inhibitor or Lv-ctrl expressing xenograft tumors. *P < 0.05. (DOC 50 kb
Bulletin No. 362 - Wyoming Hybrid Corn Performance Trials, 1955-1958
No full textBulletin No. 362 - Wyoming Hybrid Corn Performance Trials, 1955-195
Resolución UNRN N° 362/2009. Contratrar personal temporario
No full textFil: Universidad Nacional de Río Negro (U). Universidad Nacional de Río Negro. Río Negro, ArgentinaResolución UNRN N° 362/2009. Contratrar personal temporariofals
Image_1_MiR-362-5p, Which Is Regulated by Long Non-Coding RNA MBNL1-AS1, Promotes the Cell Proliferation and Tumor Growth of Bladder Cancer by Targeting QKI.tif
No full textIn this study, we found miR-362-5p was upregulated in bladder cancer tissues and we predicted that QKI is potential a target of miR-362-5p and MBNL1-AS1 might be able to directly target to miR-362-5p. We attempted to evaluate whether miR-362-5p could play its roles in bladder cancer through regulating QKI (quaking) and whether the expression and function of miR-362-5p could be mediated by lncRNA MBNL1-AS1. We performed the gain- and loss-function experiments to explore the association between miR-362-5p expression and bladder cancer proliferation. In vivo, the nude mice were injected with miR-362-5p knockdown SW780 cells to assess the effects of miR-362-5p on tumor growth. The results showed upregulation of miR-362-5p promoted cell proliferation of bladder cancer cells. MBNL1-AS1 and QKI could directly bind with miR-362-5p, and knockdown of MBNL1-AS1 or QKI could abrogate the regulatory effects of miR-362-5p on bladder cancer cell proliferation. Furthermore, downregulation of miR-362-5p inhibited bladder tumor growth and increased QKI expression. Our data unveiled that miR-362-5p may play an oncogenic role in bladder cancer through QKI and MBNL1-AS1 might function as a sponge to mediate the miR-362-5p expression and function.</p
Image_2_MiR-362-5p, Which Is Regulated by Long Non-Coding RNA MBNL1-AS1, Promotes the Cell Proliferation and Tumor Growth of Bladder Cancer by Targeting QKI.tif
No full textIn this study, we found miR-362-5p was upregulated in bladder cancer tissues and we predicted that QKI is potential a target of miR-362-5p and MBNL1-AS1 might be able to directly target to miR-362-5p. We attempted to evaluate whether miR-362-5p could play its roles in bladder cancer through regulating QKI (quaking) and whether the expression and function of miR-362-5p could be mediated by lncRNA MBNL1-AS1. We performed the gain- and loss-function experiments to explore the association between miR-362-5p expression and bladder cancer proliferation. In vivo, the nude mice were injected with miR-362-5p knockdown SW780 cells to assess the effects of miR-362-5p on tumor growth. The results showed upregulation of miR-362-5p promoted cell proliferation of bladder cancer cells. MBNL1-AS1 and QKI could directly bind with miR-362-5p, and knockdown of MBNL1-AS1 or QKI could abrogate the regulatory effects of miR-362-5p on bladder cancer cell proliferation. Furthermore, downregulation of miR-362-5p inhibited bladder tumor growth and increased QKI expression. Our data unveiled that miR-362-5p may play an oncogenic role in bladder cancer through QKI and MBNL1-AS1 might function as a sponge to mediate the miR-362-5p expression and function.</p
Image_3_MiR-362-5p, Which Is Regulated by Long Non-Coding RNA MBNL1-AS1, Promotes the Cell Proliferation and Tumor Growth of Bladder Cancer by Targeting QKI.tif
No full textIn this study, we found miR-362-5p was upregulated in bladder cancer tissues and we predicted that QKI is potential a target of miR-362-5p and MBNL1-AS1 might be able to directly target to miR-362-5p. We attempted to evaluate whether miR-362-5p could play its roles in bladder cancer through regulating QKI (quaking) and whether the expression and function of miR-362-5p could be mediated by lncRNA MBNL1-AS1. We performed the gain- and loss-function experiments to explore the association between miR-362-5p expression and bladder cancer proliferation. In vivo, the nude mice were injected with miR-362-5p knockdown SW780 cells to assess the effects of miR-362-5p on tumor growth. The results showed upregulation of miR-362-5p promoted cell proliferation of bladder cancer cells. MBNL1-AS1 and QKI could directly bind with miR-362-5p, and knockdown of MBNL1-AS1 or QKI could abrogate the regulatory effects of miR-362-5p on bladder cancer cell proliferation. Furthermore, downregulation of miR-362-5p inhibited bladder tumor growth and increased QKI expression. Our data unveiled that miR-362-5p may play an oncogenic role in bladder cancer through QKI and MBNL1-AS1 might function as a sponge to mediate the miR-362-5p expression and function.</p
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