1,806,744 research outputs found
Proceedings of the Integrated Ocean Drilling Program Expedition, Volume 335: Superfast Spreading Rate Crust 4. Expedition 335 of the riserless drilling platform Puntarenas, Costa Rica, to Balboa, Panama, Site 1256 13 April–3 June 2011
No full textAdditional file 1 of Characterization of miR-335-5p and miR-335-3p in human osteoarthritic tissues
No full textAdditional file 1: Supplemental Figure 1. A) miR-335-5p and B) miR-335-3p inhibition in late-stage knee OA fat pad explants used for custom TaqMan Gene Expression Array. n=3; -5p = miR-335-5p; -3p = miR-335-3p; inh. = inhibitor; *p < 0.05 versus control inhibitor
Additional file 5 of Characterization of miR-335-5p and miR-335-3p in human osteoarthritic tissues
No full textAdditional file 5: Supplemental Table 3. Filtered list of miR-335-5p and miR-335-3p gene targets used in custom TaqMan Gene Expression Array
Additional file 6 of Characterization of miR-335-5p and miR-335-3p in human osteoarthritic tissues
No full textAdditional file 6: Supplemental Table 4. Lists of unique and overlapping miR-335-5p and miR-335-3p gene targets reported by Ali et al. Osteoarthritis and Cartilage (2020)
Additional file 2 of Characterization of miR-335-5p and miR-335-3p in human osteoarthritic tissues
No full textAdditional file 2: Supplemental Figure 2. Gene expression changes in key adipogenesis pathway components following miR-335-5p modulation. A) PPARγ = peroxisome proliferator activated receptor gamma. B) CEBPA = CCAAT enhancer binding protein alpha. C) LEP = leptin. D) DGKD = diacylglycerol kinase delta. n=6; -5p = miR-335-5p; inh. = inhibitor; mim. = mimic; FC = fold-change; bars = 95% CI; *p < 0.05 versus control inhibitor or mimic
American Legion, Toledo Post 335 Inc., yearbook, 1968-1969
No full textA yearbook for the American Legion, Toledo Post No. 335 Inc. dated 1968 to 1969. The yearbook contains information on the history of the post, officers, committees, advertisements, and a membership directory
Resolución UNRN N° 335/2009. Contratrar personal temporario
No full textFil: Universidad Nacional de Río Negro (U). Universidad Nacional de Río Negro. Río Negro, ArgentinaResolución UNRN N° 335/2009. Contratrar personal temporariofals
LIPIcs, Volume 335, ECRTS 2025, Complete Volume
No full textLIPIcs, Volume 335, ECRTS 2025, Complete Volum
Supplementary Material for: circZMYM2 Competed Endogenously with miR-335-5p to Regulate JMJD2C in Pancreatic Cancer
No full textBackground/Aims: We aimed to study the involvement of circZMYM2 (hsa_circ_0099999) in pancreatic cancer (PC) cell proliferation, apoptosis and invasion and to figured out the underlying mechanism of circZMYM2 regulating miR-335-5p and JMJD2C. Methods: CircRNA differential expressions in twenty PC samples and paired normal tissue samples were analyzed using Arraystar Human CircRNA microarray V1. CircZMYM2 expression level was determined via qRT-PCR. The effects of circZMYM2 inhibition and overexpression on cell proliferation, cell apoptosis and cell invasion were investigated by CCK-8 assays, Flow cytometry assays and Transwell assays. An animal experiment on nude mice was put forward to test the influence of circZMYM2 knockdown on tumor growth. The relationship between circZMYM2, miR-335 and JMJD2C was verified by RNA pull down, dual-luciferase reporter assays and rescue experiment. The effect of circZMYM2 and miR-335-5p on the expression of JMJD2C protein was detected by western blot. Results: CircZMYM2 overexpression was observed in both PC tissues and cells. Knockdown of circZMYM2 inhibited proliferation, induced apoptosis, and weakened invasion ability of cancer cells. Tumor growth was restrained in vivo. CircZMYM2 repressed the expression of its target miR-335-5p. MiR-335-5p attenuated pancreatic cancer development via inhibition of JMJD2C. Conclusion: Our study demonstrated that circZMYM2 promoted PC progression. CircZMYM2 had a sponge effect on miR-335-5p and modulated the downstream oncogene JMJD2C
- …
